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191 results on '"Galley, H. F."'

18. Posters

Author

Yao, Y. M., Tian, H. M., Liang, H. P., Yu, Y., Lu, L. R., Wang, Y. P., Sheng, Z. Y., Reith, H. B., Holzheimer, R. G., Thiede, A., Galstian, G. M., Danilina, A. V., Gorodetsky, V. M., Tutelian, A. V., Galley, H. F., Webster, N. R., Djugehev, A. N., Fomin, M. D., Satalkin, A. A., Sokolov, V. A., Maskin, B., Fontán, P., Spinedi, E., Badolati, A., Endo, S., Kasai, T., Inada, K., Takakuwa, T., Yamada, Y., Suzuki, T., Taniguchi, S., Kern, H., Wittich, R., Schaffartzik, W., Kox, W., Spies, C., Ilkka, L., Takala, J., Paiva, J. A., Sousa-Dias, C., Bodas, A., Ramos, J. P., Candeias, J., Pereira, A. C., Torrinha, F., Ribeiro, T., Milting, K., Sanft, C., Brede, K., Beller, S., Andresen, M., Dougnac, A., Letelier, L. M., Díaz, O., Laterre, F. F., Reynaert, M., Valdivieso, A., Geppert, A., Zorn, G., Huber, K., Siostrzonek, P., Oberhoffer, M., Bögel, D., Meier-Hellmann, A., Vogelsang, H., Reinhart, K., Tsidemiadou, F., Farmakis, M., Bobota, A., Pragastis, D., Bilancia, R., Posca, A., Margiotta, D., Spampani, E., Roselli, P., Caputo, G., Thio, J. M., Sinaasappel, M., Ince, C., Berger, D., Boelke, E., Hiki, N., Poch, B., Beier, A., Graf, M., Seidelmann, M., Beger, H. G., Nuvials, X., Ruiz Rodriguez, J. C., Martin, M. C., Esteban, F., Garcia-Allut, J. L., Burgueno, M. J., Mourelle, M., Monasterio, J., Angles, A., Boveda, J. L., Salgado, A., Segura, R. M., Sauri, R., Beck, B., Schimmer, R. C., Pasch, T., Ward, P. A., Gruson, D., Hilbert, G., Roux, C., Coulon, V., Juzan, M., Laffort, P., Parrens, E., Gualde, N., Gbikpi-Benissan, G., Cardinaud, J. P., Shi, Hanping, Xu, Renbao, Gao, Han, Marenović, T., Miloŝević, D., Brkan, Z., Ŝurbatović, M., Gundelach, K., Engelmann, L., Pilz, U., Werner, M., Reiger, J., Tatzber, F., Oberbichler, A., Grimm, G., de Abreu, M. Gama, Kirschfink, M., Quintel, M., Albrecht, D. M., Ragaller, M., Nakae, H., Koike, S., Mavrommatis, A., Theodoridis, Th., Stavropoulos, G., Zakynthinos, S., Orfanidou, A., Sfyras, D., Christopoulou-Kokkinou, V., Roussos, Ch., Właszczyk, A., Adamik, B., Zimecki, M., Kübler, A., Berezowicz, P. S., Vasilcov, V., Kelina, N., Gengin, M., Mitroshina, S., Filippova, L., Levachova, O., Gürlich, R., Maruna, P., and Čermák, J.

Published
1996
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19. Free Papers

Author

Emmanuel, G., Carranza, S., Vettichira, S., Paradis, M., Lie, T., Kessler, P., Schini-Kerth, V. B., Koh, Y., Hybertson, B. M., Jepson, E. K., Kim, M. J., Lee, I., Lim, C. M., Lee, S. D., Kim, W. S., Kim, D. S., Kim, W. D., Repine, J. E., Takeda, T., Yukioka, T., Matsuda, H., Shimazaki, S., Kammermeier, D., Birkmann, J., Blinzler, L., Gallmeier, W. M., Heuser, D., Jourdain, M., Mangalaboyi, J., Carrette, O., Toumoys, A., Fourrier, F., Mizon, J., Chopin, C., Peake, S. L., Carter, J. K., Russ, R. G., Pierides, J., Leppard, J. P., Porter, K., Galley, H. F., Webster, N. R., Wigmore, S. J., Walsh, T. S., Hopton, P., Lee, A., Ross, J. A., Majcherczyk, P. A., Pugin, J., Glauser, M. P., Heumann, D., Zanetti, G., de Werra, I., Sablotzki, A., Welters, I., Menges, T., Görlach, G., Lehmann, N., Hempelmann, G., Giannitsis, E., Tettenborn, I., Stierle, U., Dalhoff, K., Sheikhzadeh, A., Diederich, K. W., Gant, V., Maciver, C., Treacher, D. F., Lamy, M., Thijs, L. G., Eisele, B., Keinecke, H. -O., Schuster, H. P., Matthias, F. R., Oettinger, W., Hartenauer, U., Fourrier, F., Delvos, U., Stott, S. A., Noble, D. W., Schobersberger, W., Hoffmann, G., Fandrey, J., Bellingan, G. J., Caldwell, H., Dransfield, I., Howie, S. M., Haslett, C., Liaudet, L., Feihl, F., Markert, M., Perret, C., Williams, M. A., Rhoades, C., White, S. A., Miller, J., Withington, S., Newland, A. C., Kelsey, S. M., Froon, A., Bonten, M., Gaillard, C., Greve, J., de Leeuw, P., Drent, M., Stobberingh, E., Buurman, W., Fourcade, O., Simon, M. -F., Leballe, F., Ashraf, R., Sarda, L., Cathala, B., Chap, H., Reper, P., Danckaert, R., Jeunen, R., Bruyns, T., De Hemptinne, J., and Vanderkelen, A.

Published
1996
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35. Volatile anaesthetics reduce neutrophil inflammatory response by interfering with CXC receptor-2 signalling

Author

Müller-Edenborn, B., Frick, R., Piegeler, T., Schläpfer, M., Roth-Z'graggen, B., Schlicker, A., Beck-Schimmer, B., and Galley, H. F.

Subjects
hemic and immune systems, respiratory system, biological factors
Abstract

Background Growing evidence suggests a protective effect of volatile anaesthetics in ischaemia-reperfusion (I/R)-injury, and the accumulation of neutrophils is a crucial event. Pro-inflammatory cytokines carrying the C-X-C-motif including interleukin-8 (IL-8) and CXC-ligand 1 (CXCL1) activate CXC receptor-1 (CXCR1; stimulated by IL-8), CXC receptor-2 (CXCR2; stimulated by IL-8 and CXCL1), or both to induce CD11b-dependent neutrophil transmigration. Inhibition of CXCR1, CXCR2, or both reduces I/R-injury by preventing neutrophil accumulation. We hypothesized that interference with CXCR1/CXCR2 signalling contributes to the well-established beneficial effect of volatile anaesthetics in I/R-injury. Methods Isolated human neutrophils were stimulated with IL-8 or CXCL1 and exposed to volatile anaesthetics (sevoflurane/desflurane). Neutrophil migration was assessed using an adapted Boyden chamber. Expression of CD11b, CXCR1, and CXCR2 was measured by flow cytometry. Blocking antibodies against CXCR1/CXCR2/CD11b and phorbol myristate acetate were used to investigate specific pathways. Results Volatile anaesthetics reduced CD11b-dependent neutrophil transmigration induced by IL-8 by >30% and CD11b expression by 18 and 27% with sevoflurane/desflurane, respectively. This effect was independent of CXCR1/CXCR2 expression and CXCR1/CXCR2 endocytosis. Inhibition of CXCR1 signalling did not affect downregulation of CD11b with volatile anaesthetics. Blocking of CXCR2-signalling neutralized effects by volatile anaesthetics on CD11b expression. Specific stimulation of CXCR2 with CXCL1 was sufficient to induce upregulation of CD11b, which was impaired with volatile anaesthetics. No effect of volatile anaesthetics was observed with direct stimulation of protein kinase C located downstream of CXCR1/CXCR2. Conclusion Volatile anaesthetics attenuate neutrophil inflammatory responses elicited by CXC cytokines through interference with CXCR2 signalling. This might contribute to the beneficial effect of volatile anaesthetics in I/R-injury

Published
2017

36. Reliability of lithium dilution cardiac output in anaesthetized sheep

Author

Axiak Flammer, S. M., Critchley, L. A., Weber, A., Pirbodaghi, T., Brinks, H., Vandenberghe, S., and Galley, H. F.

Abstract

Background Cardiac output (CO) measurement with lithium dilution (COLD) has not been fully validated in sheep using precise ultrasonic flow probe technology (COUFP). Sheep generate important cardiovascular research models and the use of COLD has become more popular in experimental settings. Methods Ultrasonic transit-time perivascular flow probes were surgically implanted on the pulmonary artery of 13 sheep. Paired COLD readings were taken at six time points, before and after implantation of a left ventricular assist device (LVAD) and compared with COUFP recorded just after lithium injection. Results The mean COLD was 5.7 litre min−1 (range 3.8-9.6 litre min−1) and mean COUFP 5.9 litre min−1 (range 4.0-9.2 litre min−1). The bias (standard deviation) was 0.3 (1.0) litre min−1 [5.1 (16.9)%] and limits of agreement (LOA) were −1.7 to 2.3 litre min−1 (−28.8 to 39.0%) with a percentage error (PE) of 34.4%. Data to assess trending [rate (95% confidence intervals)] included a 78 (62-93)% concordance rate in the four-quadrant plot (n=27). In the half moon polar plot (n=19), the mean polar angle was +5°, the radial LOA were −49 to +35° and 68 (47-89)% of data points fell within 22.5° of the mean polar angle. Both tests indicated moderate to poor trending ability. Conclusion COLD is not precise when evaluated against COUFP in sheep based on the statistical criteria set, but the results are comparable with previously published animal studies

Published
2017

37. Effects of early and late diabetic neuropathy on sciatic nerve block duration and neurotoxicity in Zucker diabetic fatty rats

Author

Lirk, P., Verhamme, C., Boeckh, R., Stevens, M. F., ten Hoope, W., Gerner, P., Blumenthal, S., de Girolami, U., van Schaik, I. N., Hollmann, M. W., Picardi, S., and Galley, H. F.

Abstract

Background The neuropathy of type II diabetes mellitus (DM) is increasing in prevalence worldwide. We aimed to test the hypothesis that in a rodent model of type II DM, neuropathy would lead to increased neurotoxicity and block duration after lidocaine-induced sciatic nerve block when compared with control animals. Methods Experiments were carried out in Zucker diabetic fatty rats aged 10 weeks (early diabetic) or 18 weeks (late diabetic, with or without insulin 3 units per day), and age-matched healthy controls. Left sciatic nerve block was performed using 0.2 ml lidocaine 2%. Nerve conduction velocity (NCV) and F-wave latency were used to quantify nerve function before, and 1 week after nerve block, after which sciatic nerves were used for neurohistopathology. Results Early diabetic animals did not show increased signs of nerve dysfunction after nerve block. In late diabetic animals without insulin vs control animals, NCV was 34.8 (5.0) vs 41.1 (4.1) ms s−1 (P

Published
2017

41. A reply

Author

Galley, H. F., primary, Yassen, K., additional, and Webster, N. R., additional

Published
2002
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