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Your search keyword '"Courtois, Michael R."' showing total 21 results
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21 results on '"Courtois, Michael R."'

3. Test Anxiety and Breadth of Encoding Experiences in Free Recall.

Author

Mueller, John H. and Courtois, Michael R.

Abstract

Rating-related dimensions led to narrow encodings, thus hindering low-anxiety subjects who normally encode broadly. Even explicit ratings of unrelated dimensions did not induce high-anxiety subjects to encode more broadly, which may indicate inflexibility in deploying memory strategies. (Author)

Published
1980

8. Rescue of Cardiomyopathy in Peroxisome Proliferator-Activated Receptor-α Transgenic Mice by Deletion of Lipoprotein Lipase Identifies Sources of Cardiac Lipids and Peroxisome Proliferator-Activated Receptor-α Activators

Author

Duncan, Jennifer G., primary, Bharadwaj, Kalyani G., additional, Fong, Juliet L., additional, Mitra, Riddhi, additional, Sambandam, Nandakumar, additional, Courtois, Michael R., additional, Lavine, Kory J., additional, Goldberg, Ira J., additional, and Kelly, Daniel P., additional

Published
2010
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16. Rescue of cardiomyopathy in peroxisome proliferator-activated receptor-alpha transgenic mice by deletion of lipoprotein lipase identifies sources of cardiac lipids and peroxisome proliferator-activated receptor-alpha activators.

Author

Duncan JG, Bharadwaj KG, Fong JL, Mitra R, Sambandam N, Courtois MR, Lavine KJ, Goldberg IJ, Kelly DP, Duncan, Jennifer G, Bharadwaj, Kalyani G, Fong, Juliet L, Mitra, Riddhi, Sambandam, Nandakumar, Courtois, Michael R, Lavine, Kory J, Goldberg, Ira J, and Kelly, Daniel P

Published
2010
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18. Diastolic stiffening induced by acute myocardial infarction is reduced by early reperfusion

Author

Kurnik, Peter B., Courtois, Michael R., and Ludbrook, Philip A.

Abstract

Reperfusion early during myocardial infarction improves ejection fraction and this improvement may represent myocardial salvage in the injured segment. Alternatively, reperfusion of injured myocardium may cause intramyocardial hemorrhage with resultant increased stiffness causing a dyskinetic segment to become akinetic, thus improving ejection fraction without concomitant myocardial salvage. To evaluate this possibility, diastolic stiffness was assessed in a closed chest, anesthetized, normothermic dog model immediately after a 1 or 3 h occlusion of the left anterior descending coronary artery and during the 4 weeks after occlusion.

Published
1988
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19. Sensitivity of isovolumic relaxation to hypothermia during myocardial infarction

Author

Courtois, Michael R, Kurnik, Peter B, and Ludbrook, Philip A

Abstract

Effects of moderate spontaneous hypothermia on left ventricular systolic and diastolic function during acute myocardial infarction were documented in 17 anesthetized dogs with micromanometric pressure and ventriculographic dimension recordings acquired at baseline and at 1 and 3 h after coronary occlusion. In Group 1 (n = 5), core temperature was allowed to decline spontaneously. In Groups 2 (n = 6) and 3 (n = 6), core temperature was maintained at normothermic levels. Hypothermia impaired isovolumic relaxation markedly despite its lack of effect on ventricular volumes or ejection fraction. At 32.3 °C, τ1/2, defined as the time needed for the left ventricular pressure at the time of peak negative rate of change of left ventricular pressure (dPdt) to fall by 50%, was increased by 129% 3 h after occlusion. In addition, at this temperature significant changes were found in heart rate, cardiac output, minute work, peak positive and peak negative dPdt, systolic ejection time, mean velocity of circumferential fiber shortening, mean aortic pressure and end-diastolic pressure.

Published
1988
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