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2. Disruption of Astrocyte-Dependent Dopamine Control in the Developing Medial Prefrontal Cortex Leads to Excessive Grooming in Mice

3. Tau promotes oxidative stress-associated cycling neurons in S phase as a pro-survival mechanism: Possible implication for Alzheimer’s disease

6. Neuronal A2A receptor exacerbates synapse loss and memory deficits in APP/PS1 mice.

9. Tau accumulation in astrocytes of the dentate gyrus induces neuronal dysfunction and memory deficits in Alzheimer’s disease

11. Glucose metabolism links astroglial mitochondria to cannabinoid effects

12. Dysfunction of homeostatic control of dopamine by astrocytes in the developing prefrontal cortex leads to cognitive impairments

14. Tau Transfer via Extracellular Vesicles Disturbs the Astrocytic Mitochondrial System

19. Accumulation of Tau in Extracellular Vesicles Disturbs the Astrocytic Mitochondrial System

25. List of Contributors

27. Extracellular vesicles: major actors of heterogeneity in tau spreading among human tauopathies

28. Thrombolysis by PLAT/tPA increases serum free IGF1 leading to a decrease of deleterious autophagy following brain ischemia

31. Human Induced Pluripotent Stem Cell-Derived Astrocytes Are Differentially Activated by Multiple Sclerosis-Associated Cytokines

32. Thrombolysis by PLAT/tPA increases serum free IGF1 leading to a decrease of deleterious autophagy following brain ischemia.

37. Astrocytic VMAT2 in the developing prefrontal cortex is required for normal grooming behavior in mice

49. Maximizing lentiviral vector gene transfer in the CNS

50. Efficacy of THN201, a Combination of Donepezil and Mefloquine, to Reverse Neurocognitive Deficits in Alzheimer’s Disease

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