316 results on '"Gilon, Patrick"'
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2. GLP-1R agonists demonstrate potential to treat Wolfram syndrome in human preclinical models
3. GLP-1 and GIP receptors signal through distinct β-arrestin 2-dependent pathways to regulate pancreatic β cell function
4. ER stress increases expression of intracellular calcium channel RyR1 to modify Ca2+ homeostasis in pancreatic beta cells
5. Glucose inhibits glucagon secretion by decreasing [Ca2+]c and by reducing the efficacy of Ca2+ on exocytosis via somatostatin-dependent and independent mechanisms
6. KATP channel blockers control glucagon secretion by distinct mechanisms: A direct stimulation of α-cells involving a [Ca2+]c rise and an indirect inhibition mediated by somatostatin
7. 3D evaluation of the extracellular matrix of hypoxic pancreatic islets using light sheet fluorescence microscopy
8. SGLT2 is not expressed in pancreatic α- and β-cells, and its inhibition does not directly affect glucagon and insulin secretion in rodents and humans
9. Metallothionein 1 negatively regulates glucose-stimulated insulin secretion and is differentially expressed in conditions of beta cell compensation and failure in mice and humans
10. Corrigendum to “Inter-domain tagging implicates caveolin-1 in insulin receptor trafficking and Erk signaling bias in pancreatic beta-cells” [Mol Metab 2016 May; 5 (5): 366–378]
11. Chemerin as an Inducer of β Cell Proliferation Mediates Mitochondrial Homeostasis and Promotes β Cell Mass Expansion
12. Identification of islet-enriched long non-coding RNAs contributing to β-cell failure in type 2 diabetes
13. Physiological and Pathophysiological Control of Glucagon Glucagon Secretion by Pancreatic α-Cells
14. Prolonged culture of human pancreatic islets under glucotoxic conditions changes their acute beta cell calcium and insulin secretion glucose response curves from sigmoid to bell-shaped
15. Inter-domain tagging implicates caveolin-1 in insulin receptor trafficking and Erk signaling bias in pancreatic beta-cells
16. In depth functional characterization of human induced pluripotent stem cell-derived beta cells in vitro and in vivo
17. Cocaine- and amphetamine-regulated transcript: a novel regulator of energy homeostasis expressed in a subpopulation of pancreatic islet cells
18. Physiological ER Stress: The Model of Insulin-Secreting Pancreatic b-Cells
19. Identification of a human-specific alteration of beta cell function after prolonged culture of pancreatic islets under glucotoxic conditions
20. The endoplasmic reticulum-plasma membrane tethering protein TMEM24 is a regulator of cellular Ca2+ homeostasis
21. The endoplasmic reticulum-plasma membrane tethering protein TMEM24 is a regulator of cellular Ca2+ homeostasis
22. Loss of high-frequency glucose-induced Ca²⁺ oscillations in pancreatic islets correlates with impaired glucose tolerance in Trpm5 -/- mice
23. LDHA is enriched in human islet alpha cells and upregulated in type 2 diabetes
24. Frequency-dependent mitochondrial Ca2+ accumulation regulates ATP synthesis in pancreatic β cells
25. Physiological and Pathophysiological Control of Glucagon Secretion by Pancreatic α-Cells
26. The endoplasmic reticulum-plasma membrane tethering protein TMEM24 is a regulator of cellular Ca2+ homeostasis.
27. Massive lactic- and keto- acidosis with glucagon deficiency in a chronic alcoholic patient
28. Beta-arrestin 2 is absolutely required for the potentiation of insulin secretion by GIP
29. γ-Hydroxybutyrate does not mediate glucose inhibition of glucagon secretion
30. Glucose-induced mixed [[[Ca.sup.2+]].sub.c] oscillations in mouse [beta]-cells are controlled by the membrane potential and the SERCA3 [Ca.sup.2+]-ATPase of the endoplasmic reticulum
31. Inhibition of aquaporin-1 prevents myocardial remodeling by blocking the transmembrane transport of hydrogen peroxide
32. UCP2 Regulates the Glucagon Response to Fasting and Starvation
33. Tolbutamide Controls Glucagon Release From Mouse Islets Differently Than Glucose: Involvement of KATP Channels From Both α-Cells and δ-Cells
34. In Situ Electrophysiological Examination of Pancreatic α Cells in the Streptozotocin-Induced Diabetes Model, Revealing the Cellular Basis of Glucagon Hypersecretion
35. In situ electrophysiological examination of pancreatic cells in the streptozotocin-induced diabetes model, revealing the cellular basis of glucagon hypersecretion
36. Contribution of the endoplasmic reticulum to the glucose-induced [[[Ca.sup.2+]].sub.c] response in mouse pancreatic islets
37. Mechanisms of Control of the Free Ca2+ Concentration in the Endoplasmic Reticulum of Mouse Pancreatic β-Cells: Interplay With Cell Metabolism and [Ca2+]c and Role of SERCA2b and SERCA3
38. γ-Hydroxybutyrate does not mediate glucose inhibition of glucagon secretion
39. The Role of α-Cells in Islet Function and Glucose Homeostasis in Health and Type 2 Diabetes
40. Glucose and Pharmacological Modulators of ATP-Sensitive K+ Channels Control [Ca2+]c by Different Mechanisms in Isolated Mouse α-Cells
41. SGLT2 is not expressed in pancreatic alpha and beta cells and its inhibition does not directly affect glucagon and insulin secretion in rodents and humans
42. In vitro and fully mature in vivo function of human induced pluripotent stem cell-derived beta cells
43. No evidence for a role of reverse Na+-Ca2+ exchange in insulin release from mouse pancreatic islets
44. Atypical Ca2+-induced Ca2+ release from a sarco-endoplasmic reticulum Ca2+-ATPase 3-dependent Ca2+ pool in mouse pancreatic β-cells
45. Signal Transduction
46. SERCA3 Ablation Does Not Impair Insulin Secretion but Suggests Distinct Roles of Different Sarcoendoplasmic Reticulum Ca2+ Pumps for Ca2+ Homeostasis in Pancreatic β-cells
47. Control Mechanisms of the Oscillations of Insulin Secretion In Vitro and In Vivo
48. Feedback Control of the ATP-Sensitive K+ Current by Cytosolic Ca2+ Contributes to Oscillations of the Membrane Potential in Pancreatic β-Cells
49. Mechanisms and Physiological Significance of the Cholinergic Control of Pancreatic β-Cell Function
50. Inhibition of Protein Synthesis Sequentially Impairs Distinct Steps of Stimulus-secretion Coupling in Pancreatic β Cells*
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