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20 results on '"GUI, ZHIFANG"'

7. Global chaos synchronization for modified Chua’s circuit systems via linear state error feedback control

Author

Wu Xiaofeng, Chen Yun, and Gui Zhifang

Subjects
Chua's circuit, Control theory, Synchronization of chaos, Linear system, Trajectory, Master/slave, State (computer science), Algebraic number, Synchronization, Mathematics
Abstract

This paper investigates the global chaos synchronization for master-slave modified Chuapsilas circuit systems coupled by linear state error feedback control. Some sufficient global synchronization criteria are derived in the algebraic inequality form. The numerical examples illustrate the effectiveness of the obtained criteria.

Published
2008

8. Chaos synchronization of master-slave hyperbolic-type generalized Lorenz systems via linear state error feedback control

Author

Gui Zhifang, Wu Xiaofeng, and Chen Yun

Subjects
Exponential stability, Control theory, Control system, Linear system, Symmetric matrix, Master/slave, Robust control, Synchronization, Mathematics
Abstract

This paper proposes the sufficient synchronization criteria for master and slave hyperbolic-type generalized Lorenz systems via linear state error feedback control. An algebraic synchronization criterion for the linear diagonal controller is derived and optimized. The effectiveness of the optimized criterion is verified by numerical example.

Published
2008

19. Global Finite-Time Stabilization of a Chain of Integrators and Its Application.

Author

Gui, Zhifang, Wu, Xiaofeng, and Chen, Yun

Subjects
FEEDBACK control system stability, INTEGRATORS, SYSTEMS theory, CHAOS theory, APPROXIMATION theory, CRITERION (Theory of knowledge)
Abstract

Abstract: This paper investigates the problem of global finite-time stabilization of a chain of integrators. Based on finite-time stability theory of homogeneous system, a criterion is proposed for the global finite-time stabilization of the integrators by a single-state feedback control law. The result is then applied to locally finite-time stabilize a multiscroll saturated chaotic system via homogeneous approximation. [Copyright &y& Elsevier]

Published
2011
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20. Oridonin inhibition and miR‑200b‑3p/ZEB1 axis in human pancreatic cancer.

Author

Gui Z, Luo F, Yang Y, Shen C, Li S, and Xu J

Subjects
Animals, Cell Line, Tumor, Cell Movement drug effects, Cell Proliferation drug effects, Gene Expression Regulation, Neoplastic, Humans, Mice, MicroRNAs biosynthesis, Pancreatic Neoplasms genetics, Pancreatic Neoplasms pathology, Signal Transduction drug effects, Tumor Microenvironment, Zinc Finger E-box-Binding Homeobox 1 biosynthesis, Diterpenes, Kaurane administration & dosage, MicroRNAs genetics, Pancreatic Neoplasms drug therapy, Zinc Finger E-box-Binding Homeobox 1 genetics
Abstract

The relationship among oridonin, miR-200b-3p and pancreatic cancer on epithelial-to-mesenchymal transition (EMT) was investigated for the molecular mechanism or signaling pathways on the migration in pancreatic cancer. BxPC-3 and PANC-1 cells were cultivated and the IC50 of oridonin in BxPC-3 and PANC-1 cells were obtained by the CCK-8 array. The expression of miR‑200b-3p was verified by using real-time PCR and its target gene was predicted. BxPC-3 and PANC-1 cells were treated with oridonin or transfected by miR-200b-3p, those cells were used for western blot assay, Transwell assay, ELISA, immunofluorescence staining, tumorigenesis assay in nude mice and immunohistochemical assay to verify the effects of oridonin or miR-200b-3p on pancreatic cancer. We found that oridonin inhibited the proliferation of BxPC-3 and PANC-1 cells in a dose-dependent manner. miR-200b-3p was downregulated by oridonin in BxPC-3 and PANC-1 cells. ZEB1 was a target gene for miR-200b-3p. Oridonin or overexpression of miR‑200b-3p can inhibit the cell migration in BxPC-3 and PANC-1 cells. miR-200b-3p can inhibit the EMT and oridonin can inhibit the expression of ZEB1, N-cadherin and fibronectin but not increase the expression of E-cadherin, while the cell adhesion molecules ICAM-1 and VCAM-1 were decreased by oridonin in BxPC-3 and PANC-1 cells and the cytoskeleton was altered by oridonin in PANC-1 cells compared with the control. In summary, the results demonstrate that miR‑200b-3p was able to inhibit the EMT of human pancreatic cancer in vivo and in vitro by targeted ZEB1. In vitro, oridonin had a certain effect on the migration in BxPC-3 and PANC-1 cells, but not though type III EMT by miR-200-3p/ZEB1 axis, and may be related to type Ⅱ EMT, tumor microenvironment or altering the cytoskeleton. In vivo, oridonin inhibited the cancer migration in the nude mouse model though inhibiting EMT.

Published
2017
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