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3. Rewiring cancer drivers to activate apoptosis

5. Loss of the neural-specific BAF subunit ACTL6B relieves repression of early response genes and causes recessive autism

7. Small Molecule Targeting of Specific BAF (mSWI/SNF) Complexes for HIV Latency Reversal

9. Rescue of deficits by Brwd1 copy number restoration in the Ts65Dn mouse model of Down syndrome

10. The CD8+ T cell tolerance checkpoint triggers a distinct differentiation state defined by protein translation defects

13. TOP2 synergizes with BAF chromatin remodeling for both resolution and formation of facultative heterochromatin

16. Large-scale animal model study uncovers altered brain pH and lactate levels as a transdiagnostic endophenotype of neuropsychiatric disorders involving cognitive impairment

18. Large-scale animal model study uncovers altered brain pH and lactate levels as a transdiagnostic endophenotype of neuropsychiatric disorders involving cognitive impairment

19. Author Response: Large-scale animal model study uncovers altered brain pH and lactate levels as a transdiagnostic endophenotype of neuropsychiatric disorders involving cognitive impairment

22. Chromatin regulators mediate anthracycline sensitivity in breast cancer

23. The neuron-specific chromatin regulatory subunit BAF53b is necessary for synaptic plasticity and memory

24. Medulloblastoma exome sequencing uncovers subtype-specific somatic mutations.

31. Borrowing Transcriptional Kinases to Activate Apoptosis

44. Supplementary Figure 2 from A CRISPR/Cas9-Engineered ARID1A-Deficient Human Gastric Cancer Organoid Model Reveals Essential and Nonessential Modes of Oncogenic Transformation

45. Supplementary Figure 5 from A CRISPR/Cas9-Engineered ARID1A-Deficient Human Gastric Cancer Organoid Model Reveals Essential and Nonessential Modes of Oncogenic Transformation

46. Supplementary Figure 4 from A CRISPR/Cas9-Engineered ARID1A-Deficient Human Gastric Cancer Organoid Model Reveals Essential and Nonessential Modes of Oncogenic Transformation

47. Data from A CRISPR/Cas9-Engineered ARID1A-Deficient Human Gastric Cancer Organoid Model Reveals Essential and Nonessential Modes of Oncogenic Transformation

48. Supplementary Tables from A CRISPR/Cas9-Engineered ARID1A-Deficient Human Gastric Cancer Organoid Model Reveals Essential and Nonessential Modes of Oncogenic Transformation

49. Supplementary Figure 1 from A CRISPR/Cas9-Engineered ARID1A-Deficient Human Gastric Cancer Organoid Model Reveals Essential and Nonessential Modes of Oncogenic Transformation

50. Supplementary Figure 3 from A CRISPR/Cas9-Engineered ARID1A-Deficient Human Gastric Cancer Organoid Model Reveals Essential and Nonessential Modes of Oncogenic Transformation

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