891 results on '"Dib-Hajj, Sulayman"'
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2. Functionally-selective inhibition of threshold sodium currents and excitability in dorsal root ganglion neurons by cannabinol
3. Disordered but effective: short linear motifs as gene therapy targets for hyperexcitability disorders
4. Pain-causing stinging nettle toxins target TMEM233 to modulate NaV1.7 function
5. Compartment-specific regulation of NaV1.7 in sensory neurons after acute exposure to TNF-α
6. Maximizing treatment efficacy through patient stratification in neuropathic pain trials
7. High-throughput combined voltage-clamp/current-clamp analysis of freshly isolated neurons
8. The fates of internalized NaV1.7 channels in sensory neurons: Retrograde cotransport with other ion channels, axon-specific recycling, and degradation
9. Gain-of-function mutation of a voltage-gated sodium channel NaV1.7 associated with peripheral pain and impaired limb development
10. Exome Sequencing Implicates Impaired GABA Signaling and Neuronal Ion Transport in Trigeminal Neuralgia
11. Sodium currents in naïve mouse dorsal root ganglion neurons: No major differences between sexes
12. Lessons learned in translating pain knowledge into practice
13. Pediatric Erythromelalgia and SCN9A Mutations: Systematic Review and Single-Center Case Series
14. NaV1.7: A central role in pain
15. Genetic, electrophysiological, and pathological studies on patients with SCN9A‐related pain disorders
16. Pharmacological characterization of a rat Nav1.7 loss-of-function model with insensitivity to pain
17. Dexpramipexole blocks Nav1.8 sodium channels and provides analgesia in multiple nociceptive and neuropathic pain models
18. Genomic analysis of 21 patients with corneal neuralgia after refractive surgery
19. Tetrodotoxin-Sensitive Na + Channels and Muscarinic and Purinergic Receptors Identified in Human Erythroid Progenitor Cells and Red Blood Cell Ghosts
20. Nav1.7 gain-of-function mutation I228M triggers age-dependent nociceptive insensitivity and C-LTMR dysregulation
21. TRPA1 rare variants in chronic neuropathic and nociplastic pain patients
22. Sensory Neuron-Specific Sodium Channel SNS Is Abnormally Expressed in the Brains of Mice with Experimental Allergic Encephalomyelitis and Humans with Multiple Sclerosis
23. Conserved but not critical: Trafficking and function of NaV1.7 are independent of highly conserved polybasic motifs
24. Inflammation differentially controls transport of depolarizing Nav versus hyperpolarizing Kv channels to drive rat nociceptor activity
25. Nav1.7 P610T mutation in two siblings with persistent ocular pain after corneal axon transection: impaired slow inactivation and hyperexcitable trigeminal neurons
26. Kv7-specific activators hyperpolarize resting membrane potential and modulate human iPSC-derived sensory neuron excitability
27. Paclitaxel effects on axonal localization and vesicular trafficking of NaV1.8
28. Integrative miRNA–mRNA profiling of human epidermis: unique signature of SCN9A painful neuropathy
29. A TRPM7 mutation linked to familial trigeminal neuralgia: Omega current and hyperexcitability of trigeminal ganglion neurons
30. Structural Modelling and Mutant Cycle Analysis Predict Pharmacoresponsiveness of a NaV1.7 Mutant Channel
31. Recent advances for using human induced-pluripotent stem cells as pain-in-a-dish models of neuropathic pain
32. Maximizing treatment efficacy through patient stratification in neuropathic pain trials
33. Non-psychotropic phytocannabinoid interactions with voltage-gated sodium channels: An update on cannabidiol and cannabigerol
34. Fibroblast growth factor homologous factor 2 attenuates excitability of DRG neurons
35. Sodium channel [Na.sub.v]1.9 mutations associated with insensitivity to pain dampen neuronal excitability
36. Characterization of a de novo SCN8A mutation in a patient with epileptic encephalopathy
37. A TRPM7 mutation linked to familial trigeminal neuralgia: Omega current and hyperexcitability of trigeminal ganglion neurons
38. V
39. Genetic Profiling of Sodium Channels in Diabetic Painful and Painless and Idiopathic Painful and Painless Neuropathies.
40. Multiple myosin motors interact with sodium/potassium-ATPase alpha 1 subunits
41. Loss-of-function mutations of SCN10A encoding NaV1.8 α subunit of voltage-gated sodium channel in patients with human kidney stone disease
42. Wound-healing growth factor, basic FGF, induces Erk1/2-dependent mechanical hyperalgesia
43. A Gain-of-Function Mutation in Nav1.6 in a Case of Trigeminal Neuralgia
44. Voltage-gated sodium channels and pain associated with nerve injury and neuropathies
45. Depolarizing NaV and Hyperpolarizing KV Channels Are Co-Trafficked in Sensory Neurons
46. Inhibition of sodium conductance by cannabigerol contributes to a reduction of dorsal root ganglion neuron excitability
47. Sodium channels and the molecular basis for pain
48. Gain-of-function Na v 1.8 mutations in painful neuropathy
49. Familial gain-of-function Nav1.9 mutation in a painful channelopathy
50. COL6A5 variants in familial neuropathic chronic itch
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