176 results on '"Ciechomska, Marzena"'
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2. Impact of methotrexate treatment on vaccines immunogenicity in adult rheumatological patients – Lessons learned from the COVID-19 pandemic
3. Potential Mechanism of Fatigue Induction and Its Management by JAK Inhibitors in Inflammatory Rheumatic Diseases
4. Mechanisms involved in the recognition of apoptotic cells by B lymphocytes
5. Changes in MiRNA-5196 Expression as a Potential Biomarker of Anti-TNF-α Therapy in Rheumatoid Arthritis and Ankylosing Spondylitis Patients
6. Circulating miRNA-19b as a biomarker of disease progression and treatment response to baricitinib in rheumatoid arthritis patients through miRNA profiling of monocytes
7. The Kinetics of Humoral and Cellular Responses after the Booster Dose of COVID-19 Vaccine in Inflammatory Arthritis Patients
8. Potential Mechanism of Fatigue Induction and Its Management by JAK Inhibitors in Inflammatory Rheumatic Diseases
9. Pacjent post-COVID-owy Co już wiemy, a co przed nami? / A post-COVID patient What we know and what is ahead of us?
10. S100A8 and S100A12 Proteins as Biomarkers of High Disease Activity in Patients with Rheumatoid Arthritis That Can Be Regulated by Epigenetic Drugs
11. The kinetics of humoral and cellular responses after the booster dose of COVID-19 vaccine in inflammatory arthritis patients
12. Effectiveness of Soluble CTLA-4-Fc in the Inhibition of Bone Marrow T-Cell Activation in Context of Indoleamine 2.3-Dioxygenase (IDO) and CD4+Foxp3+ Treg Induction
13. Significance of Polymorphism and Expression of miR-146a and NFkB1 Genetic Variants in Patients with Rheumatoid Arthritis
14. Humoral and cellular immunogenicity of COVID-19 booster dose vaccination in inflammatory arthritis patients
15. Impact of Adipose-Derived Mesenchymal Stem Cells (ASCs) of Rheumatic Disease Patients on T Helper Cell Differentiation
16. Effectiveness of Soluble CTLA-4-Fc in the Inhibition of Bone Marrow T-Cell Activation in Context of Indoleamine 2.3-Dioxygenase (IDO) and CD4+Foxp3+ Treg Induction
17. 03.15 Identification of novel micrornas in monocytes from rheumatoid arthritis and systemic sclerosis patients using next generation sequencing
18. Interleukin-6, its role in fibrosing conditions
19. Circulating miRNA Correlates with Lipid Profile and Disease Activity in Psoriatic Arthritis, Rheumatoid Arthritis, and Ankylosing Spondylitis Patients
20. Effect of crystallinity and related surface properties on gene expression of primary fibroblasts
21. Histone Demethylation and Toll-like Receptor 8–Dependent Cross-Talk in Monocytes Promotes Transdifferentiation of Fibroblasts in Systemic Sclerosis Via Fra-2
22. S100A8 and S100A12 Proteins as Biomarkers of High Disease Activity in Patients with Rheumatoid Arthritis That Can Be Regulated by Epigenetic Drugs.
23. Effectiveness of Soluble CTLA-4-Fc in the Inhibition of Bone Marrow T-Cell Activation in Context of Indoleamine 2.3-Dioxygenase (IDO) and CD4+Foxp3+ Treg Induction.
24. Current frontiers in systemic sclerosis pathogenesis
25. Serum amyloid A induces interleukin-6 in dermal fibroblasts via Toll-like receptor 2, interleukin-1 receptor-associated kinase 4 and nuclear factor-κB
26. Tuning Monocytes and Macrophages for Personalized Therapy and Diagnostic Challenge in Rheumatoid Arthritis
27. Comprehensive microRNA and transcriptomic profiling of rheumatoid arthritis monocytes: role of microRNA-146b in pro-inflammatory progression
28. Antigen-specific B lymphocytes acquire proteoglycan aggrecan from cartilage extracellular matrix resulting in antigen presentation and CD4+ T-cell activation
29. Biologic Drugs for Rheumatoid Arthritis in the Context of Biosimilars, Genetics, Epigenetics and COVID-19 Treatment
30. Toll-like receptor-mediated, enhanced production of profibrotic TIMP-1 in monocytes from patients with systemic sclerosis: role of serum factors
31. Antigen-specific B cells can acquire and present the rheumatoid arthritis candidate autoantigen aggrecan from a non-internalisable surface
32. Small Molecule Inhibitors in the Treatment of Rheumatoid Arthritis and Beyond: Latest Updates and Potential Strategy for Fighting COVID-19
33. Global miRNA and mRNA expression profiles identify miRNA‐26a‐2‐3p‐dependent repression of IFN signature in systemic sclerosis human monocytes
34. DNA Methylation as a Future Therapeutic and Diagnostic Target in Rheumatoid Arthritis
35. Targeting interferons as a strategy for systemic sclerosis treatment
36. IL-13 mediates collagen deposition via STAT6 and microRNA-135b : A role for epigenetics
37. Epigenetic Modulation as a Therapeutic Prospect for Treatment of Autoimmune Rheumatic Diseases
38. The role of microRNA‐5196 in the pathogenesis of systemic sclerosis
39. CpG-specific methylation at rheumatoid arthritis diagnosis as a marker of treatment response
40. 03.15 Identification of novel micrornas in monocytes from rheumatoid arthritis and systemic sclerosis patients using next generation sequencing
41. IL-13 mediates collagen deposition via STAT6 and microRNA-135b : A role for epigenetics
42. Corrigendum:IL-13 mediates collagen deposition via STAT6 and microRNA-135b: a role for epigenetics (Scientific Reports (2016) 6 (25066) DOI: 10.1038/srep25066)
43. Corrigendum:IL-13 mediates collagen deposition via STAT6 and microRNA-135b: a role for epigenetics (Scientific Reports (2016) 6 (25066) DOI: 10.1038/srep25066)
44. IL-13 mediates collagen deposition via STAT6 and microRNA-135b: A role for epigenetics
45. Histone Demethylation and Toll-like Receptor 8-Dependent Cross-Talk in Monocytes Promotes Transdifferentiation of Fibroblasts in Systemic Sclerosis Via Fra-2
46. Correction: Corrigendum: IL-13 mediates collagen deposition via STAT6 and microRNA-135b: a role for epigenetics
47. Current frontiers in systemic sclerosis pathogenesis
48. MiR-29a Reduces TIMP-1 Production by Dermal Fibroblasts via Targeting TGF-β Activated Kinase 1 Binding Protein 1, Implications for Systemic Sclerosis
49. MiR-29a Reduces TIMP-1 Production by Dermal Fibroblasts via Targeting TGF-β Activated Kinase 1 Binding Protein 1, Implications for Systemic Sclerosis
50. The role of micro RNA-5196 in the pathogenesis of systemic sclerosis.
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