35 results on '"Di Gioia, Marco"'
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2. EBASI: IoT-Based Emotion and Behaviour Recognition System Against Elderly People Social Isolation
3. CD14: Not Just Chaperone, But a Key-Player in Inflammation
4. Zinc-dependent histone deacetylases drive neutrophil extracellular trap formation and potentiate local and systemic inflammation
5. EBASI: IoT-Based Emotion and Behaviour Recognition System Against Elderly People Social Isolation
6. Host-derived oxidized phospholipids initiate effector-triggered immunity fostering lethality upon microbial encounter.
7. Endogenous oxidized phospholipids reprogram cellular metabolism and boost hyperinflammation
8. Homodimeric Granzyme A Opsonizes Mycobacterium tuberculosis and Inhibits Its Intracellular Growth in Human Monocytes via Toll-Like Receptor 4 and CD14
9. Quantitative cytofluorimetric analysis of mouse neutrophil extracellular traps
10. CD14: Not Just Chaperone, But a Key-Player in Inflammation
11. An endogenous caspase-11 ligand elicits interleukin-1 release from living dendritic cells
12. Prioritized single-cell proteomics reveals molecular and functional polarization across primary macrophages
13. An adjuvant strategy enabled by modulation of the physical properties of microbial ligands expands antigen immunogenicity
14. Inhibition of transcription factor NFAT activity in activated platelets enhances their aggregation and exacerbates gram-negative bacterial septicemia
15. By Capturing Inflammatory Lipids Released from Dying Cells, the Receptor CD14 Induces Inflammasome-Dependent Phagocyte Hyperactivation
16. Toll-like receptor co-receptors as master regulators of the immune response
17. Aged vasculature drives neutrophils mad
18. Inositol 1,4,5-trisphosphate 3-kinase B promotes Ca 2+ mobilization and the inflammatory activity of dendritic cells
19. Dooming Phagocyte Responses: Inflammatory Effects of Endogenous Oxidized Phospholipids
20. Endogenous oxidized phospholipids reprogram cellular metabolism and boost hyperinflammation
21. Endogenous oxidized phospholipids reprogram cellular metabolism and boost hyperinflammation
22. Inositol 1,4,5-trisphosphate 3-kinase B promotes Ca2+ mobilization and the inflammatory activity of dendritic cells.
23. Prolonged contact with dendritic cells turns lymph node-resident NK cells into anti-tumor effectors
24. Prolonged contact with dendritic cells turns lymph node‐resident NK cells into anti‐tumor effectors
25. Toll-like receptor co-receptors as master regulators of the immune response
26. Defining the consequences of CD14 engagement and NFAT activation in innate immune cells: from molecular mechanisms to a preclinical model
27. CD14 and NFAT mediate lipopolysaccharide-induced skin edema formation in mice
28. IL-15 cis Presentation Is Required for Optimal NK Cell Activation in Lipopolysaccharide-Mediated Inflammatory Conditions
29. Purinergic Signaling to Terminate TLR Responses in Macrophages.
30. Clinical correlates and cognitive underpinnings of verbal fluency impairment after chronic subthalamic stimulation in Parkinson's disease
31. Lipopolysaccharide Regulation of Dendritic Cells Activation and Life Cycle: in vitro and in vivo Studies Towards Antitumor Immunoactivity
32. IL-15 cisPresentation Is Required for Optimal NK Cell Activation in Lipopolysaccharide-Mediated Inflammatory Conditions
33. Inositol 1,4,5-trisphosphate 3-kinase B promotes Ca2+mobilization and the inflammatory activity of dendritic cells
34. CD14 and NFAT mediate lipopolysaccharide-induced skin edema formation in mice.
35. Homodimeric Granzyme A Opsonizes Mycobacterium tuberculosis and Inhibits Its Intracellular Growth in Human Monocytes via Toll-Like Receptor 4 and CD14.
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