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1. New Trends in Aryl Hydrocarbon Receptor Biology.

2. Dioxin receptor regulates aldehyde dehydrogenase to block melanoma tumorigenesis and metastasis.

3. The Dioxin receptor modulates Caveolin-1 mobilization during directional migration: role of cholesterol.

4. The dioxin receptor controls β1 integrin activation in fibroblasts through a Cbp-Csk-Src pathway.

5. Dioxin receptor expression inhibits basal and transforming growth factor β-induced epithelial-to-mesenchymal transition.

6. A remarkable new target gene for the dioxin receptor: The Vav3 proto-oncogene links AhR to adhesion and migration.

7. Dioxin receptor deficiency impairs angiogenesis by a mechanism involving VEGF-A depletion in the endothelium and transforming growth factor-beta overexpression in the stroma.

8. Loss of dioxin-receptor expression accelerates wound healing in vivo by a mechanism involving TGFbeta.

9. The dioxin receptor regulates the constitutive expression of the vav3 proto-oncogene and modulates cell shape and adhesion.

10. Fitting a xenobiotic receptor into cell homeostasis: how the dioxin receptor interacts with TGFbeta signaling.

11. Recruitment of CREB1 and histone deacetylase 2 (HDAC2) to the mouse Ltbp-1 promoter regulates its constitutive expression in a dioxin receptor-dependent manner.

12. Genome-wide B1 retrotransposon binds the transcription factors dioxin receptor and Slug and regulates gene expression in vivo.

13. The aryl hydrocarbon receptor, more than a xenobiotic-interacting protein.

14. Non-genomic action of resveratrol on androgen and oestrogen receptors in prostate cancer: modulation of the phosphoinositide 3-kinase pathway.

15. Mechanisms involved in resveratrol-induced apoptosis and cell cycle arrest in prostate cancer-derived cell lines.

16. The dioxin receptor is silenced by promoter hypermethylation in human acute lymphoblastic leukemia through inhibition of Sp1 binding.

17. LTBP-1 blockade in dioxin receptor-null mouse embryo fibroblasts decreases TGF-beta activity: Role of extracellular proteases plasmin and elastase.

18. Resveratrol-induced apoptosis in MCF-7 human breast cancer cells involves a caspase-independent mechanism with downregulation of Bcl-2 and NF-kappaB.

19. Overexpression of latent transforming growth factor-beta binding protein 1 (LTBP-1) in dioxin receptor-null mouse embryo fibroblasts.

20. Proteasome inhibition induces nuclear translocation of the dioxin receptor through an Sp1 and protein kinase C-dependent pathway.

21. Down-regulation of CYP1A2 induction during the maturation of mouse cerebellar granule cells in culture: role of nitric oxide accumulation.

22. The antiproliferative activity of resveratrol results in apoptosis in MCF-7 but not in MDA-MB-231 human breast cancer cells: cell-specific alteration of the cell cycle.

23. Proteasome inhibition induces nuclear translocation and transcriptional activation of the dioxin receptor in mouse embryo primary fibroblasts in the absence of xenobiotics.

24. Amelioration of TCDD-induced teratogenesis in aryl hydrocarbon receptor (AhR)-null mice.

25. The involvement of aryl hydrocarbon receptor in the activation of transforming growth factor-beta and apoptosis.

26. Dihydropyrimidine dehydrogenase pharmacogenetics in Caucasian subjects.

27. Lesions of aryl-hydrocarbon receptor-deficient mice.

28. Aryl hydrocarbon receptor knockout mice (AHR-/-) exhibit liver retinoid accumulation and reduced retinoic acid metabolism.

29. Lack of correlation between phenotype and genotype for the polymorphically expressed dihydropyrimidine dehydrogenase in a family of Pakistani origin.

30. Aryl-hydrocarbon receptor-deficient mice are resistant to 2,3,7,8-tetrachlorodibenzo-p-dioxin-induced toxicity.

31. Targeted disruption of specific cytochromes P450 and xenobiotic receptor genes.

32. Targeted disruption of the alpha isoform of the peroxisome proliferator-activated receptor gene in mice results in abolishment of the pleiotropic effects of peroxisome proliferators.

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