502 results on '"Ciriolo, Maria Rosa"'
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2. N-acetylaspartate promotes glycolytic-to-oxidative fiber-type switch and resistance to atrophic stimuli in myotubes
3. Effect of membrane depolarization against Aspergillus niger GM31 resistant by ultra nanoclusters characterized by Ag2+ and Ag3+ oxidation state
4. Hindering NAT8L expression in hepatocellular carcinoma increases cytosolic aspartate delivery that fosters pentose phosphate pathway and purine biosynthesis promoting cell proliferation
5. Adaptive antioxidant response to mitochondrial fatty acid oxidation determines the proliferative outcome of cancer cells
6. Contributors
7. Modulation of cancer cell proliferation by interfering with metabolic ROS production
8. Hypoxia-Induced Reactive Oxygen Species: Their Role in Cancer Resistance and Emerging Therapies to Overcome It.
9. ROS-mediated activation of p38 protects hepatocellular carcinoma cells from caspase-independent death elicited by lysosomal damage
10. Extracellular vesicles in endothelial cells: from mediators of cell-to-cell communication to cargo delivery tools
11. Label-free metabolic clustering through unsupervised pixel classification of multiparametric fluorescent images
12. Aconitase 2 sensitizes MCF-7 cells to cisplatin eliciting p53-mediated apoptosis in a ROS-dependent manner
13. Reprogrammed mitochondria: a central hub of cancer cell metabolism
14. Therapeutic targeting of replicative immortality
15. Designing a broad-spectrum integrative approach for cancer prevention and treatment
16. FoxO1 localizes to mitochondria of adipose tissue and is affected by nutrient stress
17. ROS-dependent HIF1α activation under forced lipid catabolism entails glycolysis and mitophagy as mediators of higher proliferation rate in cervical cancer cells
18. Superoxide Dismutases in Saccharomyces cerevisiae
19. Aconitase 2 inhibits the proliferation of MCF-7 cells promoting mitochondrial oxidative metabolism and ROS/FoxO1-mediated autophagic response
20. Antiproliferative and apoptosis-inducing effect of common Tunisian date seed (var. Korkobbi and Arechti) phytochemical-rich methanolic extract
21. Inhibition of JNK increases the sensitivity of hepatocellular carcinoma cells to lysosomotropic drugs via LAMP2A destabilization
22. Editorial: Hallmark of cancer: sustained proliferative signalling
23. The TCA cycle as a bridge between oncometabolism and DNA transactions in cancer
24. Antigen-Presenting Dendritic Cells Provide the Reducing Extracellular Microenvironment Required for T Lymphocyte Activation
25. Molecular Basis for Anticancer and Antiparasite Activities of Copper-Based Drugs
26. Forcing ATGL expression in hepatocarcinoma cells imposes glycolytic rewiring through PPAR-α/p300-mediated acetylation of p53
27. N-Acetylaspartate Drives Oligodendroglial Differentiation via Histone Deacetylase Activation
28. Glutamine Addiction of Cancer Cells
29. A multi-targeted approach to suppress tumor-promoting inflammation
30. Cancer prevention and therapy through the modulation of the tumor microenvironment
31. Sustained proliferation in cancer: Mechanisms and novel therapeutic targets
32. Genomic instability in human cancer: Molecular insights and opportunities for therapeutic attack and prevention through diet and nutrition
33. Immune evasion in cancer: Mechanistic basis and therapeutic strategies
34. Broad targeting of resistance to apoptosis in cancer
35. Evasion of anti-growth signaling: A key step in tumorigenesis and potential target for treatment and prophylaxis by natural compounds
36. Broad targeting of angiogenesis for cancer prevention and therapy
37. Mitochondrial dysfunctions in cancer: Genetic defects and oncogenic signaling impinging on TCA cycle activity
38. Data from Glutamine Deprivation Enhances Antitumor Activity of 3-Bromopyruvate through the Stabilization of Monocarboxylate Transporter-1
39. Supplementary Figure 4 from Glutamine Deprivation Enhances Antitumor Activity of 3-Bromopyruvate through the Stabilization of Monocarboxylate Transporter-1
40. Supplementary Figure 9 from Glutamine Deprivation Enhances Antitumor Activity of 3-Bromopyruvate through the Stabilization of Monocarboxylate Transporter-1
41. Supplementary Figure 1 from Glutamine Deprivation Enhances Antitumor Activity of 3-Bromopyruvate through the Stabilization of Monocarboxylate Transporter-1
42. Supplementary Figure Legends 1-9, Methods from Glutamine Deprivation Enhances Antitumor Activity of 3-Bromopyruvate through the Stabilization of Monocarboxylate Transporter-1
43. Supplementary Figure 7 from Glutamine Deprivation Enhances Antitumor Activity of 3-Bromopyruvate through the Stabilization of Monocarboxylate Transporter-1
44. Supplementary Figure 2 from Glutamine Deprivation Enhances Antitumor Activity of 3-Bromopyruvate through the Stabilization of Monocarboxylate Transporter-1
45. Supplementary Figure 3 from Glutamine Deprivation Enhances Antitumor Activity of 3-Bromopyruvate through the Stabilization of Monocarboxylate Transporter-1
46. Supplementary Figure 5 from Glutamine Deprivation Enhances Antitumor Activity of 3-Bromopyruvate through the Stabilization of Monocarboxylate Transporter-1
47. Supplementary Figure 8 from Glutamine Deprivation Enhances Antitumor Activity of 3-Bromopyruvate through the Stabilization of Monocarboxylate Transporter-1
48. Supplementary Figure 6 from Glutamine Deprivation Enhances Antitumor Activity of 3-Bromopyruvate through the Stabilization of Monocarboxylate Transporter-1
49. Managing lipid metabolism in proliferating cells: New perspective for metformin usage in cancer therapy
50. Aberrations of the TCA Cycle in Cancer
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