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A cognitive antenna array, which is designed by using the Deep Reinforcement Learning (DRL) is proposed in this paper to adapt the complex electromagnetic environment. Specifically, the phased array antenna is utilized as the manipulatable component to achieve the characteristic of beam steering with the help of the DRL algorithm. We begin by establishing a DRL-based framework, which is comprised of a microprogrammed control unit, power divider, digital phase shifter, and the patch antenna array. In the DRL algorithm, the desired beam steering is obtained through trial-and-error interactions with the environment, which is required to observe predefined rewards based on current state and action. Next, the system is trained to perform beam steering, and a set of hyper-parameters of the deep neutral network are obtained and stored for practical usage. Good agreement is achieved between the simulated and measured radiation patterns of the planar phased array antenna, which validates of the DRL-based phase distribution regulation algorithm. Finally, the algorithm is implemented in the design process of a conformal phased array antenna, and it is shown that the measured radiation performance of the array is satisfactory for different beam scan angles.

Purpose Changrui enema, a traditional Chinese medicine prescription, is used as a supplementary treatment for acute radiation proctitis (ARP). Herein we explored the inhibition effects of Changrui enema on NF-κB and VEGF in ARP mice. Methods A total of 120 C57BL/6 mice were divided randomly into normal mice group, ARP mice group, western medicine enema group (dexamethasone combined with gentamicin), and Changrui enema group. ARP mice were established by pelvic local irradiation. The expression of IL-1β, NF-κB, VEGF, AQP1, AQP3, p-ERK1/2 and p-JNK was determined by immunohistochemistry or western blot. Results The study firstly found that Changrui enema alleviated ARP mice. The expression of IL-1β, NF-κB, VEGF, AQP1 and p-ERK1/2 was increased in ARP mice, and was reserved by Changrui enema. However, the expression of AQP3 and p-JNK was decreased in ARP mice, and was up-regulated by Changrui enema. Conclusions Changrui enema is an effective treatment with fewer side effects for ARP. The mechanism of Changrui enema may be related to the inhibition of inflammation-induced angiogenesis. Changrui enema inhibits IL-1β and NF-κB expression as well as VEGF expression. Interestingly, AQP1 promotes angiogenesis, while AQP3 inhibits inflammation. Changrui enema probably inhibits AQP1 expression by down-regulating p-ERK1/2, and improves AQP3 expression by up-regulating p-JNK.