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3. Reemergence of a Big Brown Bat Lyssavirus rabies Variant in Striped Skunks in Flagstaff, Arizona, USA, 2021–2023.

Author

Gilbert, Amy T., Van Pelt, Lolita I., Hastings, Lias A., Gigante, Crystal M., Orciari, Lillian A., Kelley, Sabrina, Fitzpatrick, Kathryn, Condori, Rene E. Condori, Li, Yu, Brunt, Scott, Davis, April, Hopken, Matthew W., Mankowski, Clara C. P., Wallace, Ryan M., Rupprecht, Charles E., Chipman, Richard B., and Bergman, David L.

Subjects
RABIES vaccines, RABIES, VIRAL load, AUTUMN, ZOONOSES
Abstract

Background: Throughout the Americas, Lyssavirus rabies (RV) perpetuates as multiple variants among bat and mesocarnivore species. Interspecific RV spillover occurs on occasion, but clusters and viral host shifts are rare. The spillover and host shift of a big brown bat (Eptesicus fuscus) RV variant Ef-W1 into mesocarnivores was reported previously on several occasions during 2001–2009 in Flagstaff, Arizona, USA, and controlled through rabies vaccination of target wildlife. During autumn 2021, a new cluster of Ef-W1 RV cases infecting striped skunks (Mephitis mephitis) was detected from United States Department of Agriculture enhanced rabies surveillance in Flagstaff. The number of Ef-W1 RV spillover cases within a short timeframe suggested the potential for transmission between skunks and an emerging host shift. Materials and Methods: Whole and partial RV genomic sequencing was performed to evaluate the phylogenetic relationships of the 2021–2023 Ef-W1 cases infecting striped skunks with earlier outbreaks. Additionally, real-time reverse-transcriptase PCR (rtRT-PCR) was used to opportunistically compare viral RNA loads in brain and salivary gland tissues of naturally infected skunks. Results: Genomic RV sequencing revealed that the origin of the 2021–2023 epizootic of Ef-W1 RV was distinct from the multiple outbreaks detected from 2001–2009. Naturally infected skunks with the Ef-W1 RV showed greater viral RNA loads in the brain, but equivalent viral RNA loads in the mandibular salivary glands, compared to an opportunistic sample of skunks naturally infected with a South-Central skunk RV from northern Colorado, USA. Conclusion: Considering a high risk for onward transmission and spread of the Ef-W1 RV in Flagstaff, public outreach, enhanced rabies surveillance, and control efforts, focused on education, sample characterization, and vaccination, have been ongoing since 2021 to mitigate and prevent the spread and establishment of Ef-W1 RV in mesocarnivores. [ABSTRACT FROM AUTHOR]

Published
2024
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10. The Basic Domain of Herpes Simplex Virus 1 pUS9 Recruits Kinesin-1 To Facilitate Egress from Neurons

Author

Diefenbach, Russell J, Davis, April, Miranda-Saksena, Monica, Fernandez, Marian A, Kelly, Barbara J, Jones, Cheryl A, LaVail, Jennifer H, Xue, Jing, Lai, Joey, and Cunningham, Anthony L

Subjects
Biomedical and Clinical Sciences, Clinical Sciences, Sexually Transmitted Infections, Infectious Diseases, HIV/AIDS, Neurosciences, 2.2 Factors relating to the physical environment, Aetiology, Infection, Good Health and Well Being, Amino Acid Sequence, Animals, Axonal Transport, Binding Sites, Cytological Techniques, DNA Mutational Analysis, Disease Models, Animal, Female, Ganglia, Spinal, Herpes Simplex, Herpesvirus 1, Human, Host-Pathogen Interactions, Humans, Intracellular Signaling Peptides and Proteins, Kinesins, Lipoproteins, Mice, Inbred C57BL, Models, Biological, Molecular Sequence Data, Mutagenesis, Site-Directed, Neurons, Phosphoproteins, Protein Binding, Protein Interaction Mapping, Rats, Wistar, Severity of Illness Index, Skin, Viral Load, Viral Proteins, Virus Release, Biological Sciences, Agricultural and Veterinary Sciences, Medical and Health Sciences, Virology, Agricultural, veterinary and food sciences, Biological sciences, Biomedical and clinical sciences
Abstract

UnlabelledThe alphaherpesviral envelope protein pUS9 has been shown to play a role in the anterograde axonal transport of herpes simplex virus 1 (HSV-1), yet the molecular mechanism is unknown. To address this, we used an in vitro pulldown assay to define a series of five arginine residues within the conserved pUS9 basic domain that were essential for binding the molecular motor kinesin-1. The mutation of these pUS9 arginine residues to asparagine blocked the binding of both recombinant and native kinesin-1. We next generated HSV-1 with the same pUS9 arginine residues mutated to asparagine (HSV-1pUS9KBDM) and then restored them being to arginine (HSV-1pUS9KBDR). The two mutated viruses were analyzed initially in a zosteriform model of recurrent cutaneous infection. The primary skin lesion scores were identical in severity and kinetics, and there were no differences in viral load at dorsal root ganglionic (DRG) neurons at day 4 postinfection (p.i.) for both viruses. In contrast, HSV-1pUS9KBDM showed a partial reduction in secondary skin lesions at day 8 p.i. compared to the level for HSV-1pUS9KBDR. The use of rat DRG neuronal cultures in a microfluidic chamber system showed both a reduction in anterograde axonal transport and spread from axons to nonneuronal cells for HSV-1pUS9KBDM. Therefore, the basic domain of pUS9 contributes to anterograde axonal transport and spread of HSV-1 from neurons to the skin through recruitment of kinesin-1.ImportanceHerpes simplex virus 1 and 2 cause genital herpes, blindness, encephalitis, and occasionally neonatal deaths. There is also increasing evidence that sexually transmitted genital herpes increases HIV acquisition, and the reactivation of HSV increases HIV replication and transmission. New antiviral strategies are required to control resistant viruses and to block HSV spread, thereby reducing HIV acquisition and transmission. These aims will be facilitated through understanding how HSV is transported down nerves and into skin. In this study, we have defined how a key viral protein plays a role in both axonal transport and spread of the virus from nerve cells to the skin.

Published
2016

11. Mycobiome of the Bat White Nose Syndrome (WNS) Affected Caves and Mines reveals High Diversity of Fungi and Local Adaptation by the Fungal Pathogen Pseudogymnoascus (Geomyces) destructans

Author

Zhang, Tao, Victor, Tanya R., Rajkumar, Sunanda S., Li, Xiaojiang, Okoniewski, Joseph C., Hicks, Alan C., Davis, April D., Broussard, Kelly, LaDeau, Shannon L., Chaturvedi, Sudha, and Chaturvedi, Vishnu

Subjects
Quantitative Biology - Populations and Evolution
Abstract

The investigations of the bat White Nose Syndrome (WNS) have yet to provide answers as to how the causative fungus Pseudogymnoascus (Geomyces) destructans (Pd) first appeared in the Northeast and how a single clone has spread rapidly in the US and Canada. We aimed to catalogue Pd and all other fungi (mycobiome) by the culture-dependent (CD) and culture-independent (CI) methods in four Mines and two Caves from the epicenter of WNS zoonotic. Six hundred sixty-five fungal isolates were obtained by CD method including the live recovery of Pd. Seven hundred three nucleotide sequences that met the definition of operational taxonomic units (OTUs) were recovered by CI methods. Most OTUs belonged to unidentified clones deposited in the databases as environmental nucleic acid sequences (ENAS). The core mycobiome of WNS affected sites comprised of 46 species of fungi from 31 genera recovered in culture, and 17 fungal genera and 31 ENAS identified from clone libraries. Fungi such as Arthroderma spp., Geomyces spp., Kernia spp., Mortierella spp., Penicillium spp., and Verticillium spp. were predominant in culture while Ganoderma spp., Geomyces spp., Mortierella spp., Penicillium spp. and Trichosporon spp. were abundant is clone libraries. Alpha diversity analyses from CI data revealed that fungal community structure was highly diverse. However, the true species diversity remains undetermined due to under sampling. The frequent recovery of Pd indicated that the pathogen has adapted to WNS-afflicted habitats. Further, this study supports the hypothesis that Pd is an introduced species. These findings underscore the need for integrated WNS control measures that target both bats and the fungal pathogen., Comment: 59 pages, 7figures

Published
2014
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13. Origin of 3 Rabid Terrestrial Animals in Raccoon Rabies Virus-Free Zone, Long Island, New York, USA, 2016-2017

Author

Brunt, Scott, Solomon, Heather, Leavitt, Hilaire, Lasek-Nesselquist, Erica, LaPierre, Pascal, Shudt, Matt, Bigler, Laura, Singh, Navjot, and Davis, April D.

Subjects
Disease transmission -- Analysis, DNA sequencing -- Analysis, Phylogeny -- Analysis, Genomics -- Analysis, Rabies -- Analysis, Health
Abstract

Rabies has been endemic to the eastern United States in raccoons (Procyon lotor) since 1960 and endemic to New York, USA, since the 1990s (1). The spread of rabies virus [...]

Published
2020
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14. Contributors

Author

Banyard, Ashley C., primary, Biek, Roman, additional, Blanton, Jesse, additional, Briggs, Deborah J., additional, Chipman, Richard B., additional, Cleaveland, Sarah, additional, Coetzer, Andre, additional, Conzelmann, Karl-Klaus, additional, Davis, Alicia, additional, Davis, April, additional, Ertl, Hildegund C.J., additional, Fooks, Anthony R., additional, Freuling, Conrad M., additional, Gilbert, Amy T., additional, Golding, Megan, additional, Gordon, Chandra R., additional, Hampson, Katie, additional, Jackson, Alan C., additional, Knobel, Darryn L., additional, Lafon, Monique, additional, Lembo, Tiziana, additional, Mani, Reeta S., additional, Markotter, Wanda, additional, Marston, Denise A., additional, McElhinney, Lorraine M., additional, Metcalf, C. Jessica E., additional, Moore, Susan M., additional, Müller, Thomas, additional, Nadin-Davis, Susan A., additional, Nagarajan, Thirumeni, additional, Nel, Louis H., additional, Palacios, Carlos, additional, Rajeev, Malavika, additional, Rossiter, John P., additional, Rupprecht, Charles E., additional, Sabeta, Claude T., additional, Scott, Terrence P., additional, Streicker, Daniel G., additional, Wallace, Ryan MacLaren, additional, Ward, Michael, additional, and Wunner, William H., additional

Published
2020
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15. Bat rabies

Author

Banyard, Ashley C., primary, Davis, April, additional, Gilbert, Amy T., additional, and Markotter, Wanda, additional

Published
2020
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17. Data from Notch Reporter Activity in Breast Cancer Cell Lines Identifies a Subset of Cells with Stem Cell Activity

Author

D'Angelo, Rosemarie C., primary, Ouzounova, Maria, primary, Davis, April, primary, Choi, Daejin, primary, Tchuenkam, Stevie M., primary, Kim, Gwangil, primary, Luther, Tahra, primary, Quraishi, Ahmed A., primary, Senbabaoglu, Yasin, primary, Conley, Sarah J., primary, Clouthier, Shawn G., primary, Hassan, Khaled A., primary, Wicha, Max S., primary, and Korkaya, Hasan, primary

Published
2023
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18. Supplementary Figures 1-4 from Notch Reporter Activity in Breast Cancer Cell Lines Identifies a Subset of Cells with Stem Cell Activity

Author

D'Angelo, Rosemarie C., primary, Ouzounova, Maria, primary, Davis, April, primary, Choi, Daejin, primary, Tchuenkam, Stevie M., primary, Kim, Gwangil, primary, Luther, Tahra, primary, Quraishi, Ahmed A., primary, Senbabaoglu, Yasin, primary, Conley, Sarah J., primary, Clouthier, Shawn G., primary, Hassan, Khaled A., primary, Wicha, Max S., primary, and Korkaya, Hasan, primary

Published
2023
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20. Supplementary Figure 4 from The EGFR T790M Mutation Is Acquired through AICDA-Mediated Deamination of 5-Methylcytosine following TKI Treatment in Lung Cancer

Author

El Kadi, Najwa, primary, Wang, Luo, primary, Davis, April, primary, Korkaya, Hasan, primary, Cooke, Alexander, primary, Vadnala, Varun, primary, Brown, Noah A., primary, Betz, Bryan L., primary, Cascalho, Marilia, primary, Kalemkerian, Gregory P., primary, and Hassan, Khaled A., primary

Published
2023
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21. Supplementary Figure 2 from The EGFR T790M Mutation Is Acquired through AICDA-Mediated Deamination of 5-Methylcytosine following TKI Treatment in Lung Cancer

Author

El Kadi, Najwa, primary, Wang, Luo, primary, Davis, April, primary, Korkaya, Hasan, primary, Cooke, Alexander, primary, Vadnala, Varun, primary, Brown, Noah A., primary, Betz, Bryan L., primary, Cascalho, Marilia, primary, Kalemkerian, Gregory P., primary, and Hassan, Khaled A., primary

Published
2023
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23. Supplementary figure legends from RAD51 Mediates Resistance of Cancer Stem Cells to PARP Inhibition in Triple-Negative Breast Cancer

Author

Liu, Yajing, primary, Burness, Monika L., primary, Martin-Trevino, Rachel, primary, Guy, Joey, primary, Bai, Shoumin, primary, Harouaka, Ramdane, primary, Brooks, Michael D., primary, Shang, Li, primary, Fox, Alex, primary, Luther, Tahra K., primary, Davis, April, primary, Baker, Trenton L., primary, Colacino, Justin, primary, Clouthier, Shawn G., primary, Shao, Zhi-ming, primary, Wicha, Max S., primary, and Liu, Suling, primary

Published
2023
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24. Supplementary Figures from RAD51 Mediates Resistance of Cancer Stem Cells to PARP Inhibition in Triple-Negative Breast Cancer

Author

Liu, Yajing, primary, Burness, Monika L., primary, Martin-Trevino, Rachel, primary, Guy, Joey, primary, Bai, Shoumin, primary, Harouaka, Ramdane, primary, Brooks, Michael D., primary, Shang, Li, primary, Fox, Alex, primary, Luther, Tahra K., primary, Davis, April, primary, Baker, Trenton L., primary, Colacino, Justin, primary, Clouthier, Shawn G., primary, Shao, Zhi-ming, primary, Wicha, Max S., primary, and Liu, Suling, primary

Published
2023
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26. Supplementary Figure 1 from The EGFR T790M Mutation Is Acquired through AICDA-Mediated Deamination of 5-Methylcytosine following TKI Treatment in Lung Cancer

Author

El Kadi, Najwa, primary, Wang, Luo, primary, Davis, April, primary, Korkaya, Hasan, primary, Cooke, Alexander, primary, Vadnala, Varun, primary, Brown, Noah A., primary, Betz, Bryan L., primary, Cascalho, Marilia, primary, Kalemkerian, Gregory P., primary, and Hassan, Khaled A., primary

Published
2023
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27. Supplementary Figure 6 from The EGFR T790M Mutation Is Acquired through AICDA-Mediated Deamination of 5-Methylcytosine following TKI Treatment in Lung Cancer

Author

El Kadi, Najwa, primary, Wang, Luo, primary, Davis, April, primary, Korkaya, Hasan, primary, Cooke, Alexander, primary, Vadnala, Varun, primary, Brown, Noah A., primary, Betz, Bryan L., primary, Cascalho, Marilia, primary, Kalemkerian, Gregory P., primary, and Hassan, Khaled A., primary

Published
2023
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28. Supplementary Figure 3 from The EGFR T790M Mutation Is Acquired through AICDA-Mediated Deamination of 5-Methylcytosine following TKI Treatment in Lung Cancer

Author

El Kadi, Najwa, primary, Wang, Luo, primary, Davis, April, primary, Korkaya, Hasan, primary, Cooke, Alexander, primary, Vadnala, Varun, primary, Brown, Noah A., primary, Betz, Bryan L., primary, Cascalho, Marilia, primary, Kalemkerian, Gregory P., primary, and Hassan, Khaled A., primary

Published
2023
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29. Supplementary Figure 5 from The EGFR T790M Mutation Is Acquired through AICDA-Mediated Deamination of 5-Methylcytosine following TKI Treatment in Lung Cancer

Author

El Kadi, Najwa, primary, Wang, Luo, primary, Davis, April, primary, Korkaya, Hasan, primary, Cooke, Alexander, primary, Vadnala, Varun, primary, Brown, Noah A., primary, Betz, Bryan L., primary, Cascalho, Marilia, primary, Kalemkerian, Gregory P., primary, and Hassan, Khaled A., primary

Published
2023
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31. Data from A Novel IL6 Antibody Sensitizes Multiple Tumor Types to Chemotherapy Including Trastuzumab-Resistant Tumors

Author

Zhong, Haihong, primary, Davis, April, primary, Ouzounova, Maria, primary, Carrasco, Rosa A., primary, Chen, Cui, primary, Breen, Shannon, primary, Chang, Yong S., primary, Huang, Jiaqi, primary, Liu, Zheng, primary, Yao, Yihong, primary, Hurt, Elaine, primary, Moisan, Jacques, primary, Fung, Michael, primary, Tice, David A., primary, Clouthier, Shawn G., primary, Xiao, Zhan, primary, Wicha, Max S., primary, Korkaya, Hasan, primary, and Hollingsworth, Robert E., primary

Published
2023
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32. Supplemental Figures from A Novel IL6 Antibody Sensitizes Multiple Tumor Types to Chemotherapy Including Trastuzumab-Resistant Tumors

Author

Zhong, Haihong, primary, Davis, April, primary, Ouzounova, Maria, primary, Carrasco, Rosa A., primary, Chen, Cui, primary, Breen, Shannon, primary, Chang, Yong S., primary, Huang, Jiaqi, primary, Liu, Zheng, primary, Yao, Yihong, primary, Hurt, Elaine, primary, Moisan, Jacques, primary, Fung, Michael, primary, Tice, David A., primary, Clouthier, Shawn G., primary, Xiao, Zhan, primary, Wicha, Max S., primary, Korkaya, Hasan, primary, and Hollingsworth, Robert E., primary

Published
2023
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33. Data from HER2 Drives Luminal Breast Cancer Stem Cells in the Absence of HER2 Amplification: Implications for Efficacy of Adjuvant Trastuzumab

Author

Ithimakin, Suthinee, primary, Day, Kathleen C., primary, Malik, Fayaz, primary, Zen, Qin, primary, Dawsey, Scott J., primary, Bersano-Begey, Tom F., primary, Quraishi, Ahmed A., primary, Ignatoski, Kathleen Woods, primary, Daignault, Stephanie, primary, Davis, April, primary, Hall, Christopher L., primary, Palanisamy, Nallasivam, primary, Heath, Amber N., primary, Tawakkol, Nader, primary, Luther, Tahra K., primary, Clouthier, Shawn G., primary, Chadwick, Whitney A., primary, Day, Mark L., primary, Kleer, Celina G., primary, Thomas, Dafydd G., primary, Hayes, Daniel F., primary, Korkaya, Hasan, primary, and Wicha, Max S., primary

Published
2023
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34. Supplementary Methods, Figures 1 - 7 from HER2 Drives Luminal Breast Cancer Stem Cells in the Absence of HER2 Amplification: Implications for Efficacy of Adjuvant Trastuzumab

Author

Ithimakin, Suthinee, primary, Day, Kathleen C., primary, Malik, Fayaz, primary, Zen, Qin, primary, Dawsey, Scott J., primary, Bersano-Begey, Tom F., primary, Quraishi, Ahmed A., primary, Ignatoski, Kathleen Woods, primary, Daignault, Stephanie, primary, Davis, April, primary, Hall, Christopher L., primary, Palanisamy, Nallasivam, primary, Heath, Amber N., primary, Tawakkol, Nader, primary, Luther, Tahra K., primary, Clouthier, Shawn G., primary, Chadwick, Whitney A., primary, Day, Mark L., primary, Kleer, Celina G., primary, Thomas, Dafydd G., primary, Hayes, Daniel F., primary, Korkaya, Hasan, primary, and Wicha, Max S., primary

Published
2023
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35. Phylogenomic Diversity Elucidates Mechanistic Insights into Lyme Borreliae-Host Association

Author

Combs, Matthew, primary, Marcinkiewicz, Ashley L., additional, Dupuis, Alan P., additional, Davis, April D., additional, Lederman, Patricia, additional, Nowak, Tristan A., additional, Stout, Jessica L., additional, Strle, Klemen, additional, Fingerle, Volker, additional, Margos, Gabriele, additional, Ciota, Alexander T., additional, Diuk-Wasser, Maria A., additional, Kolokotronis, Sergios-Orestis, additional, and Lin, Yi-Pin, additional

Published
2022
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36. Coinfection of Canine Distemper Virus and Rabies Virus in Wildlife Samples Submitted for Routine Rabies Testing.

Author

Moessner, Heather, Brunt, Scott, Diaz, Alexander, and Davis, April

Abstract

Canine distemper virus (also known as Canine morbillivirus), the etiologic agent of canine distemper, is a highly contagious pathogen causing a multisystemic infection in carnivores globally. Canine distemper may be clinically indistinguishable from rabies, and outbreaks of either disease are major concerns. In the US, both diseases are endemic and managed by parenteral vaccination in domestic animals. In wildlife, oral vaccination and trap-vaccinate-release programs are available for rabies prevention, but no such strategies exist for canine distemper. We evaluated the prevalence at which canine distemper virus occurred concurrently in animals infected with rabies virus. Real-time quantitative reverse transcriptase PCR (qRT-PCR) was performed on specimens previously diagnosed with rabies during 2017–19 by the New York State Rabies Laboratory. Real-time qRT-PCR detected concurrent canine distemper virus infection in 73 of 1,302 animals with rabies virus. Coinfection rates were approximately 9% in Procyon lotor, 2% in Vulpes vulpes, and 0.4% in Mephitis mephitis, with an overall prevalence of 5.6%. As comorbidities in wildlife occur, laboratory-based surveillance and confirmatory testing are critical to rapid decision making for disease prevention. Rabies virus incursions are expensive and difficult to manage, and spillover events create health risks to humans and domestic animals as well as to free-roaming wildlife. [ABSTRACT FROM AUTHOR]

Published
2023
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37. Electronic Supplementary Material from Cellular and immunological mechanisms influence host-adapted phenotypes in a vector-borne microparasite

Author

Lin, Yi-Pin, Tufts, Danielle M., Combs, Matthew, Dupuis, Alan P., Marcinkiewicz, Ashley L., Hirsbrunner, Andrew D., Diaz, Alexander J., Stout, Jessica L., Blom, Anna M., Strle, Klemen, Davis, April D., Kramer, Laura D., Kolokotronis, Sergios-Orestis, and Diuk-Wasser, Maria A.

Abstract

This file includes Supplementary Materials and Methods, Supplementary Figure Legends, Supplementary Tables, Supplementary Figures, the References, and acknowledgement for this file

Published
2022
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39. Cellular and immunological mechanisms influence host-adapted phenotypes in a vector-borne microparasite

Author

Lin, Yi-Pin, primary, Tufts, Danielle M., additional, Combs, Matthew, additional, Dupuis, Alan P., additional, Marcinkiewicz, Ashley L., additional, Hirsbrunner, Andrew D., additional, Diaz, Alexander J., additional, Stout, Jessica L., additional, Blom, Anna M., additional, Strle, Klemen, additional, Davis, April D., additional, Kramer, Laura D., additional, Kolokotronis, Sergios-Orestis, additional, and Diuk-Wasser, Maria A., additional

Published
2022
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42. School Counselors' Interest in Professional Literature and Research.

Author

Bauman, Sheri, Siegel, Jason T., and Davis, April

Abstract

Study examines "Professional School Counseling" to determine the type of material this flagship journal has included. Additionally, the inquiry was expanded to survey school counselors regarding their research interests and the most widely accessed sources of research information for this group. (Contains 10 references and 5 tables.) (GCP)

Published
2002

46. Contributors

Author

Aaronson, Stuart A., primary, Abbruzzese, James L., additional, Abel, Erika L., additional, Acharyya, Swarnali, additional, Alobeid, Bachir, additional, Amengual, Jennifer, additional, Anderson, Kenneth C., additional, Barbash, Olena, additional, Bast,, Robert C., additional, Baylin, Stephen B., additional, Bertino, Joseph R., additional, Bhagat, Govind, additional, Bissell, Mina J., additional, Boerner, Scott A., additional, Bolden, Jessica E., additional, Borden, Ernest C., additional, Brock, Malcolm V., additional, Brown, Nelson E., additional, Califano, Andrea, additional, Calin, George Adrian, additional, Carroll, Peter R., additional, Chennubhotla, Chakra, additional, Choueiri, Toni K., additional, Corless, Christopher L., additional, Correia, Ana Luísa, additional, Critchley-Thorne, Rebecca, additional, Croce, Carlo Maria, additional, D’Andrea, Alan D., additional, Davidson, Nancy E., additional, Davis, April, additional, Deng, Changchun, additional, Diehl, J. Alan, additional, DiGiovanni, John, additional, Eder, Joseph Paul, additional, Ekmekcioglu, Suhendan, additional, Ernstoff, Marc S., additional, Faeder, James R., additional, Fearon, Eric R., additional, Finley, Lydia W.S., additional, Fisher, Susan, additional, Fletcher, Christopher D.M., additional, Ghajar, Cyrus M., additional, Glick, Adam B., additional, Gough, Albert, additional, Govindan, Ramaswamy, additional, Grandis, Jennifer R., additional, Gray, Joe W., additional, Green, Douglas R., additional, Greene, Kirsten L., additional, Grimm, Elizabeth A., additional, Grumolato, Luca, additional, Gu, Jian, additional, Guertin, David A., additional, Hahn, William C., additional, Hait, William N., additional, Havrda, Matthew C., additional, Hedberg, Matthew L., additional, Herbst, Roy S., additional, Hinds, Philip W., additional, Hoehn, Daniela, additional, Howley, Peter M., additional, Hsieh, Andrew C., additional, Hwu, Patrick, additional, Israel, Mark A., additional, Jacks, Tyler, additional, Joyce, Johanna A., additional, Kaelin,, William G., additional, Kantarjian, Hagop, additional, Keith, Brian, additional, Kelly, Ronan, additional, Korkaya, Hasan, additional, Korn, W. Michael, additional, Kurzrock, Razelle, additional, Larsen, Jill E., additional, Lee, Adrian V., additional, Lee, J. Jack, additional, Letai, Anthony G., additional, Lezon, Timothy, additional, Li, Long-Cheng, additional, Li, Yan, additional, Lippman, Scott M., additional, Liu, Yong-Jun, additional, Lizée, Gregory, additional, Look, A. Thomas, additional, LoRusso, Patricia M., additional, Lowe, Scott W., additional, Malkin, David, additional, Mardis, Elaine R., additional, Margolin, Judith, additional, Massagué, Joan, additional, Matrisian, Lynn, additional, McCormick, Frank, additional, Mendelsohn, John, additional, Mills, Gordon B., additional, Minna, John D., additional, Mollapour, Mehdi, additional, Morgensztern, Daniel, additional, Murphy, Robert F., additional, Neckers, Len, additional, O’Connor, Owen A., additional, O’Donnell, Peter H., additional, Oesterreich, Steffi, additional, Pardoll, Drew, additional, Payne, Elspeth, additional, Poplack, David G., additional, Post, Sean M., additional, Quail, Daniela F., additional, Quintás-Cardama, Alfonso, additional, Rabin, Karen R., additional, Ratain, Mark J., additional, Reimann, Julie D.R., additional, Romero, Ignacio, additional, Rubin, Eric, additional, Ruggero, Davide, additional, Sabatini, David M., additional, Sawas, Ahmed, additional, Schafer, Eric S., additional, Schroff, Rachna T., additional, Sherman, Steven I., additional, Signoretti, Sabina, additional, Sikic, Branimir I., additional, Simon, M. Celeste, additional, Spellman, Paul T., additional, Stuart, Josh, additional, Surana, Rishi, additional, Tamguney, Tanja Meyer, additional, Taylor, D. Lansing, additional, Terán, Mario D., additional, Thompson, Craig B., additional, Tonon, Giovanni, additional, Weinberg, Robert A., additional, Weiner, Louis M., additional, Welch, Danny R., additional, White, Eileen, additional, Wicha, Max S., additional, Winslow, Monte M., additional, Witkowska, H. Ewa, additional, Wu, Xifeng, additional, and Yuspa, Stuart H., additional

Published
2015
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