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1. Human genetic structure in Northwest France provides new insights into West European historical demography

2. Genome-wide association analyses identify new Brugada syndrome risk loci and highlight a new mechanism of sodium channel regulation in disease susceptibility

3. Rare Coding Variants in ANGPTL6 Are Associated with Familial Forms of Intracranial Aneurysm

4. Author Correction: Genome-wide association analyses identify new Brugada syndrome risk loci and highlight a new mechanism of sodium channel regulation in disease susceptibility

5. How local reference panels improve imputation in French populations.

6. Increased Tpeak-Tend interval is highly and independently related to arrhythmic events in Brugada syndrome

7. Prevalence and Prognostic Role of Various Conduction Disturbances in Patients With the Brugada Syndrome

8. Early Repolarization Disease

9. Genetic population structure across Brittany and the downstream Loire basin provides new insights on the demographic history of Western Europe

10. Testing the burden of rare variation in arrhythmia-susceptibility genes provides new insights into molecular diagnosis for Brugada syndrome

11. Prediction of Unruptured Intracranial Aneurysm Evolution: The UCAN Project

13. Moment estimators of relatedness from low-depth whole-genome sequencing data.

14. Integrated clinical and omics approach to rare diseases : Novel genes and oligogenic inheritance in holoprosencephaly

15. Integrated clinical and omics approach to rare diseases: Novel genes and oligogenic inheritance in holoprosencephaly

16. Biallelic Variants in UBA5 Reveal that Disruption of the UFM1 Cascade Can Result in Early-Onset Encephalopathy

18. Rare Coding Variants in ANGPTL6 Are Associated with Familial Forms of Intracranial Aneurysm

19. Variants in the SCN5A Promoter Associated With Various Arrhythmia Phenotypes

20. Biallelic Variants in UBA5 Reveal that Disruption of the UFM1 Cascade Can Result in Early-Onset Encephalopathy

21. Dysfunction of the Voltage‐Gated K + Channel β2 Subunit in a Familial Case of Brugada Syndrome

22. Variants of Transient Receptor Potential Melastatin Member 4 in Childhood Atrioventricular Block

23. Common variants at SCN5A-SCN10A and HEY2 are associated with Brugada syndrome, a rare disease with high risk of sudden cardiac death

24. Understanding the Pathophysiology of Intracranial Aneurysm: The ICAN Project.

25. Genetic association study of QT interval highlights role for calcium signaling pathways in myocardial repolarization.

26. Common variants at SCN5A-SCN10A and HEY2 are associated with Brugada syndrome, a rare disease with high risk of sudden cardiac death

27. Early repolarization disease

28. Erratum: Common variants at SCN5A-SCN10A and HEY2 are associated with Brugada syndrome, a rare disease with high risk of sudden cardiac death

29. Molecular Genetics and Functional Anomalies in a Series of 248 Brugada Cases with 11 Mutations in the TRPM4 Channel

30. Identification of Large Families in Early Repolarization Syndrome

31. Parental Electrocardiographic Screening Identifies a High Degree of Inheritance for Congenital and Childhood Nonimmune Isolated Atrioventricular Block

32. 325 Clinical presentation and long-term clinical outcomes of non immune, isolated atrioventricular block diagnosed in utero or early childhood

33. 341 Parental electrocardiographic screening identifies a high degree of inheritance for congenital and childhood non-immune isolated atrioventricular block

35. Molecular Genetics and Functional Anomalies in a Series of 248 Brugada Cases with 11 Mutations in the TRPM4 Channel.

36. Ventricular Fibrillation with Prominent Early Repolarization Associated with a Rare Variant of KCNJ8/KATP Channel.

37. Genetic association study of QT interval highlights role for calcium signaling pathways in myocardial repolarization

38. Dysfunction of the Voltage-Gated K+ Channel β2 Subunit in a Familial Case of Brugada Syndrome.

39. Heritable defects in telomere and mitotic function selectively predispose to sarcomas.

40. Common variants at SCN5A-SCN10A and HEY2 are associated with Brugada syndrome, a rare disease with high risk of sudden cardiac death.

41. Risk Stratification and Therapeutic Approach in Brugada Syndrome.

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