47 results on '"Ghasabyan, Arsen"'
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2. Hemorrhagic shock and tissue injury provoke distinct components of trauma-induced coagulopathy in a swine model
3. Beyond uterine atony: characterizing postpartum hemorrhage coagulopathy
4. Zone 1 REBOA in a combat DCBI swine model does not worsen brain injury
5. REBOA for the Treatment of Blast Polytrauma: Zone 3 Provides Cerebral Perfusion, Attenuates Organ Dysfunction and Reperfusion Coagulopathy Compared to Zone 1 in a Swine Model
6. Succinate Activation of SUCNR1 Predisposes Severely Injured Patients to Neutrophil-mediated ARDS
7. Postinjury complement C4 activation is associated with adverse outcomes and is potentially influenced by plasma resuscitation
8. Whole Blood Thrombin Generation in Severely Injured Patients Requiring Massive Transfusion
9. STudy of Alteplase for Respiratory failure in SARS-Cov2 COVID-19 (STARS): A Vanguard Multicenter, Rapidly Adaptive, Pragmatic, Randomized, Controlled Trial
10. Goal-directed Hemostatic Resuscitation of Trauma-induced Coagulopathy
11. The α-globin chain of hemoglobin potentiates tissue plasminogen activator induced hyperfibrinolysis in vitro
12. Trauma Resuscitation Consideration: Sex Matters
13. Succinate Activation of SUCNR1 Predisposes Severely Injured Patients to Neutrophil-Mediated ARDS
14. Rapid TEG efficiently guides hemostatic resuscitation in trauma patients
15. Empiric transfusion strategies during life-threatening hemorrhage
16. Tranexamic acid is associated with reduced complement activation in trauma patients with hemorrhagic shock and hyperfibrinolysis on thromboelastography
17. Platelet adenosine diphosphate receptor inhibition provides no advantage in predicting need for platelet transfusion or massive transfusion
18. Untangling Sex Dimorphisms in Coagulation: Initial Steps Toward Precision Medicine for Trauma Resuscitation
19. Obesity is associated with postinjury hypercoagulability
20. Variability in international normalized ratio and activated partial thromboplastin time after injury are not explained by coagulation factor deficits
21. Severe traumatic brain injury is associated with a unique coagulopathy phenotype
22. Hemorrhagic shock and tissue injury provoke distinct components of trauma-induced coagulopathy in a swine model
23. Do Venous and Arterial Base Excess Agree in Trauma Resuscitation? Experience of a Military-Relevant Severe Injury Swine Model
24. All animals are equal but some animals are more equal than others: Plasma lactate and succinate in hemorrhagic shock—A comparison in rodents, swine, nonhuman primates, and injured patients
25. Fibrinolysis shutdown is associated with a fivefold increase in mortality in trauma patients lacking hypersensitivity to tissue plasminogen activator
26. Study of Alteplase for Respiratory Failure in SARS-CoV-2 COVID-19
27. Traumatic brain injury provokes low fibrinolytic activity in severely injured patients
28. Role of Fibrinogen in Trauma-Induced Coagulopathy
29. The α-globin chain of hemoglobin potentiates tissue plasminogen activator induced hyperfibrinolysis in vitro
30. Exploring ethical conflicts in emergency trauma research: The COMBAT (Control of Major Bleeding after Trauma) study experience
31. Association of Prehospital Plasma Transfusion With Survival in Trauma Patients With Hemorrhagic Shock When Transport Times Are Longer Than 20 Minutes
32. Untangling Sex Dimorphisms in Coagulation
33. Elucidating the Molecular Mechanisms of Fibrinolytic Shutdown after Severe Injury: The Role of Thrombin Activatable Fibrinolysis Inhibitor
34. Less Is More: Adjusted Indices of Shock Do Not Improve Predictive Capability
35. Plasma-first resuscitation to treat haemorrhagic shock during emergency ground transportation in an urban area: a randomised trial
36. Goal-directed Hemostatic Resuscitation of Trauma-induced Coagulopathy: A Pragmatic Randomized Clinical Trial Comparing a Viscoelastic Assay to Conventional Coagulation Assays
37. Trauma Resuscitation Consideration: Sex Matters
38. Overwhelming tPA release, not PAI-1 degradation, is responsible for hyperfibrinolysis in severely injured trauma patients
39. The “Death Diamond”
40. Combat
41. Plasma succinate is a predictor of mortality in critically injured patients.
42. Postinjury Hyperfibrinogenemia Compromises Efficacy of Heparin-Based Venous Thromboembolism Prophylaxis
43. Fibrinolysis greater than 3% is the critical value for initiation of antifibrinolytic therapy
44. Platelets are dominant contributors to hypercoagulability after injury
45. FUNCTIONAL FIBRINOGEN ASSAY INDICATES THAT FIBRINOGEN IS CRITICAL IN CORRECTING ABNORMAL CLOT STRENGTH FOLLOWING TRAUMA
46. The "Death Diamond": Rapid thrombelastography identifies lethal hyperfibrinolysis.
47. COMBAT: INITIAL EXPERIENCE WITH A RANDOMIZED CLINICAL TRIAL OF PLASMA-BASED RESUSCITATION IN THE FIELD FOR TRAUMATIC HEMORRHAGIC SHOCK.
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