135 results on '"Carothers, Adelaide"'
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2. Perspective from Aberrant Crypt Foci in the Adenoma Prevention with Celecoxib Trial
3. Supplementary Figure Legends 1-5, Table 1 Legend from Chronic Cyclooxygenase-2 Inhibition Promotes Myofibroblast-Associated Intestinal Fibrosis
4. Supplementary Figures 1-5, Table 1 from Chronic Cyclooxygenase-2 Inhibition Promotes Myofibroblast-Associated Intestinal Fibrosis
5. Data from Chronic Cyclooxygenase-2 Inhibition Promotes Myofibroblast-Associated Intestinal Fibrosis
6. Supplementary Data from Galectin-3 Targeted Therapy with a Small Molecule Inhibitor Activates Apoptosis and Enhances Both Chemosensitivity and Radiosensitivity in Papillary Thyroid Cancer
7. Supplementary Figure Legends 1-3 from Persistent Cyclooxygenase-2 Inhibition Downregulates NF-κB, Resulting in Chronic Intestinal Inflammation in the Min/+ Mouse Model of Colon Tumorigenesis
8. Supplementary Figure 1 from Mesenchymal Stromal Cell Mutations and Wound Healing Contribute to the Etiology of Desmoid Tumors
9. Data from Persistent Cyclooxygenase-2 Inhibition Downregulates NF-κB, Resulting in Chronic Intestinal Inflammation in the Min/+ Mouse Model of Colon Tumorigenesis
10. Supplementary Table 1 from Persistent Cyclooxygenase-2 Inhibition Downregulates NF-κB, Resulting in Chronic Intestinal Inflammation in the Min/+ Mouse Model of Colon Tumorigenesis
11. Data from Mesenchymal Stromal Cell Mutations and Wound Healing Contribute to the Etiology of Desmoid Tumors
12. Supplementary Table 1 from Mesenchymal Stromal Cell Mutations and Wound Healing Contribute to the Etiology of Desmoid Tumors
13. Supplementary Table 2 from Mesenchymal Stromal Cell Mutations and Wound Healing Contribute to the Etiology of Desmoid Tumors
14. Supplementary Figures 1-3 from Persistent Cyclooxygenase-2 Inhibition Downregulates NF-κB, Resulting in Chronic Intestinal Inflammation in the Min/+ Mouse Model of Colon Tumorigenesis
15. Supplementary Figure 3 from Mesenchymal Stromal Cell Mutations and Wound Healing Contribute to the Etiology of Desmoid Tumors
16. Supplementary Figure 4 from Mesenchymal Stromal Cell Mutations and Wound Healing Contribute to the Etiology of Desmoid Tumors
17. Supplementary Figure 2 from Mesenchymal Stromal Cell Mutations and Wound Healing Contribute to the Etiology of Desmoid Tumors
18. Supplementary Figure Legends 1-4 from Mesenchymal Stromal Cell Mutations and Wound Healing Contribute to the Etiology of Desmoid Tumors
19. Characterization of Mutations Induced by 2-(N-acetoxy-N-acetyl)aminofluorene in the Dihydrofolate Reductase Gene of Cultured Hamster Cells
20. Efficient Cloning of Single-Copy Genes Using Specialized Cosmid Vectors: Isolation of Mutant Dihydrofolate Reductase Genes
21. Splicing Mutations in the CHO DHFR Gene Preferentially Induced by (±)-3α, 4β-dihydroxy-1α, 2α-epoxy-1,2,3,4- Tetrahydrobenzo [k c ] Phenanthrene
22. DNAs Strand-Specific Mutations Induced by (±)-3α,4β- Dihydroxy-1α2α-Epoxy-1,2,3,4,-Tetrahydrobenz[c]phenanthrene in the Dihydrofolate Reductase Gene
23. DNA Strand-Specific Repair of (±)-3α,4β-Dihydroxy-1α,2 α-Epoxy-1,2,3,4-Tetrahydrobenzo[c]Phenanthrene Adducts in the Hamster Dihydrofolate Reductase Gene
24. Estrogen receptor α or β loss in the colon of Min/+ mice promotes crypt expansion and impairs TGFβ and HNF3β signaling
25. Adenomatous polyposis coli truncation alters cytoskeletal structure and microtubule stability in early intestinal tumorigenesis
26. Deficient E-cadherin adhesion in C57BL/6J-Min/+ mice is associated with increased tyrosine kinase activity and RhoA-dependent actomyosin contractility
27. Modulation of tumor formation and intestinal cell migration by estrogens in the ApcMin/+ mouse model of colorectal cancer
28. DNA strand-specific repair of (+/-)-3-alpha,4-beta-dihydroxy-1-alpha, 2-alpha-epoxy-1,2,3,4-tetrahydrobenzo(c)phenanthrene adducts in the hamster dihydrofolate reductase gene
29. Mapping and characterization of mutations induced by benzo[a]pyrene diol epoxide at dihydrofolate reductase locus in CHO cells
30. Lack of anti-tumor activity with the β-catenin expression inhibitor EZN-3892 in the C57BL/6J Min/+ model of intestinal carcinogenesis
31. Estrogen receptor α or β loss in the colon of Min/+ mice promotes crypt expansion and impairs TGFβ and HNF3β signaling
32. Immunohistochemical and Molecular Analysis of Tyrosine Kinase Activity in Desmoid Tumors
33. Abstract 3304: Hyaluronan mediates interactions between CD44+ MSCs and CD34+ fibrocytes in Desmoid Tumors.
34. Abstract 1521: Expression of the tight junctions protein IGSF5 in the colorectal adenoma-carcinoma sequence
35. Mesenchymal Stromal Cell Mutations and Wound Healing Contribute to the Etiology of Desmoid Tumors
36. Dietary celecoxib treatment normalized excessive wound healing responses in Min/+ relative to Apc+/+ (WT) mice
37. Abstract 1429: Apc heterozygosity in the C57BL/6J-Min/+ mouse produces an abnormal exuberant proliferative response in excisional wound healing
38. Abstract 484: Persistent activation of mesenchymal stromal cells (MSCs) and tissue repair in the etiology of desmoid tumors (DT)
39. Abstract 2292: Absent E2 liganded AhR inhibits tumor inhibition by ERα in Min mice
40. The Mobilization and Effect of Endogenous Bone Marrow Progenitor Cells in Diabetic Wound Healing
41. Sulindac Reverses Aberrant Expression and Localization of β-Catenin in Papillary Thyroid Cancer Cells with the BRAFV600E Mutation
42. Persistent Cyclooxygenase-2 Inhibition Downregulates NF-κB, Resulting in Chronic Intestinal Inflammation in the Min/+ Mouse Model of Colon Tumorigenesis
43. Abstract 2863: Long term COX-2 inhibition in the small intestine of C57BL/6J Apc+/+ mice altered TGFβ signaling
44. Chronic Cyclooxygenase-2 Inhibition Promotes Myofibroblast-Associated Intestinal Fibrosis
45. Galectin-3 Targeted Therapy with a Small Molecule Inhibitor Activates Apoptosis and Enhances Both Chemosensitivity and Radiosensitivity in Papillary Thyroid Cancer
46. Poly-N-Acetyl Glucosamine Nanofibers
47. Abstract A31: Intestinal resistance to celecoxib in Min/+ mice involves ECM changes and an expanded population of TGFβ-expressing myofibroblasts
48. Aberrant Crypt Foci in the Adenoma Prevention with Celecoxib Trial
49. Abstract LB-242: Aberrant crypt foci (ACF) are not surrogate biomarkers of sporadic adenoma prevention with celecoxib: Results from the Adenoma Prevention with Celecoxib (APC) trial
50. Estrogen Receptors α and β Are Inhibitory Modifiers of Apc-Dependent Tumorigenesis in the Proximal Colon of Min/+ Mice
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