413 results on '"D’Silva, Nisha J."'
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2. Clinical, morphologic and molecular heterogeneity of HPV-associated oropharyngeal cancer
3. Salivary microbiome changes distinguish response to chemoradiotherapy in patients with oral cancer
4. Neural Influence on Cancer Invasion and Metastasis
5. Cytokines secreted by inflamed oral mucosa: implications for oral cancer progression
6. Roadmap for the Emerging Field of Cancer Neuroscience
7. Galanin mediates tumor-induced immunosuppression in head and neck squamous cell carcinoma
8. Influence of tumor cell-derived TGF-β on macrophage phenotype and macrophage-mediated tumor cell invasion
9. Neuroimmune cell interactions and chronic infections in oral cancers.
10. Radiation resistance in head and neck squamous cell carcinoma: dire need for an appropriate sensitizer
11. Supplementary Fig 6 from Whole-genome CpG-resolution DNA Methylation Profiling of HNSCC Reveals Distinct Mechanisms of Carcinogenesis for Fine-scale HPV+ Cancer Subtypes
12. Supplementary Table S2 from Whole-genome CpG-resolution DNA Methylation Profiling of HNSCC Reveals Distinct Mechanisms of Carcinogenesis for Fine-scale HPV+ Cancer Subtypes
13. FIGURE 1 from Whole-genome CpG-resolution DNA Methylation Profiling of HNSCC Reveals Distinct Mechanisms of Carcinogenesis for Fine-scale HPV+ Cancer Subtypes
14. Supplementary Fig 1 from Whole-genome CpG-resolution DNA Methylation Profiling of HNSCC Reveals Distinct Mechanisms of Carcinogenesis for Fine-scale HPV+ Cancer Subtypes
15. FIGURE 4 from Whole-genome CpG-resolution DNA Methylation Profiling of HNSCC Reveals Distinct Mechanisms of Carcinogenesis for Fine-scale HPV+ Cancer Subtypes
16. TABLE 2 from Whole-genome CpG-resolution DNA Methylation Profiling of HNSCC Reveals Distinct Mechanisms of Carcinogenesis for Fine-scale HPV+ Cancer Subtypes
17. Supplementary Fig 5 from Whole-genome CpG-resolution DNA Methylation Profiling of HNSCC Reveals Distinct Mechanisms of Carcinogenesis for Fine-scale HPV+ Cancer Subtypes
18. Supplementary Fig 3 from Whole-genome CpG-resolution DNA Methylation Profiling of HNSCC Reveals Distinct Mechanisms of Carcinogenesis for Fine-scale HPV+ Cancer Subtypes
19. Supplementary Fig 4 from Whole-genome CpG-resolution DNA Methylation Profiling of HNSCC Reveals Distinct Mechanisms of Carcinogenesis for Fine-scale HPV+ Cancer Subtypes
20. Data from Whole-genome CpG-resolution DNA Methylation Profiling of HNSCC Reveals Distinct Mechanisms of Carcinogenesis for Fine-scale HPV+ Cancer Subtypes
21. FIGURE 2 from Whole-genome CpG-resolution DNA Methylation Profiling of HNSCC Reveals Distinct Mechanisms of Carcinogenesis for Fine-scale HPV+ Cancer Subtypes
22. Supplementary Fig 2 from Whole-genome CpG-resolution DNA Methylation Profiling of HNSCC Reveals Distinct Mechanisms of Carcinogenesis for Fine-scale HPV+ Cancer Subtypes
23. Supplementary Results from Whole-genome CpG-resolution DNA Methylation Profiling of HNSCC Reveals Distinct Mechanisms of Carcinogenesis for Fine-scale HPV+ Cancer Subtypes
24. FIGURE 3 from Whole-genome CpG-resolution DNA Methylation Profiling of HNSCC Reveals Distinct Mechanisms of Carcinogenesis for Fine-scale HPV+ Cancer Subtypes
25. TABLE 1 from Whole-genome CpG-resolution DNA Methylation Profiling of HNSCC Reveals Distinct Mechanisms of Carcinogenesis for Fine-scale HPV+ Cancer Subtypes
26. FIGURE 6 from Whole-genome CpG-resolution DNA Methylation Profiling of HNSCC Reveals Distinct Mechanisms of Carcinogenesis for Fine-scale HPV+ Cancer Subtypes
27. FIGURE 5 from Whole-genome CpG-resolution DNA Methylation Profiling of HNSCC Reveals Distinct Mechanisms of Carcinogenesis for Fine-scale HPV+ Cancer Subtypes
28. Supplementary Fig 8 from Whole-genome CpG-resolution DNA Methylation Profiling of HNSCC Reveals Distinct Mechanisms of Carcinogenesis for Fine-scale HPV+ Cancer Subtypes
29. Supplementary Fig 7 from Whole-genome CpG-resolution DNA Methylation Profiling of HNSCC Reveals Distinct Mechanisms of Carcinogenesis for Fine-scale HPV+ Cancer Subtypes
30. Supplementary Methods from Whole-genome CpG-resolution DNA Methylation Profiling of HNSCC Reveals Distinct Mechanisms of Carcinogenesis for Fine-scale HPV+ Cancer Subtypes
31. Whole genome CpG-resolution DNA methylation profiling of HNSCC reveals distinct mechanisms of carcinogenesis for fine-scale HPV+ cancer subtypes
32. Supplementary Figure S4 from Increased Nerve Density Adversely Affects Outcome in Oral Cancer
33. Supplementary Table S2 from Increased Nerve Density Adversely Affects Outcome in Oral Cancer
34. Data from Increased Nerve Density Adversely Affects Outcome in Oral Cancer
35. Redefining Perineural Invasion: Integration of Biology With Clinical Outcome
36. Non-murine models to investigate tumor-immune interactions in head and neck cancer
37. Immune-relevant aspects of murine models of head and neck cancer
38. Receptor‐interacting protein (RIP) and Sirtuin‐3 (SIRT3) are on opposite sides of anoikis and tumorigenesis
39. 5-Hydroxymethylation highlights the heterogeneity in keratinization and cell junctions in head and neck cancers
40. Sirtuin‐3 (SIRT3), a novel potential therapeutic target for oral cancer
41. The CS1 segment of fibronectin is involved in human OSCC pathogenesis by mediating OSCC cell spreading, migration, and invasion
42. Oral Microbiome Community Composition in Head and Neck Squamous Cell Carcinoma
43. Increased Nerve Density Adversely Affects Outcome in Oral Cancer
44. Cytokines in saliva increase in head and neck cancer patients after treatment
45. Biomarkers for Individualized Oral Cancer Therapy
46. Supplementary Figure 2 from Inactivation or Loss of TTP Promotes Invasion in Head and Neck Cancer via Transcript Stabilization and Secretion of MMP9, MMP2, and IL-6
47. Supplementary Figure 3 from Inactivation or Loss of TTP Promotes Invasion in Head and Neck Cancer via Transcript Stabilization and Secretion of MMP9, MMP2, and IL-6
48. Supplementary Figure 4 from Inactivation or Loss of TTP Promotes Invasion in Head and Neck Cancer via Transcript Stabilization and Secretion of MMP9, MMP2, and IL-6
49. Supplementary Figure 5 from Inactivation or Loss of TTP Promotes Invasion in Head and Neck Cancer via Transcript Stabilization and Secretion of MMP9, MMP2, and IL-6
50. Supplementary Methods, Table 1 from Inactivation or Loss of TTP Promotes Invasion in Head and Neck Cancer via Transcript Stabilization and Secretion of MMP9, MMP2, and IL-6
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