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Your search keyword '"D'Armiento, F.P."' showing total 27 results
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27 results on '"D'Armiento, F.P."'

1. P301 Non-familial small bowel carcinomas in Crohnʼs disease: clinico-pathological, molecular and prognostic features

Author

Di Sabatino, A., Vanoli, A., Furlan, D., Giuffrida, P., Klersy, C., Grillo, F., Fiocca, R., Mescoli, C., Rugge, M., Nesi, G., Fociani, P., Sampietro, G., Ardizzone, S., Luinetti, O., Calabrò, A., Tonelli, F., Volta, U., Santini, D., Caio, G., Elli, L., Ferrero, S., Latella, G., Ciardi, A., Solina, G., Rizzo, A., Ciacci, C., DʼArmiento, F.P., Salemme, M., Villanacci, V., Cannizzaro, R., Canzonieri, V., Reggiani Bonetti, L., Biancone, L., Monteleone, G., Orlandi, A., Santeusanio, G., Macciomei, M.C., DʼIncà, R., Perfetti, V., Sandri, G., Silano, M., Florena, A.M., Giannone, A.G., Papi, C., Coppola, L., Usai, P., Maccioni, A., Astegiano, M., Migliora, P., Manca, R., Martino, M., Trapani, D., Cerutti, R., Alberizzi, P., Riboni, R., Sessa, F., Paulli, M., Solcia, E., and Corazza, G.R.

Published
2017
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7. OC.11.1 PROGRAMMED CELL DEATH LIGAND-1 (PD-L1) EXPRESSION IS COUPLED WITH MICROSATELLITE INSTABILITY IN NON-FAMILIAL SMALL BOWEL CARCINOMAS ASSOCIATED OR NOT WITH COELIAC DISEASE OR CROHN'S DISEASE: RESULTS FROM THE SMALL BOWEL CANCER ITALIAN CONSORTIUM

Author

Vanoli, A., primary, Giuffrida, P., additional, Martino, M., additional, Persichella, A., additional, Viglio, A., additional, Furlan, D., additional, Klersy, C., additional, Grillo, F., additional, Fiocca, R., additional, Mescoli, C., additional, Rugge, M., additional, Nesi, G., additional, Fociani, P., additional, Sampietro, G., additional, Ardizzone, S., additional, Luinetti, O., additional, Calabrò, A., additional, Tonelli, F., additional, Volta, U., additional, Santini, D., additional, Caio, G., additional, Elli, L., additional, Ferrero, S., additional, Latella, G., additional, Ciardi, A., additional, Caronna, R., additional, Solina, G., additional, Rizzo, A.G., additional, Ciacci, C., additional, D'Armiento, F.P., additional, Salemme, M., additional, Villanacci, V., additional, Cannizzaro, R., additional, Canzonieri, V., additional, Bonetti, L. Reggiani, additional, Biancone, L., additional, Monteleone, G., additional, Orlandi, A., additional, Santeusanio, G., additional, Macciomei, M., additional, D'Incà, R., additional, Perfetti, V., additional, Sandri, G., additional, Silano, M., additional, Florena, A., additional, Giannone, A., additional, Papi, C., additional, Coppola, L., additional, Usai, P., additional, Maccioni, A., additional, Astegiano, M., additional, Migliora, P., additional, Sessa, F., additional, Solcia, E., additional, Corazza, G.R., additional, Paulli, M., additional, and Di Sabatino, A., additional

Published
2018
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8. OC.10.4: Survival of Non-Familial Small Bowel Carcinomas Depends on the Underlying Intestinal Immune-Mediated Disorder

Author

Di Sabatino, A., primary, Vanoli, A., additional, Furlan, D., additional, Giuffrida, P., additional, Klersy, C., additional, Grillo, F., additional, Fiocca, R., additional, Mescoli, C., additional, Rugge, M., additional, Nesi, G., additional, Fociani, P., additional, Sampietro, G.M., additional, Ardizzone, S., additional, Luinetti, O., additional, Calabrò, A., additional, Tonelli, F., additional, Volta, U., additional, Santini, D., additional, Elli, L., additional, Ferrero, S., additional, Latella, G., additional, Ciardi, A., additional, Solina, G., additional, Rizzo, A., additional, Ciacci, C., additional, D’Armiento, F.P., additional, Villanacci, V., additional, Cannizzaro, R., additional, Canzonieri, V., additional, Reggiani Bonetti, L., additional, Biancone, L., additional, Monteleone, G., additional, Orlandi, A., additional, Santeusanio, G., additional, Macciomei, M.C., additional, D’Incà, R., additional, Perfetti, V., additional, Sandri, G., additional, Silano, M., additional, Giannone, A.G., additional, Papi, C., additional, Coppola, L., additional, Usai, P., additional, Maccioni, A., additional, Astegiano, M., additional, Migliora, P., additional, Manca, R., additional, Martino, M., additional, Trapani, D., additional, Cerutti, R., additional, Alberizzi, P., additional, Riboni, R., additional, Sessa, F., additional, Paulli, M., additional, Solcia, E., additional, and Corazza, G.R., additional

Published
2017
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19. Occurrence of the same peroxidative compounds in low density lipoprotein and in atherosclerotic lesions from a homozygous familial hypercholesterolemic patient: a case report

Author

Napoli, C, primary, D'Armiento, F.P, additional, Corso, G, additional, Ambrosio, G, additional, Palumbo, G, additional, Zuliani, P, additional, Malorni, A, additional, Gallotta, G, additional, Somma, S, additional, Postiglione, A, additional, and Chiariello, M, additional

Published
1997
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21. Cerebellar ataxia and hypogonadism

Author

De Michele, G., primary, Filla, A., additional, D'Armiento, F.P., additional, Striano, S., additional, Barbieri, F., additional, Salvatore, G., additional, Di Carlo, V., additional, and Mansi, D., additional

Published
1990
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23. Decreased low-density lipoprotein oxidation after repeated selective apheresis in homozygous familial hypercholesterolemia

Author

Clinical, the ^bDepartment of, Medicine, Experimental, Cellular, the ^cDepartment of, From the ^aDivision of Cardiology, Molecular B, Napoli, C., Ambrosio, G., Scarpato, N., Corso, G., Palumbo, G., D'Armiento, F.P., Mancini, F.P., Malorni, A., Formisano, S., Ruocco, A., Cali, A., and Chiariello, M.

Abstract

Familial hypercholesterolemia was the first genetic disorder recognized to cause myocardial infarction. Patients with homozygous familial hypercholesterolemia have rapidly progressive coronary atherosclerosis with angina pectoris, myocardial infarction, or sudden death at a young age. Selective apheresis on dextran sulfate cellulose columns reduces mortality and may induce regression of coronary lesions. These patients have both increased levels and prolonged circulation residence time of low-density lipoprotein (LDL), which is not removed by cellular receptor. LDL oxidation may play a pivotal role in atherogenesis. LDL undergoes oxidation before being taken up by macrophages and then transformed into arterial wall foam cells. The aim of this study was to investigate LDL oxidation in eight homozygous patients with familial hypercholesterolemia during repeated LDL apheresis. LDL lipid peroxidation, estimated by conjugated-diene absorbance at 234 nm, lipid peroxides, and malondialdehyde showed an increased resistance against oxidation after repeated LDL apheresis. This phenomenon was also observed in the oxidative indexes of protein moiety of LDL (apolipoprotein-B "1"0"0 fragmentation, trinitrobenzenesulfonic acid reactivity, and electrophoresis agarose mobility). Similarly, cholesteryl esterification was decreased after LDL apheresis. Thus selective LDL apheresis not only decreases the pool of LDL, but it also induces changes that render LDL less susceptible to oxidation. This phenomenon might contribute to reduce coronary atherosclerosis and thus mortality of these particular patients. (Am Heart J 1997;133:585-95.)

Published
1997
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27. Small Bowel Carcinomas in Coeliac or Crohn’s Disease: Clinico-pathological, Molecular, and Prognostic Features. A Study From the Small Bowel Cancer Italian Consortium

Author

Claudia Mescoli, Michele Martino, Fausto Sessa, Massimo Rugge, Ombretta Luinetti, Gabriella Nesi, Vincenzo Canzonieri, Alessandro Vanoli, Luca Reggiani Bonetti, Ada Maria Florena, Giovanni Monteleone, Daniela Furlan, Umberto Volta, Paolo Fociani, Vincenzo Villanacci, Antonino Giulio Giannone, Marco Silano, Antonio Di Sabatino, Antonio Maccioni, Paolo Usai, G. Solina, Vittorio Perfetti, Federica Grillo, Maria Cristina Macciomei, Rachele Manca, Stefano Ferrero, Renato Cannizzaro, Aroldo Rizzo, Livia Biancone, Luca Elli, Claudio Papi, Giacomo Caio, Giovanni Latella, Antonio Calabrò, Roberta Cerutti, Marianna Salemme, Paolo Giuffrida, Gianluca M. Sampietro, Gino Roberto Corazza, Paola Migliora, Donatella Santini, Sandro Ardizzone, Augusto Orlandi, Francesco Tonelli, Antonio Ciardi, Catherine Klersy, Carolina Ciacci, Davide Trapani, Renata D'Incà, Francesco Paolo D'Armiento, Marco Paulli, Marco Astegiano, Roberto Caronna, Roberto Fiocca, Luigi Michele Coppola, Paola Alberizzi, Enrico Solcia, Giuseppe Santeusanio, G. Sandri, Roberta Riboni, Vanoli, Alessandro, Sabatino, Antonio Di, Furlan, Daniela, Klersy, Catherine, Grillo, Federica, Fiocca, Roberto, Mescoli, Claudia, Rugge, Massimo, Nesi, Gabriella, Fociani, Paolo, Sampietro, Gianluca, Ardizzone, Sandro, Luinetti, Ombretta, Calabrò, Antonio, Tonelli, Francesco, Volta, Umberto, Santini, Donatella, Caio, Giacomo, Giuffrida, Paolo, Elli, Luca, Ferrero, Stefano, Latella, Giovanni, Ciardi, Antonio, Caronna, Roberto, Solina, Gaspare, Rizzo, Aroldo, Ciacci, Carolina, D'Armiento, FRANCESCO PAOLO, Salemme, Marianna, Villanacci, Vincenzo, Cannizzaro, Renato, Canzonieri, Vincenzo, Bonetti, Luca Reggiani, Biancone, Livia, Monteleone, Giovanni, Orlandi, Augusto, Santeusanio, Giuseppe, Macciomei, Maria C, D'Incà, Renata, Perfetti, Vittorio, Sandri, Giancarlo, Silano, Marco, Florena, Ada M, Giannone, Antonino G, Papi, Claudio, Coppola, Luigi, Usai, Paolo, Maccioni, Antonio, Astegiano, Marco, Migliora, Paola, Manca, Rachele, Martino, Michele, Trapani, Davide, Cerutti, Roberta, Alberizzi, Paola, Riboni, Roberta, Sessa, Fausto, Paulli, Marco, Solcia, Enrico, Corazza, Gino R., Di Sabatino, Antonio, D’Armiento, Francesco P., Reggiani Bonetti, Luca, Macciomei, Maria C., D’Incà, Renata, Florena, Ada M., Giannone, Antonino G., Vanoli A., Di Sabatino A., Furlan D., Klersy C., Grillo F., Fiocca R., Mescoli C., Rugge M., Nesi G., Fociani P., Sampietro G., Ardizzone S., Luinetti O., Calabro A., Tonelli F., Volta U., Santini D., Caio G., Giuffrida P., Elli L., Ferrero S., Latella G., Ciardi A., Caronna R., Solina G., Rizzo A., Ciacci C., D'Armiento F.P., Salemme M., Villanacci V., Cannizzaro R., Canzonieri V., Bonetti L.R., Biancone L., Monteleone G., Orlandi A., Santeusanio G., Macciomei M.C., D'Inca R., Perfetti V., Sandri G., Silano M., Florena A.M., Giannone A.G., Papi C., Coppola L., Usai P., Maccioni A., Astegiano M., Migliora P., Manca R., Martino M., Trapani D., Cerutti R., Alberizzi P., Riboni R., Sessa F., Paulli M., Solcia E., and Corazza G.R.

Subjects
Male, 0301 basic medicine, Neuroblastoma RAS viral oncogene homolog, Oncology, Survival, Receptor, ErbB-2, Colorectal cancer, medicine.disease_cause, Inflammatory bowel disease, tumour-infiltrating lymphocyte, ErbB-2, 0302 clinical medicine, Crohn Disease, Retrospective Studie, Risk Factors, 80 and over, Child, Class I Phosphatidylinositol 3-Kinase, Aged, 80 and over, Colonic Neoplasm, Settore MED/12 - Gastroenterologia, Crohn's disease, MLH1 methylation, Tumour-infiltrating lymphocytes, Gastroenterology, General Medicine, Middle Aged, Prognosis, Microsatellite instability, MLH1 promoter methylation, 030220 oncology & carcinogenesis, Colonic Neoplasms, Survival Analysi, KRAS, Human, Receptor, Adult, Proto-Oncogene Proteins B-raf, medicine.medical_specialty, Prognosi, Class I Phosphatidylinositol 3-Kinases, Settore MED/08 - Anatomia Patologica, NO, Proto-Oncogene Proteins p21(ras), Young Adult, 03 medical and health sciences, Internal medicine, medicine, Carcinoma, Humans, inflammatory bowel disease, microsatellite instability, survival, tumour-infiltrating lymphocytes, neoplasms, Aged, Retrospective Studies, Celiac Disease, Microsatellite Instability, Survival Analysis, Tumor Suppressor Protein p53, business.industry, Tumour-infiltrating lymphocyte, Risk Factor, Cancer, medicine.disease, eye diseases, digestive system diseases, 030104 developmental biology, business
Abstract

Background and aims An increased risk of small bowel carcinoma [SBC] has been reported in coeliac disease [CD] and Crohn's disease [CrD]. We explored clinico-pathological, molecular, and prognostic features of CD-associated SBC [CD-SBC] and CrD-associated SBC [CrD-SBC] in comparison with sporadic SBC [spo-SBC]. Methods A total of 76 patients undergoing surgical resection for non-familial SBC [26 CD-SBC, 25 CrD-SBC, 25 spo-SBC] were retrospectively enrolled to investigate patients' survival and histological and molecular features including microsatellite instability [MSI] and KRAS/NRAS, BRAF, PIK3CA, TP53, HER2 gene alterations. Results CD-SBC showed a significantly better sex-, age-, and stage-adjusted overall and cancer-specific survival than CrD-SBC, whereas no significant difference was found between spo-SBC and either CD-SBC or CrD-SBC. CD-SBC exhibited a significantly higher rate of MSI and median tumour-infiltrating lymphocytes [TIL] than CrD-SBC and spo-SBC. Among the whole SBC series, both MSI─which was the result of MLH1 promoter methylation in all but one cases─and high TIL density were associated with improved survival at univariable and stage-inclusive multivariable analysis. However, only TILs retained prognostic power when clinical subgroups were added to the multivariable model. KRAS mutation and HER2 amplification were detected in 30% and 7% of cases, respectively, without prognostic implications. Conclusions In comparison with CrD-SBC, CD-SBC patients harbour MSI and high TILs more frequently and show better outcome. This seems mainly due to their higher TIL density, which at multivariable analysis showed an independent prognostic value. MSI/TIL status, KRAS mutations and HER2 amplification might help in stratifying patients for targeted anti-cancer therapy.

Published
2017

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