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33. Cuneo tendinous suture - the story of one publication

Author

D. G. Nakonechny, A. N. Kiseleva, and A. Cambon-Binder

Subjects
hand surgery, tendon suture, damage to flexor tendons of fingers, fexor tendon repair, flexor tendon leasure, Orthopedic surgery, RD701-811
Abstract

In Russia and in post-Soviet countries tendon Cuneo suture is still widely known and is applied in clinical practice because of its strength and simplicity. One can find its sketch alongwith the sketches of Rozov and Kozakov sutures in most Russian handbooks on operative surgery. In foreign Uterature, however, this term is never used, and the authorship of the technique is attributed to S. Bunnell. According to the original source, the tendon suture technique suggested by S. Bunnell is different from that of B. Cuneo. Likewise, Cuneo tendon suture cannot be appUed with the use of tendon forceps, as suggested by S. Bunnell. Besides, to confirm proper use of B. Cuneo’s name in the case of the tendon suture in question, we cite an adapted translation of a certain paper by B. Cuneo and A. TaUhefer, devoted to a case study where the authors used suture of flexor tendon of little finger. We also provide historical background, concerning some interesting facts and people relevant to the topic.

Published
2018
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39. Liste des auteurs

Author

Abelin-genevois, Kariman, primary, Beaudet, Philippe, additional, Blondel, Benjamin, additional, Bourg, Véronique, additional, Bourgeois, Emeline, additional, Boyer, Bertrand, additional, Brilhault, Jean, additional, Cambon-binder, Adeline, additional, Cazorla, Céline, additional, Cognet, Jean-michel, additional, Cornu, Olivier, additional, Crenn, Vincent, additional, Dieterich, Klaus, additional, Fassier, Alice, additional, Fuentes, Stéphane, additional, Gaudias, Jeannot, additional, Gayet, Louis-Etienne, additional, Geffroy, Loïc, additional, Griffet, Jacques, additional, Huten, Denis, additional, Jeremic, Dragan, additional, Lambotte, Jean-Christophe, additional, Lintz, François, additional, Manon, Julie, additional, Mares, Olivier, additional, Maximen, Julien, additional, Métais, Pierre, additional, Moukoko, Didier, additional, Ohl, Xavier, additional, Ollat, Didier, additional, Pailhé, Régis, additional, Pesenti, Sébastien, additional, Prost, Solène, additional, Putineanu, Dan, additional, Richardi, Gérard, additional, Rivière, Charles, additional, Ropars, Mickaël, additional, Siboni, Renaud, additional, Tribak, Karim, additional, Tronchot, Alexandre, additional, Tropiano, Patrick, additional, Vendeuvre, Tanguy, additional, Vendittoli, Pascal-André, additional, and Villet, Loic, additional

Published
2020
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46. Intraneural perineurioma: a retrospective study of 19 patients

Author

Jaber Alkhaili, Adeline Cambon-Binder, and Zoubir Belkheyar

Subjects
intraneural perineurioma, hypertrophic neuropathy, pseudo-onion bulbs, Medicine
Abstract

Intraneural perineurioma is a benign neoplasm of peripheral nerve sheath with perineurial cell origin that typically affects teenagers and young adults and tends to result in a motor-predominant neuropathy. The aim of our study is to present the epidemiology, clinical presentation, way of diagnosis and management plan in a consecutive patient series. Ninteen patients diagnosed as having intraneural perineurioma were retrospectively chart reviewed. Diagnosis was done by MRI and/or biopsy with morphological, immunohistochemical staining study confirmation. Patients assessments included gender, age, symptoms, tumor localization, radiological aspect, management and pathological confirmation.Sex ratio was 10 males to 9 females with mean age of 31.2 (15-64). All the patients presented with motor deficit, ten with sensory deficit. Upper limb was involved in 11 cases (among them 4 lesions of brachial plexus), the lower limb in 8 cases. On magnetic resonance imaging, 16 patients showed a nerve enlargement whereas 5 presented with atypical fusiform tumor. Eighteen patients were operated for excision biopsy and/or palliative treatment for their motor deficit. Anatomopathological analysis confirmed the diagnosis in seventeen cases with a morphological pseudo-onion bulb shape and/or specific immunohistochemical assay. One patient had only palliative treatment without excision biopsy. Our data confirmed the equal penetration of intraneural perineurioma to both sex and affected limb. Because of the benignity of the tumor, the surgical treatment focused on optimizing the functional outcome. A prospective study with long term follow-up is required for this under-diagnosed tumor.

Published
2018
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49. Deciphering pathological remodelling of the human cartilage extracellular matrix in osteoarthritis at the supramolecular level

Author

Ilhem Lilia Jaabar, Pauline Cornette, Antoine Miche, Kristell Wanherdrick, Vincent Dupres, François-Paul Ehkirch, Adeline Cambon Binder, Francis Berenbaum, Xavier Houard, Jessem Landoulsi, Laboratoire de Réactivité de Surface (LRS), Institut de Chimie du CNRS (INC)-Sorbonne Université (SU)-Centre National de la Recherche Scientifique (CNRS), Pathologies articulaires associées aux maladies métaboliques et à l’âge [CRSA], Centre de Recherche Saint-Antoine (CRSA), Assistance publique - Hôpitaux de Paris (AP-HP) (AP-HP)-Institut National de la Santé et de la Recherche Médicale (INSERM)-Sorbonne Université (SU)-Assistance publique - Hôpitaux de Paris (AP-HP) (AP-HP)-Institut National de la Santé et de la Recherche Médicale (INSERM)-Sorbonne Université (SU), Centre d’Infection et d’Immunité de Lille - INSERM U 1019 - UMR 9017 - UMR 8204 (CIIL), Institut Pasteur de Lille, Réseau International des Instituts Pasteur (RIIP)-Réseau International des Instituts Pasteur (RIIP)-Institut National de la Santé et de la Recherche Médicale (INSERM)-Université de Lille-Centre Hospitalier Régional Universitaire [Lille] (CHRU Lille)-Centre National de la Recherche Scientifique (CNRS), Clinique Maussins-Nollet, 67, rue de Romainville, 75019 Paris, France, parent, CHU Saint-Antoine [AP-HP], and Assistance publique - Hôpitaux de Paris (AP-HP) (AP-HP)-Sorbonne Université (SU)

Subjects
Cartilage, Articular, Chondrocytes, [SDV]Life Sciences [q-bio], Osteoarthritis, Humans, General Materials Science, Aggrecans, Collagen, Extracellular Matrix
Abstract

The extracellular matrix (ECM) of articular cartilage is a three-dimensional network mainly constituted of entangled collagen fibrils and interfibrillar aggrecan aggregates. During the development of osteoarthritis (OA), the most common musculoskeletal disorder, the ECM is subjected to a combination of chemical and structural changes that play a pivotal role in the initiation and the progress of the disease. While the molecular mechanisms involved in the pathological remodelling of the ECM are considered as decisive, they remain, however, not completely elucidated. Herein, we report a relevant way for unravelling the role and nature of OA progress on human cartilage tissues, in terms of chemical composition and morphological and mechanical properties at the level of supramolecular assemblies constituting the cartilage ECM. For this purpose, we used X-ray photoelectron spectroscopy (XPS), and developed an innovative methodological approach that provides the molecular composition of the ECM. Moreover, we used atomic force microscopy (AFM) to probe the tissues at the level of individual collagen fibrils, both imaging and force spectroscopy modes being explored to this end. Taken together, these nanoscale characterization studies reveal the existence of two stages in the OA progress. At the early stage, a marked increase in the aggrecan and collagen content is observed, reflecting the homeostatic chondrocyte activity that tends to repair the cartilage ECM. At the late stage, we observe a failed attempt to stabilize and/or restore the tissue, yielding significant degradation of the supramolecular assemblies. This suggests an imbalance in the chondrocyte activity that turns in favor of catabolic events. Chemical changes are also accompanied by ECM structural changes and stiffening. Interestingly, we showed the possibility to mimic the imbalanced activities of chondrocytes by applying enzymatic digestions of healthy cartilage, through the combined action of hyaluronidase and collagenase. This yields damage strictly analogous to that observed at high OA severity. These findings bring mechanistic insights leading to a better understanding of the mechanism by which OA is initiated and progresses in the cartilage ECM. They offer guidelines for the development of curative treatments, such as targeting the homeostatic balance of chondrocyte metabolism through the control of enzymatic reactions involved in catabolic processes.

Published
2022
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