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1. Hemostatic effects of two desogestrel-containing combined oral contraceptive regimens: a multinational, multicenter, randomized, open-label study.

2. What role for Qlaira in contraception?

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3. Estradiol + dienogest. Oral contraception: estradiol does not provide a therapeutic advantage.

4. Effects on serum hormone levels of low-dose estrogen in place of placebo during the hormone-free interval of an oral contraceptive.

5. Oral contraceptive update: new agents and regimens.

6. Oral contraceptives and libido in women.

7. Divergent effects of weight reduction and oral anticonception treatment on adrenergic lipolysis regulation in obese women with the polycystic ovary syndrome.

8. Comparison of the effects of triphasic oral contraceptives with desogestrel or levonorgestrel on apolipoprotein A-I-containing high-density lipoprotein particles.

9. Behavioral and neurobiological effects of estrogen replacement therapy and a history of triphasic oral contraceptive exposure.

10. Biochemical effects in women following one year's exposure to a new triphasic contraceptive--II. coagulation profiles.

11. Biochemical effects in women following one year's exposure to a new triphasic contraceptive--I. chemistry profiles.

12. Effects of oral contraceptives on plasma neutral amino acids and cholesterol during a menstrual cycle.

13. Efficacy and safety of a monophasic and a triphasic oral contraceptive containing norgestimate.

14. A clinical comparison of two triphasic oral contraceptives with levonorgestrel or norethindrone: a prospective, randomized, single-blind study.

15. The effect of a biphasic desogestrel-containing oral contraceptive on carbohydrate metabolism and various hormonal parameters.

16. Effect on lipid metabolism of a biphasic desogestrel-containing oral contraceptive: divergent changes in apolipoprotein B and E and transitory decrease in Lp(a) levels.

17. [Comparative evaluation of the effect of mono-, bi- and three-phase oral contraceptive tablets on the endometrium].

18. Influence of oral contraceptives on integrated secretion of gonadotropins.

19. Influence of triphasic oral contraceptives on blood rheology and hemostatic and metabolic patterns in young women. Results of a three-year study.

20. [The effect of oral ovulation inhibiting (mono-, bi- and triphase) contraceptives and of Continuin and Postinor on proteo- and steroid hormones].

21. The effect of follicular growth and luteal function of "missing the pill". A comparison between a monophasic and a triphasic combined oral contraceptive.

22. The effects of different formulations of oral contraceptive agents on lipid and carbohydrate metabolism.

23. Attenuation of mild hyperandrogenic activity in postpubertal acne by a triphasic oral contraceptive containing low doses of ethynyl estradiol and d,l-norgestrel.

24. [Hormonal contraception using estroprogestins].

25. [The action of Sequostat in comparison to Sequence Ovosiston on selected metabolic parameters].

26. Longitudinal ultrasonographic study of the ovarian suppressive activity of a low-dose triphasic oral contraceptive during correct and incorrect pill intake.

27. Immune reactivity of women on hormonal contraceptives: dinitrochlorobenzene sensitization test and skin reactivity to irritants.

28. Glucose and insulin levels after six months of treatment with a triphasic oral contraceptive containing ethinyl estradiol and norethindrone.

29. [Triquilar/Trinordiol. A new pill with 3 sequences].

30. [3-phase-usage contraceptives].

31. Carbohydrate studies in women using a norethindrone triphasic oral contraceptive for eighteen months.

32. The effect of oral contraceptives on vitamin B12 metabolism.

34. [Gynecological, hematological and metabolic studies during therapy with an estrogen-gestagen combination preparation].

35. Elevation of serum prolactin during application of oral contraceptives.

36. Immune reactivity of women on hormonal contraceptives. Phytohemagglutinin and concanavalin-A induced lymphocyte response.

37. [Contraceptive efficiency of triphasic inhibitors (author's transl)].

38. Progestational potency of oral contraceptives.

39. Study design for the Triphasic Randomized Clinical Trial.

40. Comparison of ethinylestradiol and mestranol in sequential-type oral contraceptives in their effects on blood glucose and serum insulin in oral glucose tolerance tests.

41. Potencies of oral contraceptives.

42. Ultrastructure of cervical mucus and sperm penetration during use of a triphasic oral contraceptive.

43. Dinitrochlorobenzene sensitisation test in women on hormonal contraceptives.

44. Studies on liver function under the influence of oral contraceptives.

45. Enhanced platelet aggregation by oral contraceptives: effect of PG synthetase inhibitors.

46. Evaluation of the clinical performance of three triphasic oral contraceptives: a multicenter, randomized comparative trial.

47. Effects of three low-dose oral contraceptive formulations on lipid metabolism.

48. Spermagglutinating antibodies and sperm penetration of cervical mucus from infertile women with spermagglutinating antibodies in serum.

49. [Effect of the steroid sex hormones on the LH and FSH responses to LHRH in the normal subject].

50. Does progestogen reduction in oral contraception parallel reduced lipid metabolic effects?