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Online social networks are often subject to influence campaigns by malicious actors through the use of automated accounts known as bots. We consider the problem of detecting bots in online social networks and assessing their impact on the opinions of individuals. We begin by analyzing the behavior of bots in social networks and identify that they exhibit heterophily, meaning they interact with humans more than other bots. We use this property to develop a detection algorithm based on the Ising model from statistical physics. The bots are identified by solving a minimum cut problem. We show that this Ising model algorithm can identify bots with higher accuracy while utilizing much less data than other state of the art methods. We then develop a a function we call generalized harmonic influence centrality to estimate the impact bots have on the opinions of users in social networks. This function is based on a generalized opinion dynamics model and captures how the activity level and network connectivity of the bots shift equilibrium opinions. To apply generalized harmonic influence centrality to real social networks, we develop a deep neural network to measure the opinions of users based on their social network posts. Using this neural network, we then calculate the generalized harmonic influence centrality of bots in multiple real social networks. For some networks we find that a limited number of bots can cause non-trivial shifts in the population opinions. In other networks, we find that the bots have little impact. Overall we find that generalized harmonic influence centrality is a useful operational tool to measure the impact of bots in social networks., 58 pages, 11 figures

We consider the problem of optimizing the placement of stubborn agents in a social network in order to maximally influence the population. We assume the network contains stubborn users whose opinions do not change, and non-stubborn users who can be persuaded. We further assume the opinions in the network are in an equilibrium that is common to many opinion dynamics models, including the well-known DeGroot model. We develop a discrete optimization formulation for the problem of maximally shifting the equilibrium opinions in a network by targeting users with stubborn agents. The opinion objective functions we consider are the opinion mean, the opinion variance, and the number of individuals whose opinion exceeds a fixed threshold. We show that the mean opinion is a monotone submodular function, allowing us to find a good solution using a greedy algorithm. We find that on real social networks in Twitter consisting of tens of thousands of individuals, a small number of stubborn agents can non-trivially influence the equilibrium opinions. Furthermore, we show that our greedy algorithm outperforms several common benchmarks. We then propose an opinion dynamics model where users communicate noisy versions of their opinions, communications are random, users grow more stubborn with time, and there is heterogeneity is how users' stubbornness increases. We prove that under fairly general conditions on the stubbornness rates of the individuals, the opinions in this model converge to the same equilibrium as the DeGroot model, despite the randomness and user heterogeneity in the model., 40 pages, 11 figures

The pathogenic chytrid fungusBatrachochytrium dendrobatidis(Bd) can cause precipitous population declines in its amphibian hosts. Responses of individuals to infection vary greatly with the capacity of their immune system to respond to the pathogen. We used a combination of comparative and experimental approaches to identify major histocompatibility complex class II (MHC-II) alleles encoding molecules that foster the survival of Bd-infected amphibians. We found that Bd-resistant amphibians across four continents share common amino acids in three binding pockets of the MHC-II antigen-binding groove. Moreover, strong signals of selection acting on these specific sites were evident among all species co-existing with the pathogen. In the laboratory, we experimentally inoculated Australian tree frogs with Bd to test how each binding pocket conformation influences disease resistance. Only the conformation of MHC-II pocket 9 of surviving subjects matched those of Bd-resistant species. This MHC-II conformation thus may determine amphibian resistance to Bd, although other MHC-II binding pockets also may contribute to resistance. Rescuing amphibian biodiversity will depend on our understanding of amphibian immune defence mechanisms against Bd. The identification of adaptive genetic markers for Bd resistance represents an important step forward towards that goal.