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2. Targeting the immune epigenome in Crohn’s disease

3. Mucosal macrophages express elevated levels of HDAC9 in inflamed and uninflamed mucosa of Crohn's disease, but not ulcerative colitis

7. Carboxylesterase 1 directs the metabolic profile of dendritic cells to a reduced inflammatory phenotype.

8. Exploring the Immunomodulatory Potential of Human Milk: Aryl Hydrocarbon Receptor Activation and Its Impact on Neonatal Gut Health.

9. The Epigenetic Reader Protein SP140 Regulates Dendritic Cell Activation, Maturation and Tolerogenic Potential.

10. Sustained Diet-Induced Remission in Pediatric Crohn's Disease Is Associated With Kynurenine and Serotonin Pathways.

11. Successful Dietary Therapy in Paediatric Crohn's Disease is Associated with Shifts in Bacterial Dysbiosis and Inflammatory Metabotype Towards Healthy Controls.

12. Metabolome Changes With Diet-Induced Remission in Pediatric Crohn's Disease.

13. Modulation of macrophage inflammatory function through selective inhibition of the epigenetic reader protein SP140.

14. Carboxylesterase-1 Assisted Targeting of HDAC Inhibitors to Mononuclear Myeloid Cells in Inflammatory Bowel Disease.

15. The Progress of Intestinal Epithelial Models from Cell Lines to Gut-On-Chip.

16. Selective Targeting of Epigenetic Readers and Histone Deacetylases in Autoimmune and Inflammatory Diseases: Recent Advances and Future Perspectives.

17. Tyrosine Kinase 2 Signalling Drives Pathogenic T cells in Colitis.

18. Nutritional Therapy Strategies in Pediatric Crohn's Disease.

19. HDAC3 Mediates the Inflammatory Response and LPS Tolerance in Human Monocytes and Macrophages.

20. Nutritional Therapy to Modulate Tryptophan Metabolism and Aryl Hydrocarbon-Receptor Signaling Activation in Human Diseases.

21. Whole-Genome DNA Methylation Profiling of CD14+ Monocytes Reveals Disease Status and Activity Differences in Crohn's Disease Patients.

22. Reduced IL-37 Production Increases Spontaneous Chemokine Expressions in Colon Epithelial Cells.

24. Phospholipid flippases attenuate LPS-induced TLR4 signaling by mediating endocytic retrieval of Toll-like receptor 4.

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