432 results on '"Gale, Rosemary E."'
Search Results
2. Author Correction: Genome-wide association study identifies susceptibility loci for acute myeloid leukemia
3. Molecular, clinical and therapeutic determinants of outcome in NPM1 mutated AML
4. Additional impact of mutational genotype on prognostic determination in resistant and relapsed acute myeloid leukaemia
5. Genome-wide association study identifies susceptibility loci for acute myeloid leukemia
6. Genomic landscape and clonal evolution of acute myeloid leukemia with t(8;21): an international study on 331 patients
7. A randomized assessment of adding the kinase inhibitor lestaurtinib to first-line chemotherapy for FLT3-mutated AML
8. Downregulation of the Wnt inhibitor CXXC5 predicts a better prognosis in acute myeloid leukemia
9. Clonality Studies in Essential Thrombocythemia
10. Impact of isolated germline JAK2V617I mutation on human hematopoiesis
11. Genome-wide association study identifies susceptibility loci for acute myeloid leukemia (vol 13, 2, 2022)
12. The replication rate of human hematopoietic stem cells in vivo
13. Therapy for isocitrate dehydrogenase 2 (IDH2)R172‐mutant acute myeloid leukaemia
14. Assessment of Minimal Residual Disease in Standard-Risk AML
15. The value of molecular stratification for CEBPADM and NPM1MUTFLT3WT genotypes in older patients with acute myeloid leukaemia
16. The impact on outcome of the addition of all-trans retinoic acid to intensive chemotherapy in younger patients with nonacute promyelocytic acute myeloid leukemia: overall results and results in genotypic subgroups defined by mutations in NPM1, FLT3, and CEBPA
17. In essential thrombocythemia, multiple JAK2-V617F clones are present in most mutant-positive patients: a new disease paradigm
18. Genome-wide association study identifies susceptibility loci for acute myeloid leukemia
19. Genomic Characterization of Acute Myeloid Leukemia with Aberrations of Chromosome 7: A Multinational Cohort of 523 Patients
20. The impact of FLT3 internal tandem duplication mutant level, number, size, and interaction with NPM1 mutations in a large cohort of young adult patients with acute myeloid leukemia
21. PIM and AKT kinase inhibitors show synergistic cytotoxicity in acute myeloid leukaemia that is associated with convergence on mTOR and MCL1 pathways
22. Insertional mutagenesis combined with acquired somatic mutations causes leukemogenesis following gene therapy of SCID-X1 patients
23. FLT3 tyrosine kinase domain mutations are biologically distinct from and have a significantly more favorable prognosis than FLT3 internal tandem duplications in patients with acute myeloid leukemia
24. GATA2 mutations in sporadic and familial acute myeloid leukaemia patients with CEBPA mutations
25. Does BCR/ABL1 positive Acute Myeloid Leukaemia Exist?
26. An activating mutation in the transmembrane domain of the granulocyte colony-stimulating factor receptor in patients with acute myeloid leukemia
27. Basic sciences of the myeloproliferative diseases: Pathogenic mechanisms of ET and PV
28. Therapy for isocitrate dehydrogenase 2 (IDH2)R172‐mutant acute myeloid leukaemia.
29. Phenotypic heterogeneity and evidence of a founder effect associated with G6PC3 mutations in patients with severe congenital neutropenia
30. G6PC3 mutations are associated with a major defect of glycosylation: a novel mechanism for neutrophil dysfunction
31. The clinical impact of mutant DNMT3A R882 variant allele frequency in acute myeloid leukaemia
32. In trans early mosaic mutational escape and novel phenotypic features of germline SAMD9 mutation
33. No evidence that FLT3 status should be considered as an indicator for transplantation in acute myeloid leukemia (AML): an analysis of 1135 patients, excluding acute promyelocytic leukemia, from the UK MRC AML10 and 12 trials
34. Homozygous HAX1 mutations in severe congenital neutropenia patients with sporadic disease: a novel mutation in two unrelated British kindreds
35. Most acute myeloid leukaemia patients with intermediate mutant FLT3/ITD levels do not have detectable bi-allelic disease, indicating that heterozygous disease alone is associated with an adverse outcome
36. Acute myeloid leukaemia blast cells with a tyrosine kinase domain mutation of FLT3 are less sensitive to lestaurtinib than those with a FLT3 internal tandem duplication
37. FLT3 MUTATIONS AND LEUKAEMIA
38. Long-term follow-up of granulocyte colony-stimulating factor receptor mutations in patients with severe congenital neutropenia: implications for leukaemogenesis and therapy
39. No evidence that CD33 splicing SNP impacts the response to GO in younger adults with AML treated on UK MRC/NCRI trials
40. Lack of pathogenic mutations in the 5′-untranslated region of the thrombopoietin gene in patients with non-familial essential thrombocythaemia
41. Dynamics of telomere shortening in neutrophils and T lymphocytes during ageing and the relationship to skewed X chromosome inactivation patterns
42. Platelet c-mpl expression is dysregulated in patients with essential thrombocythaemia but this is not of diagnostic value
43. Analysis of the clinical impact of NPM1 mutant allele burden in a large cohort of younger adult patients with acute myeloid leukaemia
44. Activating point mutations in the betaC chain of the GM-CSF, IL-3 and IL-5 receptors are not a major contributory factor in the pathogenesis of acute myeloid leukaemia
45. The activating splice mutation in intron 3 of the thrombopoietin gene is not found in patients with non-familial essential thrombocythaemia
46. Mutations of the granulocyte-colony stimulating factor receptor in patients with severe congenital neutropenia are not required for transformation to acute myeloid leukaemia and may be a bystander phenomenon
47. Acquired skewing of X-chromosome inactivation patterns in myeloid cells of the elderly suggests stochastic clonal loss with age
48. Activation of the LMO2 oncogene through a somatically acquired neomorphic promoter in T-cell acute lymphoblastic leukemia
49. The retinoblastoma gene (rb1) in acute myeloid leukaemia: analysis of gene rearrangements, protein expression and comparison of disease outcome
50. Analysis of granulocyte colony stimulating factor receptor isoforms, polymorphisms and mutations in normal haemopoietic cells and acute myeloid leukaemia blasts
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