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1. Oncogenic PIK3CA mutations in colorectal cancers and polyps

2. Type I Interferons Suppress Anti-parasitic Immunity and Can Be Targeted to Improve Treatment of Visceral Leishmaniasis

4. Interleukin 2 is an Upstream Regulator of CD4+ T Cells From Visceral Leishmaniasis Patients With Therapeutic Potential

7. Distinct Roles for CD4+ Foxp3+ Regulatory T Cells and IL-10–Mediated Immunoregulatory Mechanisms during Experimental Visceral Leishmaniasis Caused by Leishmania donovani

8. Rapid loss of group 1 innate lymphoid cells during blood stage Plasmodium infection

9. Galectin-1 Impairs the Generation of Anti-Parasitic Th1 Cell Responses in the Liver during Experimental Visceral Leishmaniasis

10. Programmed death-1 ligand 2-mediated regulation of the PD-L1 to PD-1 axis is essential for establishing CD4+ T cell immunity

11. Development of immunotherapies and vaccines against visceral leishmaiasis

12. Type I Interferons Regulate Immune Responses in Humans with Blood-Stage Plasmodium falciparum Infection

13. Programmed Death-1 Ligand 2-Mediated Regulation of the PD-L1 to PD-1 Axis Is Essential for Establishing CD4 + T Cell Immunity

15. Correction: Blimp-1-Dependent IL-10 Production by Tr1 Cells Regulates TNF-Mediated Tissue Pathology

16. Combined Immune Therapy for the Treatment of Visceral Leishmaniasis

17. Blimp-1-Dependent IL-10 Production by Tr1 Cells Regulates TNF-Mediated Tissue Pathology

18. IL-17A-producing gamma delta T cells suppress early control of parasite growth by monocytes in the liver

19. IL-17A–Producing γδ T Cells Suppress Early Control of Parasite Growth by Monocytes in the Liver

20. Coinfection with Blood-Stage Plasmodium Promotes Systemic Type I Interferon Production during Pneumovirus Infection but Impairs Inflammation and Viral Control in the Lung

21. Tissue requirements for establishing long-term CD4+ T cell-mediated immunity following Leishmania donovani infection

22. Immune regulation during chronic visceral leishmaniasis

24. Tissue Requirements for Establishing Long-Term CD4+ T Cell–Mediated Immunity following Leishmania donovani Infection

25. PD-1 Dependent Exhaustion of CD8+ T Cells Drives Chronic Malaria

26. Oncogenic PIK3CA mutations in colorectal cancers and polyps

27. PD-1 Dependent Exhaustion of CD8+ T Cells Drives Chronic Malaria.

28. Coinfection with Blood-Stage PlasmodiumPromotes Systemic Type I Interferon Production during Pneumovirus Infection but Impairs Inflammation and Viral Control in the Lung

29. PD-1 Dependent Exhaustion of CD8+T Cells Drives Chronic Malaria

31. Distinct Roles for CD4+ Foxp3+ Regulatory T Cells and IL-10-Mediated Immunoregulatory Mechanisms during Experimental Visceral Leishmaniasis Caused by Leishmania donovani.

32. Programmed Death-1 Ligand 2-Mediated Regulation of the PD-L1 to PD-1 Axis Is Essential for Establishing CD4+ T Cell Immunity.

33. Tissue Requirements for Establishing Long-Term CD4+ T Cell-Mediated Immunity following Leishmania donovani Infection.

34. Distinct Roles for CD4 + Foxp3 + Regulatory T Cells and IL-10-Mediated Immunoregulatory Mechanisms during Experimental Visceral Leishmaniasis Caused by Leishmania donovani .

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