11 results on '"Cai, Huiyao"'
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2. PTPN22 through the regulation of Th17/Treg balance acts as a potential target for the treatment of Graves' disease
3. TSHR-based chimeric antigen receptor T cell specifically deplete auto-reactive B lymphocytes for treatment of autoimmune thyroid disease
4. Low Iodine in the Follicular Lumen Caused by Cytoplasm Mis-localization of Sodium Iodide Symporter may Induce Nodular Goiter
5. Iodinated TG in Thyroid Follicles Regulate TSH/TSHR Signaling for NIS Expression
6. Optimal iodine supplementation during antithyroid drug therapy for Graves’ disease is associated with lower recurrence rates than iodine restriction
7. Circular RNA HIPK3 contributes to hyperglycemia and insulin homeostasis by sponging miR-192-5p and upregulating transcription factor forkhead box O1
8. Iodinated TG in Thyroid Follicular Lumen Regulates TTF‐1 and PAX8 Expression via TSH/TSHR Signaling Pathway
9. Glycolysis-associated enzymes existing in the follicular lumen of the thyroid may interfere with energy metabolism
10. Presence of free triiodothyronine and free thyroxine in thyroid follicles may be correlated with the quick secretion of thyroid hormones under certain physiological conditions
11. Upregulation of TSHR, TTF-1, and PAX8 in Nodular Goiter Is Associated with Iodine Deficiency in the Follicular Lumen
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