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Your search keyword '"Feng, Shi‐Kai"' showing total 8 results
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8 results on '"Feng, Shi‐Kai"'

2. Fructose‐Coated Ångstrom Silver Particles Suppress Gastric Cancer Growth by Activating Gasdermin D‐Mediated Pyroptosis

Author

Li, You‐You, primary, Xia, Kun, additional, Liu, Yi‐Wei, additional, Tan, Yi‐Juan, additional, Li, Hong‐Ming, additional, Wang, Yi‐Yi, additional, Feng, Shi‐Kai, additional, Jin, Ling, additional, Wan, Teng‐Fei, additional, Cao, Jia, additional, Chen, Chun‐Yuan, additional, Liu, Zheng‐Zhao, additional, Hu, Xiong‐Ke, additional, Wu, Ben, additional, Wang, Yang, additional, He, Ze‐Hui, additional, Yin, Hao, additional, Situ, Wei‐Yi, additional, Qi, Lu‐Yue, additional, Wang, Zhen‐Xing, additional, and Xie, Hui, additional

Published
2022
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4. Neuronal Induction of Bone‐Fat Imbalance through Osteocyte Neuropeptide Y (Adv. Sci. 24/2021)

Author

Zhang, Yan, primary, Chen, Chun‐Yuan, additional, Liu, Yi‐Wei, additional, Rao, Shan‐Shan, additional, Tan, Yi‐Juan, additional, Qian, Yu‐Xuan, additional, Xia, Kun, additional, Huang, Jie, additional, Liu, Xi‐Xi, additional, Hong, Chun‐Gu, additional, Yin, Hao, additional, Cao, Jia, additional, Feng, Shi‐Kai, additional, He, Ze‐Hui, additional, Li, You‐You, additional, Luo, Zhong‐Wei, additional, Wu, Ben, additional, Yan, Zi‐Qi, additional, Chen, Tuan‐Hui, additional, Chen, Meng‐Lu, additional, Wang, Yi‐Yi, additional, Wang, Zhen‐Xing, additional, Liu, Zheng‐Zhao, additional, Luo, Ming‐Jie, additional, Hu, Xiong‐Ke, additional, Jin, Ling, additional, Wan, Teng‐Fei, additional, Yue, Tao, additional, Tang, Si‐Yuan, additional, and Xie, Hui, additional

Published
2021
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5. Deficiency of Omentin-1 leads to delayed fracture healing through excessive inflammation and reduced CD31hiEmcnhi vessels

Author

Feng, Shi-Kai, primary, Chen, Tuan-Hui, additional, Li, Hong-Ming, additional, Cao, Jia, additional, Liu, Dong-Biao, additional, Rao, Shan-Shan, additional, Liu, Jiang-Hua, additional, Zhang, Yan, additional, Wang, Zhen-Xing, additional, Li, You-You, additional, Tan, Yi-Juan, additional, Liu, Yi-Wei, additional, Hong, Chun-Gu, additional, Yan, Zi-Qi, additional, Chen, Meng-Lu, additional, Wang, Yi-Yi, additional, Yin, Hao, additional, Jin, Ling, additional, Xie, Hui, additional, Wang, Zheng-Guang, additional, and Zhou, Yong, additional

Published
2021
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6. Extracellular Vesicles from Child Gut Microbiota Enter into Bone to Preserve Bone Mass and Strength

Author

Liu, Jiang‐Hua, primary, Chen, Chun‐Yuan, additional, Liu, Zheng‐Zhao, additional, Luo, Zhong‐Wei, additional, Rao, Shan‐Shan, additional, Jin, Ling, additional, Wan, Teng‐Fei, additional, Yue, Tao, additional, Tan, Yi‐Juan, additional, Yin, Hao, additional, Yang, Fei, additional, Huang, Fei‐Yu, additional, Guo, Jian, additional, Wang, Yi‐Yi, additional, Xia, Kun, additional, Cao, Jia, additional, Wang, Zhen‐Xing, additional, Hong, Chun‐Gu, additional, Luo, Ming‐Jie, additional, Hu, Xiong‐Ke, additional, Liu, Yi‐Wei, additional, Du, Wei, additional, Luo, Juan, additional, Hu, Yin, additional, Zhang, Yan, additional, Huang, Jie, additional, Li, Hong‐Ming, additional, Wu, Ben, additional, Liu, Hao‐Ming, additional, Chen, Tuan‐Hui, additional, Qian, Yu‐Xuan, additional, Li, You‐You, additional, Feng, Shi‐Kai, additional, Chen, Yang, additional, Qi, Lu‐Yue, additional, Xu, Ran, additional, Tang, Si‐Yuan, additional, and Xie, Hui, additional

Published
2021
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7. H‐type blood vessels participate in alveolar bone remodeling during murine tooth extraction healing

Author

Yan, Zi‐Qi, primary, Wang, Xiao-Kai, additional, Zhou, Yong, additional, Wang, Zheng‐Guang, additional, Wang, Zhen‐Xing, additional, Jin, Ling, additional, Yin, Hao, additional, Xia, Kun, additional, Tan, Yi‐Juan, additional, Feng, Shi‐Kai, additional, Xie, Ping‐Li, additional, Tang, Si‐Yuan, additional, Fang, Chang‐Yun, additional, Cao, Jia, additional, and Xie, Hui, additional

Published
2020
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8. Deficiency of Omentin-1 leads to delayed fracture healing through excessive inflammation and reduced CD31 hi Emcn hi vessels.

Author

Feng SK, Chen TH, Li HM, Cao J, Liu DB, Rao SS, Liu JH, Zhang Y, Wang ZX, Li YY, Tan YJ, Liu YW, Hong CG, Yan ZQ, Chen ML, Wang YY, Yin H, Jin L, Xie H, Wang ZG, and Zhou Y

Subjects
Animals, Cell Movement, Disease Models, Animal, Female, Femoral Fractures etiology, Femoral Fractures immunology, Gene Knockout Techniques, Mice, Osteoclasts metabolism, Osteogenesis, Platelet Endothelial Cell Adhesion Molecule-1 metabolism, RAW 264.7 Cells, X-Ray Microtomography, Cytokines genetics, Femoral Fractures genetics, Fracture Healing, GPI-Linked Proteins genetics, Lectins genetics, Sialoglycoproteins metabolism
Abstract

Fracture healing is a complicated process affected by many factors, such as inflammatory responses and angiogenesis. Omentin-1 is an adipokine with anti-inflammatory properties, but whether omentin-1 affects the fracture healing process is still unknown. Here, by using global omentin-1 knockout (omentin-1 -/- ) mice, we demonstrated that omentin-1 deficiency resulted in delayed fracture healing in mice, accompanied by increased inflammation and osteoclast formation, and decreased production of platelet-derived growth factor-BB (PDGF-BB) and osteogenesis-promoting vessels that are strongly positive for CD31 and Endomucin (CD31 hi Emcn hi ) in the fracture area. In vitro, omentin-1 treatment suppressed the ability of the tumor necrosis factor-α (TNF-α)-activated macrophages to stimulate multi-nuclear osteoclast formation, resulting in a significant increase in the generation of mono-nuclear preosteoclasts and PDGF-BB, a pro-angiogenic protein that is abundantly secreted by preosteoclasts. PDGF-BB significantly augmented endothelial cell proliferation, tube formation and migration, whereas direct treatment with omentin-1 did not induce obvious effects on angiogenesis activities of endothelial cells. Our study suggests a positive role of omentin-1 in fracture healing, which may be associated with the inhibition of inflammation and stimulation of preosteoclast PDGF-BB-mediated promotion of CD31 hi Emcn hi vessel formation., (Copyright © 2021. Published by Elsevier B.V.)

Published
2021
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