324 results on '"Christmann, Markus"'
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2. Identification of a highly efficient dual type I/II FMS-like tyrosine kinase inhibitor that disrupts the growth of leukemic cells
3. Expression of DNA repair genes and its relevance for DNA repair in peripheral immune cells of patients with posttraumatic stress disorder
4. p53 triggers mitochondrial apoptosis following DNA damage-dependent replication stress by the hepatotoxin methyleugenol
5. The p21CIP1-CDK4-DREAM axis is a master regulator of genotoxic stress-induced cellular senescence
6. Localization matters: nuclear-trapped Survivin sensitizes glioblastoma cells to temozolomide by elevating cellular senescence and impairing homologous recombination
7. Acetylation-dependent control of SIAH2 links JAK2V617F to mitochondrial respiration and ROS-associated apoptosis
8. Genotoxic and Cytotoxic Activity of Fisetin on Glioblastoma Cells
9. DNA repair in personalized brain cancer therapy with temozolomide and nitrosoureas
10. Epigenetic regulation of DNA repair genes and implications for tumor therapy
11. Inherent and toxicant-provoked reduction in DNA repair capacity: A key mechanism for personalized risk assessment, cancer prevention and intervention, and response to therapy
12. Functional mismatch repair and inactive p53 drive sensitization of colorectal cancer cells to irinotecan via the IAP antagonist BV6
13. Sensitization of colorectal cancer cells to irinotecan by the Survivin inhibitor LLP3 depends on XAF1 proficiency in the context of mutated p53
14. Loss of Wilms tumor 1 protein is a marker for apoptosis in response to replicative stress in leukemic cells
15. Evaluating long-term cellular effects of the arsenic species thio-DMAV: qPCR-based gene expression as screening tool
16. Data from The Mitochondrial Disruptor Devimistat (CPI-613) Synergizes with Genotoxic Anticancer Drugs in Colorectal Cancer Therapy in a Bim-Dependent Manner
17. Supplementary Data from The Mitochondrial Disruptor Devimistat (CPI-613) Synergizes with Genotoxic Anticancer Drugs in Colorectal Cancer Therapy in a Bim-Dependent Manner
18. Supplementary Table1+2 from Temozolomide Induces Senescence and Repression of DNA Repair Pathways in Glioblastoma Cells via Activation of ATR–CHK1, p21, and NF-κB
19. Supplementary Figure5 from Temozolomide Induces Senescence and Repression of DNA Repair Pathways in Glioblastoma Cells via Activation of ATR–CHK1, p21, and NF-κB
20. Supplementary Table 1, Figures 1 - 3 from Translesion Polymerase η Is Upregulated by Cancer Therapeutics and Confers Anticancer Drug Resistance
21. Supplementary Figure Legend from Translesion Polymerase η Is Upregulated by Cancer Therapeutics and Confers Anticancer Drug Resistance
22. Data from Cisplatin-Induced Apoptosis Involves Membrane Fluidification via Inhibition of NHE1 in Human Colon Cancer Cells
23. Data from Translesion Polymerase η Is Upregulated by Cancer Therapeutics and Confers Anticancer Drug Resistance
24. Supplementary Materials from Cisplatin-Induced Apoptosis Involves Membrane Fluidification via Inhibition of NHE1 in Human Colon Cancer Cells
25. Beteiligung von ATM an der DNA‐Schadensantwort ausgelöst durch das Phenylpropanoid Methyleugenol in Leberzellen
26. DNA Alkylation Damage by Nitrosamines and Relevant DNA Repair Pathways
27. Analysis of the interplay between all-trans retinoic acid and histone deacetylase inhibitors in leukemic cells
28. DNA Damage Response and the Balance Between Cell Survival and Cell Death
29. Induction of DNA Repair Genes in Mammalian Cells in Response to Genotoxic Stress
30. DNA Methylation Damage: Formation, Repair and Biological Consequences
31. Alkyltransferases
32. Abrogation of Cellular Senescence Induced by Temozolomide in Glioblastoma Cells: Search for Senolytics
33. Human three prime exonuclease TREX1 is induced by genotoxic stress and involved in protection of glioma and melanoma cells to anticancer drugs
34. Repair gene O6‐methylguanine‐DNA methyltransferase is controlled by SP1 and up‐regulated by glucocorticoids, but not by temozolomide and radiation
35. O6-methylguanine-DNA methyltransferase (MGMT): impact on cancer risk in response to tobacco smoke
36. The chloroethylating anticancer drug ACNU induces FRA1 that is involved in drug resistance of glioma cells
37. Expression of DNA-repair genes and its relevance for DNA repair in peripheral immune cells of patients with posttraumatic stress disorder
38. Expression of DNA-repair genes and its relevance for DNA repair in peripheral immune cells of patients with posttraumatic stress disorder
39. Alterations in Molecular Profiles Affecting Glioblastoma Resistance to Radiochemotherapy: Where Does the Good Go?
40. Human monocytes are severely impaired in base and DNA double-strand break repair that renders them vulnerable to oxidative stress
41. O6-Methylguanine-DNA methyltransferase (MGMT) in normal tissues and tumors: Enzyme activity, promoter methylation and immunohistochemistry
42. Senescence Is the Main Trait Induced by Temozolomide in Glioblastoma Cells
43. List of Contributors
44. DNA repair and damage response in personalized glioblastoma chemotherapy
45. Identification of senolytic drugs in temozolomide-induced senescent glioblastoma cells
46. The Mitochondrial Disruptor Devimistat (CPI-613) Synergizes with Genotoxic Anticancer Drugs in Colorectal Cancer Therapy in a Bim-Dependent Manner
47. Delayed c-Fos activation in human cells triggers XPF induction and an adaptive response to UVC-induced DNA damage and cytotoxicity
48. WRN protects against topo I but not topo II inhibitors by preventing DNA break formation
49. Targeting O6-methylguanine-DNA methyltransferase with specific inhibitors as a strategy in cancer therapy
50. Thermisch-elektrisch optimierte Luftfahrtantriebssysteme (TELOS) : Schlussbericht zu Nr. 8.2 NKBF 98 : im Förderprogramm LUFO-V2
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