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897 results on '"Gerald, William"'

1. MYC cooperates with AKT in prostate tumorigenesis and alters sensitivity to mTOR inhibitors.

Author

Clegg, Nicola J, Couto, Suzana S, Wongvipat, John, Hieronymus, Haley, Carver, Brett S, Taylor, Barry S, Ellwood-Yen, Katharine, Gerald, William L, Sander, Chris, and Sawyers, Charles L

Subjects
Prostate, Animals, Mice, Transgenic, Humans, Mice, Prostatic Intraepithelial Neoplasia, Prostatic Neoplasms, Neoplasm Invasiveness, Precancerous Conditions, Disease Models, Animal, Disease Progression, Proto-Oncogene Proteins c-myc, Protein Kinase Inhibitors, Signal Transduction, Apoptosis, Enzyme Activation, Protein Binding, Phenotype, Male, Proto-Oncogene Proteins c-akt, TOR Serine-Threonine Kinases, Disease Models, Animal, Transgenic, General Science & Technology
Abstract

MYC and phosphoinositide 3-kinase (PI3K)-pathway deregulation are common in human prostate cancer. Through examination of 194 human prostate tumors, we observed statistically significant co-occurrence of MYC amplification and PI3K-pathway alteration, raising the possibility that these two lesions cooperate in prostate cancer progression. To investigate this, we generated bigenic mice in which both activated human AKT1 and human MYC are expressed in the prostate (MPAKT/Hi-MYC model). In contrast to mice expressing AKT1 alone (MPAKT model) or MYC alone (Hi-MYC model), the bigenic phenotype demonstrates accelerated progression of mouse prostate intraepithelial neoplasia (mPIN) to microinvasive disease with disruption of basement membrane, significant stromal remodeling and infiltration of macrophages, B- and T-lymphocytes, similar to inflammation observed in human prostate tumors. In contrast to the reversibility of mPIN lesions in young MPAKT mice after treatment with mTOR inhibitors, Hi-MYC and bigenic MPAKT/Hi-MYC mice were resistant. Additionally, older MPAKT mice showed reduced sensitivity to mTOR inhibition, suggesting that additional genetic events may dampen mTOR dependence. Since increased MYC expression is an early feature of many human prostate cancers, these data have implications for treatment of human prostate cancers with PI3K-pathway alterations using mTOR inhibitors.

Published
2011

2. Stat3 Mediates Expression of Autotaxin in Breast Cancer

Author

Azare, Janeen, Doane, Ashley, Leslie, Kenneth, Chang, Qing, Berishaj, Marjan, Nnoli, Jennifer, Mark, Kevin, Al-Ahmadie, Hikmat, Gerald, William, Hassimi, Maryam, Viale, Agnes, Stracke, Mary, Lyden, David, and Bromberg, Jacqueline

Subjects
Biochemistry and Cell Biology, Biomedical and Clinical Sciences, Oncology and Carcinogenesis, Biological Sciences, Cancer, Breast Cancer, Genetics, Breast Neoplasms, Cell Line, Tumor, Cluster Analysis, Female, Gene Expression Regulation, Neoplastic, Genes, Neoplasm, Humans, Lymph Nodes, Neoplasm Metastasis, Phosphoric Diester Hydrolases, Phosphorylation, Phosphotyrosine, STAT3 Transcription Factor, General Science & Technology
Abstract

We determined that signal transducer and activator of transcription 3 (Stat3) is tyrosine phosphorylated in 37% of primary breast tumors and 63% of paired metastatic axillary lymph nodes. Examination of the distribution of tyrosine phosphorylated (pStat3) in primary tumors revealed heterogenous expression within the tumor with the highest levels found in cells on the edge of tumors with relatively lower levels in the central portion of tumors. In order to determine Stat3 target genes that may be involved in migration and metastasis, we identified those genes that were differentially expressed in primary breast cancer samples as a function of pStat3 levels. In addition to known Stat3 transcriptional targets (Twist, Snail, Tenascin-C and IL-8), we identified ENPP2 as a novel Stat3 regulated gene, which encodes autotaxin (ATX), a secreted lysophospholipase which mediates mammary tumorigenesis and cancer cell migration. A positive correlation between nuclear pStat3 and ATX was determined by immunohistochemical analysis of primary breast cancer samples and matched axillary lymph nodes and in several breast cancer derived cell lines. Inhibition of pStat3 or reducing Stat3 expression led to a decrease in ATX levels and cell migration. An association between Stat3 and the ATX promoter, which contains a number of putative Stat3 binding sites, was determined by chromatin immunoprecipitation. These observations suggest that activated Stat3 may regulate the migration of breast cancer cells through the regulation of ATX.

Published
2011

5. Copy number and gene expression differences between African American and Caucasian American prostate cancer

Author

Rose, Amy E, Satagopan, Jaya M, Oddoux, Carole, Zhou, Qin, Xu, Ruliang, Olshen, Adam B, Yu, Jessie Z, Dash, Atreya, Jean-Gilles, Jerome, Reuter, Victor, Gerald, William L, Lee, Peng, and Osman, Iman

Abstract

Abstract Background The goal of our study was to investigate the molecular underpinnings associated with the relatively aggressive clinical behavior of prostate cancer (PCa) in African American (AA) compared to Caucasian American (CA) patients using a genome-wide approach. Methods AA and CA patients treated with radical prostatectomy (RP) were frequency matched for age at RP, Gleason grade, and tumor stage. Array-CGH (BAC SpectralChip2600) was used to identify genomic regions with significantly different DNA copy number between the groups. Gene expression profiling of the same set of tumors was also evaluated using Affymetrix HG-U133 Plus 2.0 arrays. Concordance between copy number alteration and gene expression was examined. A second aCGH analysis was performed in a larger validation cohort using an oligo-based platform (Agilent 244K). Results BAC-based array identified 27 chromosomal regions with significantly different copy number changes between the AA and CA tumors in the first cohort (Fisher's exact test, P < 0.05). Copy number alterations in these 27 regions were also significantly associated with gene expression changes. aCGH performed in a larger, independent cohort of AA and CA tumors validated 4 of the 27 (15%) most significantly altered regions from the initial analysis (3q26, 5p15-p14, 14q32, and 16p11). Functional annotation of overlapping genes within the 4 validated regions of AA/CA DNA copy number changes revealed significant enrichment of genes related to immune response. Conclusions Our data reveal molecular alterations at the level of gene expression and DNA copy number that are specific to African American and Caucasian prostate cancer and may be related to underlying differences in immune response.

Published
2010

7. Editor's Note: Activator Protein-1 Transcription Factors Are Associated with Progression and Recurrence of Prostate Cancer

Author

Ouyang, Xuesong, primary, Jessen, Walter J., additional, Al-Ahmadie, Hikmat, additional, Serio, Angel M., additional, Lin, Yong, additional, Shih, Weichung-Joseph, additional, Reuter, Victor E., additional, Scardino, Peter T., additional, Shen, Michael M., additional, Aronow, Bruce J., additional, Vickers, Andrew J., additional, Gerald, William L., additional, and Abate-Shen, Cory, additional

Published
2023
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8. Mutations in the EGFR kinase domain mediate STAT3 activation via IL-6 production in human lung adenocarcinomas.

Author

Gao, Sizhi Paul, Mark, Kevin G, Leslie, Kenneth, Pao, William, Motoi, Noriko, Gerald, William L, Travis, William D, Bornmann, William, Veach, Darren, Clarkson, Bayard, and Bromberg, Jacqueline F

Subjects
Cell Line, Tumor, Animals, Humans, Mice, Mice, Nude, Adenocarcinoma, Lung Neoplasms, RNA, Small Interfering, Interleukin-6, Enzyme Inhibitors, Neoplasm Transplantation, Signal Transduction, Transcription, Genetic, Gene Expression Regulation, Neoplastic, Phosphorylation, Mutation, Female, STAT3 Transcription Factor, Cytokine Receptor gp130, Janus Kinases, ErbB Receptors, Lung, Cancer, Lung Cancer, Medical and Health Sciences, Immunology
Abstract

Persistently activated or tyrosine-phosphorylated STAT3 (pSTAT3) is found in 50% of lung adenocarcinomas. pSTAT3 is found in primary adenocarcinomas and cell lines harboring somatic-activating mutations in the tyrosine kinase domain of EGFR. Treatment of cell lines with either an EGFR inhibitor or an src kinase inhibitor had no effect on pSTAT3 levels, whereas a pan-JAK inhibitor (P6) blocked activation of STAT3 and inhibited tumorigenesis. Cell lines expressing these persistently activated mutant EGFRs also produced high IL-6 levels, and blockade of the IL-6/gp130/JAK pathway led to a decrease in pSTAT3 levels. In addition, reduction of IL-6 levels by RNA interference led to a decrease in tumorigenesis. Introduction of persistently activated EGFR into immortalized breast epithelial cells led to tumorigenesis, IL-6 expression, and STAT3 activation, all of which could be inhibited with P6 or gp130 blockade. Furthermore, inhibition of EGFR activity in multiple cell lines partially blocked transcription of IL-6 and concurrently decreased production and release of IL-6. Finally, immunohistochemical analysis revealed a positive correlation between pSTAT3 and IL-6 positivity in primary lung adenocarcinomas. Therefore, mutant EGFR could activate the gp130/JAK/STAT3 pathway by means of IL-6 upregulation in primary human lung adenocarcinomas, making this pathway a potential target for cancer treatment.

Published
2007

9. Multiclass Cancer Diagnosis Using Tumor Gene Expression Signatures

Author

Ramaswamy, Sridhar, Tamayo, Pablo, Rifkin, Ryan, Mukherjee, Sayan, Yeang, Chen-Hsiang, Angelo, Michael, Ladd, Christine, Reich, Michael, Latulippe, Eva, Mesirov, Jill P., Poggio, Tomaso, Gerald, William, Loda, Massimo, Lander, Eric S., and Golub, Todd R.

Published
2001

12. Supplementary Table 1 from GROα Is Highly Expressed in Adenocarcinoma of the Colon and Down-Regulates Fibulin-1

Author

Wen, Yu, primary, Giardina, Sarah F., primary, Hamming, David, primary, Greenman, Jennifer, primary, Zachariah, Emmanuel, primary, Bacolod, Manny D., primary, Liu, Hao, primary, Shia, Jinru, primary, Amenta, Peter S., primary, Barany, Francis, primary, Paty, Phillip, primary, Gerald, William, primary, and Notterman, Daniel, primary

Published
2023
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13. Data from Global Levels of Specific Histone Modifications and an Epigenetic Gene Signature Predict Prostate Cancer Progression and Development

Author

Bianco-Miotto, Tina, primary, Chiam, Karen, primary, Buchanan, Grant, primary, Jindal, Shalini, primary, Day, Tanya K., primary, Thomas, Mervyn, primary, Pickering, Marie A., primary, O'Loughlin, Melissa A., primary, Ryan, Natalie K., primary, Raymond, Wendy A., primary, Horvath, Lisa G., primary, Kench, James G., primary, Stricker, Phillip D., primary, Marshall, Villis R., primary, Sutherland, Robert L., primary, Henshall, Susan M., primary, Gerald, William L., primary, Scher, Howard I., primary, Risbridger, Gail P., primary, Clements, Judith A., primary, Butler, Lisa M., primary, Tilley, Wayne D., primary, Horsfall, David J., primary, and Ricciardelli, Carmela, primary

Published
2023
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14. Supplementary Figure 1 from Global Levels of Specific Histone Modifications and an Epigenetic Gene Signature Predict Prostate Cancer Progression and Development

Author

Bianco-Miotto, Tina, primary, Chiam, Karen, primary, Buchanan, Grant, primary, Jindal, Shalini, primary, Day, Tanya K., primary, Thomas, Mervyn, primary, Pickering, Marie A., primary, O'Loughlin, Melissa A., primary, Ryan, Natalie K., primary, Raymond, Wendy A., primary, Horvath, Lisa G., primary, Kench, James G., primary, Stricker, Phillip D., primary, Marshall, Villis R., primary, Sutherland, Robert L., primary, Henshall, Susan M., primary, Gerald, William L., primary, Scher, Howard I., primary, Risbridger, Gail P., primary, Clements, Judith A., primary, Butler, Lisa M., primary, Tilley, Wayne D., primary, Horsfall, David J., primary, and Ricciardelli, Carmela, primary

Published
2023
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15. Supplementary Figure 3 from Global Levels of Specific Histone Modifications and an Epigenetic Gene Signature Predict Prostate Cancer Progression and Development

Author

Bianco-Miotto, Tina, primary, Chiam, Karen, primary, Buchanan, Grant, primary, Jindal, Shalini, primary, Day, Tanya K., primary, Thomas, Mervyn, primary, Pickering, Marie A., primary, O'Loughlin, Melissa A., primary, Ryan, Natalie K., primary, Raymond, Wendy A., primary, Horvath, Lisa G., primary, Kench, James G., primary, Stricker, Phillip D., primary, Marshall, Villis R., primary, Sutherland, Robert L., primary, Henshall, Susan M., primary, Gerald, William L., primary, Scher, Howard I., primary, Risbridger, Gail P., primary, Clements, Judith A., primary, Butler, Lisa M., primary, Tilley, Wayne D., primary, Horsfall, David J., primary, and Ricciardelli, Carmela, primary

Published
2023
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16. Supplementary Figure 2 from Global Levels of Specific Histone Modifications and an Epigenetic Gene Signature Predict Prostate Cancer Progression and Development

Author

Bianco-Miotto, Tina, primary, Chiam, Karen, primary, Buchanan, Grant, primary, Jindal, Shalini, primary, Day, Tanya K., primary, Thomas, Mervyn, primary, Pickering, Marie A., primary, O'Loughlin, Melissa A., primary, Ryan, Natalie K., primary, Raymond, Wendy A., primary, Horvath, Lisa G., primary, Kench, James G., primary, Stricker, Phillip D., primary, Marshall, Villis R., primary, Sutherland, Robert L., primary, Henshall, Susan M., primary, Gerald, William L., primary, Scher, Howard I., primary, Risbridger, Gail P., primary, Clements, Judith A., primary, Butler, Lisa M., primary, Tilley, Wayne D., primary, Horsfall, David J., primary, and Ricciardelli, Carmela, primary

Published
2023
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17. Supplementary Figure 5 from Global Levels of Specific Histone Modifications and an Epigenetic Gene Signature Predict Prostate Cancer Progression and Development

Author

Bianco-Miotto, Tina, primary, Chiam, Karen, primary, Buchanan, Grant, primary, Jindal, Shalini, primary, Day, Tanya K., primary, Thomas, Mervyn, primary, Pickering, Marie A., primary, O'Loughlin, Melissa A., primary, Ryan, Natalie K., primary, Raymond, Wendy A., primary, Horvath, Lisa G., primary, Kench, James G., primary, Stricker, Phillip D., primary, Marshall, Villis R., primary, Sutherland, Robert L., primary, Henshall, Susan M., primary, Gerald, William L., primary, Scher, Howard I., primary, Risbridger, Gail P., primary, Clements, Judith A., primary, Butler, Lisa M., primary, Tilley, Wayne D., primary, Horsfall, David J., primary, and Ricciardelli, Carmela, primary

Published
2023
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18. Supplementary Figure A from GROα Is Highly Expressed in Adenocarcinoma of the Colon and Down-Regulates Fibulin-1

Author

Wen, Yu, primary, Giardina, Sarah F., primary, Hamming, David, primary, Greenman, Jennifer, primary, Zachariah, Emmanuel, primary, Bacolod, Manny D., primary, Liu, Hao, primary, Shia, Jinru, primary, Amenta, Peter S., primary, Barany, Francis, primary, Paty, Phillip, primary, Gerald, William, primary, and Notterman, Daniel, primary

Published
2023
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19. Supplementary Table 2 from Global Levels of Specific Histone Modifications and an Epigenetic Gene Signature Predict Prostate Cancer Progression and Development

Author

Bianco-Miotto, Tina, primary, Chiam, Karen, primary, Buchanan, Grant, primary, Jindal, Shalini, primary, Day, Tanya K., primary, Thomas, Mervyn, primary, Pickering, Marie A., primary, O'Loughlin, Melissa A., primary, Ryan, Natalie K., primary, Raymond, Wendy A., primary, Horvath, Lisa G., primary, Kench, James G., primary, Stricker, Phillip D., primary, Marshall, Villis R., primary, Sutherland, Robert L., primary, Henshall, Susan M., primary, Gerald, William L., primary, Scher, Howard I., primary, Risbridger, Gail P., primary, Clements, Judith A., primary, Butler, Lisa M., primary, Tilley, Wayne D., primary, Horsfall, David J., primary, and Ricciardelli, Carmela, primary

Published
2023
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20. Supplementary Figure 4 from Global Levels of Specific Histone Modifications and an Epigenetic Gene Signature Predict Prostate Cancer Progression and Development

Author

Bianco-Miotto, Tina, primary, Chiam, Karen, primary, Buchanan, Grant, primary, Jindal, Shalini, primary, Day, Tanya K., primary, Thomas, Mervyn, primary, Pickering, Marie A., primary, O'Loughlin, Melissa A., primary, Ryan, Natalie K., primary, Raymond, Wendy A., primary, Horvath, Lisa G., primary, Kench, James G., primary, Stricker, Phillip D., primary, Marshall, Villis R., primary, Sutherland, Robert L., primary, Henshall, Susan M., primary, Gerald, William L., primary, Scher, Howard I., primary, Risbridger, Gail P., primary, Clements, Judith A., primary, Butler, Lisa M., primary, Tilley, Wayne D., primary, Horsfall, David J., primary, and Ricciardelli, Carmela, primary

Published
2023
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21. Supplementary Figure Legend from Global Levels of Specific Histone Modifications and an Epigenetic Gene Signature Predict Prostate Cancer Progression and Development

Author

Bianco-Miotto, Tina, primary, Chiam, Karen, primary, Buchanan, Grant, primary, Jindal, Shalini, primary, Day, Tanya K., primary, Thomas, Mervyn, primary, Pickering, Marie A., primary, O'Loughlin, Melissa A., primary, Ryan, Natalie K., primary, Raymond, Wendy A., primary, Horvath, Lisa G., primary, Kench, James G., primary, Stricker, Phillip D., primary, Marshall, Villis R., primary, Sutherland, Robert L., primary, Henshall, Susan M., primary, Gerald, William L., primary, Scher, Howard I., primary, Risbridger, Gail P., primary, Clements, Judith A., primary, Butler, Lisa M., primary, Tilley, Wayne D., primary, Horsfall, David J., primary, and Ricciardelli, Carmela, primary

Published
2023
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22. Supplementary Figure B from GROα Is Highly Expressed in Adenocarcinoma of the Colon and Down-Regulates Fibulin-1

Author

Wen, Yu, primary, Giardina, Sarah F., primary, Hamming, David, primary, Greenman, Jennifer, primary, Zachariah, Emmanuel, primary, Bacolod, Manny D., primary, Liu, Hao, primary, Shia, Jinru, primary, Amenta, Peter S., primary, Barany, Francis, primary, Paty, Phillip, primary, Gerald, William, primary, and Notterman, Daniel, primary

Published
2023
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23. Supplementary Figure and Table Legends from GROα Is Highly Expressed in Adenocarcinoma of the Colon and Down-Regulates Fibulin-1

Author

Wen, Yu, primary, Giardina, Sarah F., primary, Hamming, David, primary, Greenman, Jennifer, primary, Zachariah, Emmanuel, primary, Bacolod, Manny D., primary, Liu, Hao, primary, Shia, Jinru, primary, Amenta, Peter S., primary, Barany, Francis, primary, Paty, Phillip, primary, Gerald, William, primary, and Notterman, Daniel, primary

Published
2023
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24. Supplementary Table 1 from Global Levels of Specific Histone Modifications and an Epigenetic Gene Signature Predict Prostate Cancer Progression and Development

Author

Bianco-Miotto, Tina, primary, Chiam, Karen, primary, Buchanan, Grant, primary, Jindal, Shalini, primary, Day, Tanya K., primary, Thomas, Mervyn, primary, Pickering, Marie A., primary, O'Loughlin, Melissa A., primary, Ryan, Natalie K., primary, Raymond, Wendy A., primary, Horvath, Lisa G., primary, Kench, James G., primary, Stricker, Phillip D., primary, Marshall, Villis R., primary, Sutherland, Robert L., primary, Henshall, Susan M., primary, Gerald, William L., primary, Scher, Howard I., primary, Risbridger, Gail P., primary, Clements, Judith A., primary, Butler, Lisa M., primary, Tilley, Wayne D., primary, Horsfall, David J., primary, and Ricciardelli, Carmela, primary

Published
2023
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27. Data from Integrative Genomics Identifies Distinct Molecular Classes of Neuroblastoma and Shows That Multiple Genes Are Targeted by Regional Alterations in DNA Copy Number

Author

Wang, Qun, primary, Diskin, Sharon, primary, Rappaport, Eric, primary, Attiyeh, Edward, primary, Mosse, Yael, primary, Shue, Daniel, primary, Seiser, Eric, primary, Jagannathan, Jayanti, primary, Shusterman, Suzanne, primary, Bansal, Manisha, primary, Khazi, Deepa, primary, Winter, Cynthia, primary, Okawa, Erin, primary, Grant, Gregory, primary, Cnaan, Avital, primary, Zhao, Huaqing, primary, Cheung, Nai-Kong, primary, Gerald, William, primary, London, Wendy, primary, Matthay, Katherine K., primary, Brodeur, Garrett M., primary, and Maris, John M., primary

Published
2023
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28. Supplementary Table 2 from Integrative Genomics Identifies Distinct Molecular Classes of Neuroblastoma and Shows That Multiple Genes Are Targeted by Regional Alterations in DNA Copy Number

Author

Wang, Qun, primary, Diskin, Sharon, primary, Rappaport, Eric, primary, Attiyeh, Edward, primary, Mosse, Yael, primary, Shue, Daniel, primary, Seiser, Eric, primary, Jagannathan, Jayanti, primary, Shusterman, Suzanne, primary, Bansal, Manisha, primary, Khazi, Deepa, primary, Winter, Cynthia, primary, Okawa, Erin, primary, Grant, Gregory, primary, Cnaan, Avital, primary, Zhao, Huaqing, primary, Cheung, Nai-Kong, primary, Gerald, William, primary, London, Wendy, primary, Matthay, Katherine K., primary, Brodeur, Garrett M., primary, and Maris, John M., primary

Published
2023
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29. Supplementary Table 3 from Integrative Genomics Identifies Distinct Molecular Classes of Neuroblastoma and Shows That Multiple Genes Are Targeted by Regional Alterations in DNA Copy Number

Author

Wang, Qun, primary, Diskin, Sharon, primary, Rappaport, Eric, primary, Attiyeh, Edward, primary, Mosse, Yael, primary, Shue, Daniel, primary, Seiser, Eric, primary, Jagannathan, Jayanti, primary, Shusterman, Suzanne, primary, Bansal, Manisha, primary, Khazi, Deepa, primary, Winter, Cynthia, primary, Okawa, Erin, primary, Grant, Gregory, primary, Cnaan, Avital, primary, Zhao, Huaqing, primary, Cheung, Nai-Kong, primary, Gerald, William, primary, London, Wendy, primary, Matthay, Katherine K., primary, Brodeur, Garrett M., primary, and Maris, John M., primary

Published
2023
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30. Supplementary Table 1 from Integrative Genomics Identifies Distinct Molecular Classes of Neuroblastoma and Shows That Multiple Genes Are Targeted by Regional Alterations in DNA Copy Number

Author

Wang, Qun, primary, Diskin, Sharon, primary, Rappaport, Eric, primary, Attiyeh, Edward, primary, Mosse, Yael, primary, Shue, Daniel, primary, Seiser, Eric, primary, Jagannathan, Jayanti, primary, Shusterman, Suzanne, primary, Bansal, Manisha, primary, Khazi, Deepa, primary, Winter, Cynthia, primary, Okawa, Erin, primary, Grant, Gregory, primary, Cnaan, Avital, primary, Zhao, Huaqing, primary, Cheung, Nai-Kong, primary, Gerald, William, primary, London, Wendy, primary, Matthay, Katherine K., primary, Brodeur, Garrett M., primary, and Maris, John M., primary

Published
2023
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31. Supplementary Figure S5 from Relationship of Gene Expression and Chromosomal Abnormalities in Colorectal Cancer

Author

Tsafrir, Dafna, primary, Bacolod, Manny, primary, Selvanayagam, Zachariah, primary, Tsafrir, Ilan, primary, Shia, Jinru, primary, Zeng, Zhaoshi, primary, Liu, Hao, primary, Krier, Curtis, primary, Stengel, Robert F., primary, Barany, Francis, primary, Gerald, William L., primary, Paty, Philip B., primary, Domany, Eytan, primary, and Notterman, Daniel A., primary

Published
2023
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32. Supplementary Materials & Legends from Relationship of Gene Expression and Chromosomal Abnormalities in Colorectal Cancer

Author

Tsafrir, Dafna, primary, Bacolod, Manny, primary, Selvanayagam, Zachariah, primary, Tsafrir, Ilan, primary, Shia, Jinru, primary, Zeng, Zhaoshi, primary, Liu, Hao, primary, Krier, Curtis, primary, Stengel, Robert F., primary, Barany, Francis, primary, Gerald, William L., primary, Paty, Philip B., primary, Domany, Eytan, primary, and Notterman, Daniel A., primary

Published
2023
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33. MIAME Microarry Data URL from Activator Protein-1 Transcription Factors Are Associated with Progression and Recurrence of Prostate Cancer

Author

Ouyang, Xuesong, primary, Jessen, Walter J., primary, Al-Ahmadie, Hikmat, primary, Serio, Angel M., primary, Lin, Yong, primary, Shih, Weichung-Joseph, primary, Reuter, Victor E., primary, Scardino, Peter T., primary, Shen, Michael M., primary, Aronow, Bruce J., primary, Vickers, Andrew J., primary, Gerald, William L., primary, and Abate-Shen, Cory, primary

Published
2023
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34. Supplementary Figures 1-5 and 7 from The SWI/SNF ATPase Brm Is a Gatekeeper of Proliferative Control in Prostate Cancer

Author

Shen, Hui, primary, Powers, Nathan, primary, Saini, Nitin, primary, Comstock, Clay E.S., primary, Sharma, Ankur, primary, Weaver, Katherine, primary, Revelo, Monica P., primary, Gerald, William, primary, Williams, Erin, primary, Jessen, Walter J., primary, Aronow, Bruce J., primary, Rosson, Gary, primary, Weissman, Bernard, primary, Muchardt, Christian, primary, Yaniv, Moshe, primary, and Knudsen, Karen E., primary

Published
2023
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35. Supplementary Figures 1-3, Tables 1-4 from Activator Protein-1 Transcription Factors Are Associated with Progression and Recurrence of Prostate Cancer

Author

Ouyang, Xuesong, primary, Jessen, Walter J., primary, Al-Ahmadie, Hikmat, primary, Serio, Angel M., primary, Lin, Yong, primary, Shih, Weichung-Joseph, primary, Reuter, Victor E., primary, Scardino, Peter T., primary, Shen, Michael M., primary, Aronow, Bruce J., primary, Vickers, Andrew J., primary, Gerald, William L., primary, and Abate-Shen, Cory, primary

Published
2023
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37. Data from The SWI/SNF ATPase Brm Is a Gatekeeper of Proliferative Control in Prostate Cancer

Author

Shen, Hui, primary, Powers, Nathan, primary, Saini, Nitin, primary, Comstock, Clay E.S., primary, Sharma, Ankur, primary, Weaver, Katherine, primary, Revelo, Monica P., primary, Gerald, William, primary, Williams, Erin, primary, Jessen, Walter J., primary, Aronow, Bruce J., primary, Rosson, Gary, primary, Weissman, Bernard, primary, Muchardt, Christian, primary, Yaniv, Moshe, primary, and Knudsen, Karen E., primary

Published
2023
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38. Supplementary Figure 6 from The SWI/SNF ATPase Brm Is a Gatekeeper of Proliferative Control in Prostate Cancer

Author

Shen, Hui, primary, Powers, Nathan, primary, Saini, Nitin, primary, Comstock, Clay E.S., primary, Sharma, Ankur, primary, Weaver, Katherine, primary, Revelo, Monica P., primary, Gerald, William, primary, Williams, Erin, primary, Jessen, Walter J., primary, Aronow, Bruce J., primary, Rosson, Gary, primary, Weissman, Bernard, primary, Muchardt, Christian, primary, Yaniv, Moshe, primary, and Knudsen, Karen E., primary

Published
2023
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40. Data from Control of Androgen Receptor Signaling in Prostate Cancer by the Cochaperone Small Glutamine–Rich Tetratricopeptide Repeat Containing Protein α

Author

Buchanan, Grant, primary, Ricciardelli, Carmela, primary, Harris, Jonathan M., primary, Prescott, Jennifer, primary, Yu, Zoe Chiao-Li, primary, Jia, Li, primary, Butler, Lisa M., primary, Marshall, Villis R., primary, Scher, Howard I., primary, Gerald, William L., primary, Coetzee, Gerhard A., primary, and Tilley, Wayne D., primary

Published
2023
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43. Data from The Signatures of Autozygosity among Patients with Colorectal Cancer

Author

Bacolod, Manny D., primary, Schemmann, Gunter S., primary, Wang, Shuang, primary, Shattock, Richard, primary, Giardina, Sarah F., primary, Zeng, Zhaoshi, primary, Shia, Jinru, primary, Stengel, Robert F., primary, Gerry, Norman, primary, Hoh, Josephine, primary, Kirchhoff, Tomas, primary, Gold, Bert, primary, Christman, Michael F., primary, Offit, Kenneth, primary, Gerald, William L., primary, Notterman, Daniel A., primary, Ott, Jurg, primary, Paty, Philip B., primary, and Barany, Francis, primary

Published
2023
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44. Supplementary Information from The Signatures of Autozygosity among Patients with Colorectal Cancer

Author

Bacolod, Manny D., primary, Schemmann, Gunter S., primary, Wang, Shuang, primary, Shattock, Richard, primary, Giardina, Sarah F., primary, Zeng, Zhaoshi, primary, Shia, Jinru, primary, Stengel, Robert F., primary, Gerry, Norman, primary, Hoh, Josephine, primary, Kirchhoff, Tomas, primary, Gold, Bert, primary, Christman, Michael F., primary, Offit, Kenneth, primary, Gerald, William L., primary, Notterman, Daniel A., primary, Ott, Jurg, primary, Paty, Philip B., primary, and Barany, Francis, primary

Published
2023
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45. Supplementary Tables 1-2 from TMPRSS2-ERG Gene Fusion Is Not Associated with Outcome in Patients Treated by Prostatectomy

Author

Gopalan, Anuradha, primary, Leversha, Margaret A., primary, Satagopan, Jaya M., primary, Zhou, Qin, primary, Al-Ahmadie, Hikmat A., primary, Fine, Samson W., primary, Eastham, James A., primary, Scardino, Peter T., primary, Scher, Howard I., primary, Tickoo, Satish K., primary, Reuter, Victor E., primary, and Gerald, William L., primary

Published
2023
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46. Supplementary Data 1-4 from Control of Androgen Receptor Signaling in Prostate Cancer by the Cochaperone Small Glutamine–Rich Tetratricopeptide Repeat Containing Protein α

Author

Buchanan, Grant, primary, Ricciardelli, Carmela, primary, Harris, Jonathan M., primary, Prescott, Jennifer, primary, Yu, Zoe Chiao-Li, primary, Jia, Li, primary, Butler, Lisa M., primary, Marshall, Villis R., primary, Scher, Howard I., primary, Gerald, William L., primary, Coetzee, Gerhard A., primary, and Tilley, Wayne D., primary

Published
2023
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47. Supplementary Figure and Table Legends from Genome-Wide Impact of Androgen Receptor Trapped clone-27 Loss on Androgen-Regulated Transcription in Prostate Cancer Cells

Author

Nwachukwu, Jerome C., primary, Mita, Paolo, primary, Ruoff, Rachel, primary, Ha, Susan, primary, Wang, Qianben, primary, Huang, S. Joseph, primary, Taneja, Samir S., primary, Brown, Myles, primary, Gerald, William L., primary, Garabedian, Michael J., primary, and Logan, Susan K., primary

Published
2023
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50. Supplementary Figure Legends 1-7 from The SWI/SNF ATPase Brm Is a Gatekeeper of Proliferative Control in Prostate Cancer

Author

Shen, Hui, primary, Powers, Nathan, primary, Saini, Nitin, primary, Comstock, Clay E.S., primary, Sharma, Ankur, primary, Weaver, Katherine, primary, Revelo, Monica P., primary, Gerald, William, primary, Williams, Erin, primary, Jessen, Walter J., primary, Aronow, Bruce J., primary, Rosson, Gary, primary, Weissman, Bernard, primary, Muchardt, Christian, primary, Yaniv, Moshe, primary, and Knudsen, Karen E., primary

Published
2023
Full Text
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