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6 results on '"Chciuk-Górnicka M"'

2. Influence of serotonin and selective 5-HT2 and 5-HT3 receptor agonists on the haemodynamics of the isolated, constant pressure perfused rat heart

Author

Witanowska A, Ciurzynska G, Kurenko-Deptuch M, T W Deptuch, E. Wojtecka-Lukasik, Chciuk-Górnicka M, Danuta Maslinska, Mariusz Gujski, S. Maslinski, and K Tusinska

Subjects
Male, medicine.medical_specialty, Serotonin, Immunology, Hemodynamics, 5-HT3 receptor, Internal medicine, Tachycardia, medicine, Pressure, Animals, Rats, Wistar, Receptor, 5-HT receptor, Pharmacology, biology, Chemistry, Heart, Serotonin 5-HT3 Receptor Agonists, Cardiovascular physiology, Rats, Serotonin Receptor Agonists, Perfusion, Endocrinology, biology.protein, Receptors, Serotonin, 5-HT3, Receptors, Serotonin, 5-HT2, Endogenous agonist, Serotonin 5-HT2 Receptor Agonists
Published
2005

3. The influence of intestinal ischaemia on energy balance in the myocardium during ischaemia-reperfusion induced cardiac injury in the rat

Author

Jozwicka M, Ciurzynska G, T W Deptuch, Witanowska A, M. Gajewski, Kurenko-Deptuch M, S. Maslinski, Dzierzkowska J, and Chciuk-Górnicka M

Subjects
Male, medicine.medical_specialty, Immunology, Ischemia, Endogeny, Myocardial Reperfusion Injury, chemistry.chemical_compound, INTESTINAL ISCHAEMIA, Adenosine Triphosphate, Internal medicine, medicine, Animals, Receptors, Histamine H2, cardiovascular diseases, Rats, Wistar, Receptor, Ischemic Preconditioning, Pharmacology, Cardioprotection, business.industry, Myocardium, medicine.disease, Rats, Intestines, chemistry, Histamine H2 Antagonists, Ischaemia reperfusion, Cardiology, business, Cimetidine, Energy Metabolism, Intracellular, Histamine
Abstract

Signalling pathways of cardiac ischaemic preconditioning (PC) and the final effector mechanisms involved in the process remain as yet unknown. The most likely mechanisms of “classic” preconditioning involve the release of endogenous substances from the ischaemic myocardium. Stimuli other than ischaemia of the myocardial risk territory may create cardioprotection against damage resulting from subsequent coronary artery occlusion. Gho et al. [1] demonstrated that brief renal or mesenteric ischaemia led to protection against coronary artery occlusion and suggested that a neuronal mechanism was involved. The important metabolic feature of preconditioned myocardium is decreased utilisation of ATP during ischaemia. Our previous data suggest that in the rat, histamine (H) partially contributes to ischaemiareperfusion cardiac damage [2]. The aim of our study was to examine whether intestinal preconditioning (IPC) causes changes in the intracellular ATP level of cardiomyocytes during ischaemia-reperfusion injury and to determine whether H receptors are involved in this process. Materials and methods

Published
1999

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