285 results on '"Ding, Wei-Guang"'
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2. Selective activation of adrenoceptors potentiates IKs current in pulmonary vein cardiomyocytes through the protein kinase A and C signaling pathways
3. Selective activation of adrenoceptors potentiates IKs current in pulmonary vein cardiomyocytes through the protein kinase A and C signaling pathways
4. A de novo gain-of-function KCND3 mutation in early repolarization syndrome
5. Dexmedetomidine Exerts a Negative Chronotropic Action on Sinoatrial Node Cells Through the Activation of Imidazoline Receptors
6. Postnatal developmental changes in the sensitivity of L-type Ca2+ channel to inhibition by verapamil in a mouse heart model
7. Phosphatidylinositol4-phosphate 5-kinase prevents the decrease in the HERG potassium current induced by Gq protein-coupled receptor stimulation
8. Regulation of human cardiac Kv1.5 channels by extracellular acidification
9. Interactions of Propofol With Human Voltage-gated Kv1.5 Channel Determined by Docking Simulation and Mutagenesis Analyses
10. Abstract 19140: Early Repolarization Syndrome Caused by a de novo KCND3 Gain-of-Function Mutation
11. Propofol, an Anesthetic Agent, Inhibits HCN Channels through the Allosteric Modulation of the cAMP-Dependent Gating Mechanism
12. Ca2+/calmodulin potentiates I Ks in sinoatrial node cells by activating Ca2+/calmodulin-dependent protein kinase II
13. Lysophosphatidylcholine enhances IKs currents in cardiac myocytes through activation of G protein, PKC and Rho signaling pathways
14. Serum albumin attenuates the open-channel blocking effects of propofol on the human Kv1.5 channel
15. Putative binding sites for arachidonic acid on the human cardiac Kv1.5 channel
16. Interaction of propofol with voltage-gated human Kv1.5 channel through specific amino acids within the pore region
17. KCNE2 modulation of Kv4.3 current and its potential role in fatal rhythm disorders
18. Presence and functional role of the rapidly activating delayed rectifier K + current in left and right atria of adult mice
19. Correction to: Heterogeneous functional expression of the sustained inward Na+ current in guinea pig sinoatrial node cells
20. Elevation of propofol sensitivity of cardiac IKs channel by KCNE1 polymorphism D85N.
21. Adrenergic regulation of the rapid component of delayed rectifier K + current: Implications for arrhythmogenesis in LQT2 patients
22. Long QT syndrome type 8: novel CACNA1C mutations causing QT prolongation and variant phenotypes
23. Elevation of propofol sensitivity of cardiac I Ks channel by KCNE1 polymorphism D85N
24. Regulatory mechanisms underlying the modulation of GIRK1/GIRK4 heteromeric channels by P2Y receptors
25. Hydroxyzine, a First Generation H1-Receptor Antagonist, Inhibits Human Ether-a-go-go–Related Gene (HERG) Current and Causes Syncope in a Patient With the HERG Mutation
26. Cinnamyl-3,4-dihydroxy-α-cyanocinnamate and nordihydroguaiaretic acid inhibit human Kv1.5 currents independently of lipoxygenase
27. Characterization of the Rapidly Activating Delayed Rectifier Potassium Current, I Kr, in HL-1 Mouse Atrial Myocytes
28. Gain-of-Function KCNH2 Mutations in Patients with Brugada Syndrome
29. Cardiac Channelopathies Associated with Infantile Fatal Ventricular Arrhythmias: From the Cradle to the Bench
30. Expression and functional maintenance of volume-regulated anion channels in myometrial smooth muscles of pregnant mice.
31. Expression and functional maintenance of volume-regulated anion channels in myometrial smooth muscles of pregnant mice
32. Open‐channel blocking action of volatile anaesthetics desflurane and sevoflurane on human voltage‐gated K v 1.5 channel
33. Presence and functional role of the rapidly activating delayed rectifier K+ current in left and right atria of adult mice
34. Open-channel blocking action of volatile anaesthetics desflurane and sevoflurane on human voltage-gated Kv 1.5 channel.
35. Characterization and functional role of rapid- and slow-activating delayed rectifier K+ currents in atrioventricular node cells of guinea pigs.
36. Multisite regulation of insulin secretion by cAMP-increasing agonists: evidence that glucagon-like peptide 1 and glucagon act via distinct receptors
37. Adrenaline stimulates glucagon secretion in pancreatic A-cells by increasing the Casuper2+ current and the number of granules close to the L-type Casuper2+ channels
38. Regulation of the muscarinic K+ channel by extracellular ATP through membrane phosphatidylinositol 4,5-bisphosphate in guinea-pig atrial myocytes
39. Responses of the sustained inward current to autonomic agonists in guinea-pig sino-atrial node pacemaker cells
40. Open-State Unblock Characterizes Acute Inhibition of IKs Potassium Current by Amiodarone in Guinea Pig Ventricular Myocytes
41. Phylogenetic study of serotonin-immunoreactive structures in the pancreas of various vertebrates
42. Long-term 4-AP treatment facilitates functional expression of human Kv1.5 channel
43. Glucagon-Like Peptide 1(7-36) Amide Stimulates Exocytosis in Human Pancreatic beta-Cells by Both Proximal and Distal Regulatory Steps in Stimulus-Secretion Coupling
44. Glucagon-Like Peptide I and Glucose-Dependent Insulinotropic Polypeptide Stimulate Ca sup 2+-Induced Secretion in Rat alpha-Cells by a Protein Kinase A-Mediated Mechanism
45. Protein Kinase A-Dependent Stimulation of Exocytosis in Mouse Pancreatic Beta-Cells by Glucose-Dependent Insulinotropic Polypeptide
46. Glucagon-like peptide I and glucose-dependent insulinotropic polypeptide stimulate Ca2+-induced secretion in rat alpha-cells by a protein kinase A-mediated mechanism
47. Protein kinase a-dependent stimulation of exocytosis in mouse pancreatic β-cells by glucose-dependent insulinotropic polypeptide
48. Novel intracellular transport-refractory mutations in KCNH2 identified in patients with symptomatic long QT syndrome
49. Transient Receptor Potential Canonical Channel Blockers Improve Ventricular Contractile Functions After Ischemia/Reperfusion in a Langendorff-perfused Mouse Heart Model
50. A hERG mutation E1039X produced a synergistic lesion on IKs together with KCNQ1-R174C mutation in a LQTS family with three compound mutations
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