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Your search keyword '"Bui, Cuong J."' showing total 140 results
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140 results on '"Bui, Cuong J."'

20. Impact of the COVID-19 Pandemic on Neurosurgical Residency Training in New Orleans

Author

Scullen, Tyler, Mathkour, Mansour, Maulucci, Christopher M., Dumont, Aaron S., Bui, Cuong J., and Keen, Joseph R.

Subjects
Academic Medical Centers, Pandemic, SARS-CoV-2, Pneumonia, Viral, COVID-19, New Orleans, Internship and Residency, Article, Education, Betacoronavirus, Humans, Neurological surgery, Coronavirus Infections, Pandemics
Published
2020

24. Cholangiocarcinoma Metastasis to the Spine and Cranium

Author

Hanna, Joshua A., primary, Mathkour, Mansour, additional, Gouveia, Edna E., additional, Glynn, Ryan, additional, Divagaran, Adhira, additional, Lane, JonMark B., additional, Bui, Cuong J., additional, Sulaiman, Olawale A., additional, and Smith, Roger D., additional

Published
2020
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25. Comparison of elderly and young patient populations treated with deep brain stimulation for Parkinson’s disease: long-term outcomes with up to 7 years of follow-up

Author

Hanna, Joshua A., primary, Scullen, Tyler, additional, Kahn, Lora, additional, Mathkour, Mansour, additional, Gouveia, Edna E., additional, Garces, Juanita, additional, Evans, Leah M., additional, Lea, Georgia, additional, Houghton, David J., additional, Biro, Erin, additional, Bui, Cuong J., additional, Sulaiman, Olawale A., additional, and Smith, Roger D., additional

Published
2019
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36. Craniopharyngioma marsupialization

Author

Acakpo-Satchivi, Leslie, primary, Tubbs, R. Shane, additional, Woolley, Audie L., additional, Bui, Cuong J., additional, Liechty, Peter, additional, Hammers, Yuki, additional, Wellons, John, additional, Blount, Jeffrey P., additional, and Oakes, W. Jerry, additional

Published
2007
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47. Minimally Invasive Laminectomy in Spondylolisthetic Lumbar Stenosis.

Author

Caralopoulos, Ilias N. and Bui, Cuong J.

Subjects
*SPONDYLOLISTHESIS, *SPINE abnormalities, *STENOSIS, *OLDER patients, *NEUROGENIC claudication, *SPINE diseases, *SPINAL stenosis
Abstract

Background: Degenerative lumbar stenosis associated with spondylolisthesis is common in elderly patients. The most common symptoms are those of neurogenic claudication with leg pain. Surgery is indicated for those who fail conservative management. The generally accepted recommendation is to perform a laminectomy and a fusion at the involved level. Methods: We reviewed our results for minimally invasive single-level decompression without fusion performed by the senior author in patients with symptomatic lumbar stenosis with spondylolisthesis with no dynamic instability from 2008 to 2011 at a single institution. Outcomes were measured using the visual analog scale (VAS), Prolo Economic Functional Rating Scale, and revised Oswestry Disability Index (ODI) at initial presentation and at 3-month, 6-month, and 1-year follow-up time points. Results: Records for 28 patients (19 males, 9 females) were reviewed. The success rate, defined as improvement in pain and functional outcome without the need for surgical fusion, was 86%. VAS scores decreased by 6.3 points, Prolo scores increased by 3.5 points, and the ODI decreased by 31% at 1 year. All changes were statistically significant. Conclusion: Minimally invasive decompression alone can be a reasonable alternative to decompression and fusion for patients with spondylolisthetic lumbar stenosis and neurogenic claudication with leg pain. Decompression without fusion should be considered for older patients and for patients who are not ideal fusion candidates. [ABSTRACT FROM AUTHOR]

Published
2014

48. Ca2and CaM kinase regulate neurofilament expression

Author

Bui, Cuong J., Beaman-Hall, Carol M., and Vallano, Mary L.

Abstract

Cultured cerebellar granule neurons were used to examine the hypothesis that NMDA or KCl-dependent Ca2influx activates a signaling cascade mediating transcription, translation and assembly of neurofilaments. The data indicate that 25 mM KCl and 140 μM NMDA induce neurofilament protein, mRNA, and assembly in neurites, and these require Ca2entry through voltage-sensitive Ca2channels or NMDA receptors, respectively. The CaM kinase and transcriptional inhibitors, KN-62 and actinomycin D, respectively, attenuate the stimulatory effect of Ca2. Granular neurons are a valuable model to explore the molecular basis for transcriptional regulation of neurofilament gene expression by neuronal activity.

Published
2003

49. Depolarization and Ca2+ downregulate CB1 receptors and CB1-mediated signaling in cerebellar granule neurons

Author

Vallano, Mary Lou, Beaman-Hall, Carol M., Bui, Cuong J., and Middleton, Frank A.

Subjects
*CANNABINOIDS, *CANNABIS (Genus), *HALLUCINOGENIC drugs, *NEUROSCIENCES, *NEUROCHEMISTRY
Abstract

Abstract: Presynaptic terminals of cerebellar granule neurons are primary targets of cannabinoids, which act through type 1 Gαi/o-coupled cannabinoid receptors (CB1) to modulate glutamate release. To study CB1 signaling investigators use primary cultures of granule neurons, typically grown in medium supplemented with elevated KCl to improve long-term survival. Herein, we demonstrate that CB1 expression and signaling are perturbed under these conditions. Specifically, immunochemical and RT–PCR assays indicate that depolarizing growth conditions decrease CB1 protein, mRNA and CB1-mediated inhibition of adenylyl cyclase compared to cultures grown in physiologic medium containing 5mM KCl. Depolarization-dependent downregulation of CB1 mRNA, like survival, is attenuated by L-type VDCC antagonists but not the Na+-channel antagonist, tetrodotoxin. Comparison of oligonucleotide microarrays from cultures grown in 5mM versus 25mM KCl confirms that depolarization reduces CB1 mRNA, but not mRNAs encoding several G-protein subunits or adenylyl cyclases. However, significant alterations in synaptic signaling proteins that likely lie downstream of CB1 are observed, including K+ channels, α-neurexins, cAMP-GEFII, Munc13-3, secretogranin and synaptotagmin. These findings make a compelling argument to adopt cultures grown in 5mM KCl for future study of CB1 signaling in granule neurons. Further, they suggest that a depolarization and Ca2+-dependent signaling pathway represses CB1 gene transcription. [Copyright &y& Elsevier]

Published
2006
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50. Multistage surgical repair for split notochord syndrome with neuroenteric fistula: case report.

Author

Mathkour M, Scullen T, Huang B, Werner C, Gouveia EE, Abou-Al-Shaar H, Maulucci CM, Steiner RB, St Hilaire H, and Bui CJ

Subjects
Female, Humans, Infant, Newborn, Spina Bifida Cystica complications, Spina Bifida Cystica surgery, Spinal Dysraphism complications, Spinal Dysraphism surgery, Syndrome, Treatment Outcome, Ventriculoperitoneal Shunt, Fistula surgery, Gastrointestinal Tract surgery, Neurosurgical Procedures methods, Notochord abnormalities, Notochord surgery, Spinal Cord Diseases surgery
Abstract

Split notochord syndrome (SNS) is a rare congenital defect of the central nervous system and has been associated with several anomalies affecting multiple organ systems. One association has been communication with the gastrointestinal tract and the spine, previously identified as a neuroenteric fistula (NEF). Here, the authors describe the unique case of a female infant with SNS and NEF treated with a multistage surgical repair. The three-stage operative plan included a two-stage repair of the defect and temporary subgaleal shunting followed by delayed ventriculoperitoneal shunt placement. The infant recovered well postsurgery and over a 5-year follow-up. A case description, surgical techniques, and rationale are reported. Additionally, a systematic review of the literature utilizing the MEDLINE database was performed.Treatment of SNS with NEF using a multidisciplinary multistaged approach to repair the intestinal defect, close the neural elements, and divert cerebrospinal fluid to the peritoneum is shown to be a safe and viable option for future cases.

Published
2020
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