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2. Cytokine-armed dendritic cell progenitors for antigen-agnostic cancer immunotherapy

3. Apelin-driven endothelial cell migration sustains intestinal progenitor cells and tumor growth

4. Consensus guidelines for the use and interpretation of angiogenesis assays

7. The angiopoietin-Tie2 pathway regulates Purkinje cell dendritic morphogenesis in a cell-autonomous manner

10. Optimized antiangiogenic reprogramming of the tumor microenvironment potentiates CD40 immunotherapy

11. Perivascular Macrophages Limit Permeability

15. Living With Hyraxes: Biogeography and Comparative Ecology of West African Fornasinius Beetles.

16. Antiangiogenic immunotherapy suppresses desmoplastic and chemoresistant intestinal tumors in mice

17. Medical and Personal Characteristics Can Predict the Risk of Lung Metastasis

18. Bimodal CD40/Fas-Dependent Crosstalk between iNKT Cells and Tumor-Associated Macrophages Impairs Prostate Cancer Progression

19. Cytokine-armed dendritic cell progenitors for antigen-agnostic cancer immunotherapy

23. Chemotherapy elicits pro-metastatic extracellular vesicles in breast cancer models

26. Targeting Macrophages Sensitizes Chronic Lymphocytic Leukemia to Apoptosis and Inhibits Disease Progression

28. Defining and targeting tumor-associated macrophages in malignant mesothelioma

30. Supplementary File from The Selective Tie2 Inhibitor Rebastinib Blocks Recruitment and Function of Tie2Hi Macrophages in Breast Cancer and Pancreatic Neuroendocrine Tumors

31. Data from The Selective Tie2 Inhibitor Rebastinib Blocks Recruitment and Function of Tie2Hi Macrophages in Breast Cancer and Pancreatic Neuroendocrine Tumors

36. Supplementary Figures S1-S7 from Perivascular M2 Macrophages Stimulate Tumor Relapse after Chemotherapy

37. Supplemental Figure S1 from Guidance Molecule SEMA3A Restricts Tumor Growth by Differentially Regulating the Proliferation of Tumor-Associated Macrophages

38. Supplemental Figure Legends from Guidance Molecule SEMA3A Restricts Tumor Growth by Differentially Regulating the Proliferation of Tumor-Associated Macrophages

39. Supplementary Figures 1-9, Tables 1-4, Methods and Materials from Epidermal Growth Factor Receptor Expression Identifies Functionally and Molecularly Distinct Tumor-Initiating Cells in Human Glioblastoma Multiforme and Is Required for Gliomagenesis

41. Data from Epidermal Growth Factor Receptor Expression Identifies Functionally and Molecularly Distinct Tumor-Initiating Cells in Human Glioblastoma Multiforme and Is Required for Gliomagenesis

42. Supplementary Table S1 from Perivascular M2 Macrophages Stimulate Tumor Relapse after Chemotherapy

44. Supplemental Table S1 from Guidance Molecule SEMA3A Restricts Tumor Growth by Differentially Regulating the Proliferation of Tumor-Associated Macrophages

45. Data from Perivascular M2 Macrophages Stimulate Tumor Relapse after Chemotherapy

46. Supplemental Methods and References from Guidance Molecule SEMA3A Restricts Tumor Growth by Differentially Regulating the Proliferation of Tumor-Associated Macrophages

48. Supplementary Figure Legends 1-5, Methods from Angiopoietin-2 Regulates Gene Expression in TIE2-Expressing Monocytes and Augments Their Inherent Proangiogenic Functions

49. Data from Guidance Molecule SEMA3A Restricts Tumor Growth by Differentially Regulating the Proliferation of Tumor-Associated Macrophages

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