289 results on '"Gladson, Candece L."'
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2. Integrin α3β1 promotes vessel formation of glioblastoma-associated endothelial cells through calcium-mediated macropinocytosis and lysosomal exocytosis
3. Urokinase-type Plasminogen Activator Receptor (uPAR) Ligation Induces a Raft-localized Integrin Signaling Switch That Mediates the Hypermotile Phenotype of Fibrotic Fibroblasts*
4. Expression of LC3B and FIP200/Atg17 in brain metastases of breast cancer
5. Supplemental Methods from Macropinocytosis of Bevacizumab by Glioblastoma Cells in the Perivascular Niche Affects their Survival
6. SFigure 5 from Macropinocytosis of Bevacizumab by Glioblastoma Cells in the Perivascular Niche Affects their Survival
7. SFigure 6 from Macropinocytosis of Bevacizumab by Glioblastoma Cells in the Perivascular Niche Affects their Survival
8. SFigure 1 from Macropinocytosis of Bevacizumab by Glioblastoma Cells in the Perivascular Niche Affects their Survival
9. SFigure 7 from Macropinocytosis of Bevacizumab by Glioblastoma Cells in the Perivascular Niche Affects their Survival
10. SFigure 4 from Macropinocytosis of Bevacizumab by Glioblastoma Cells in the Perivascular Niche Affects their Survival
11. SFigure 3 from Macropinocytosis of Bevacizumab by Glioblastoma Cells in the Perivascular Niche Affects their Survival
12. SFigure 2 from Macropinocytosis of Bevacizumab by Glioblastoma Cells in the Perivascular Niche Affects their Survival
13. Supplementary Figure 4 from Endothelial Expression of TNF Receptor-1 Generates a Proapoptotic Signal Inhibited by Integrin α6β1 in Glioblastoma
14. Supplementary Figure Legends 1-7 from Endothelial Expression of TNF Receptor-1 Generates a Proapoptotic Signal Inhibited by Integrin α6β1 in Glioblastoma
15. Supplementary Figure 1 from Endothelial Expression of TNF Receptor-1 Generates a Proapoptotic Signal Inhibited by Integrin α6β1 in Glioblastoma
16. Supplementary Figure 5 from Endothelial Expression of TNF Receptor-1 Generates a Proapoptotic Signal Inhibited by Integrin α6β1 in Glioblastoma
17. Data from Endothelial Expression of TNF Receptor-1 Generates a Proapoptotic Signal Inhibited by Integrin α6β1 in Glioblastoma
18. Supplementary Figure Legends 1-3 from Plasminogen Kringle 5 Induces Apoptosis of Brain Microvessel Endothelial Cells: Sensitization by Radiation and Requirement for GRP78 and LRP1
19. Supplementary Figure 2 from Endothelial Expression of TNF Receptor-1 Generates a Proapoptotic Signal Inhibited by Integrin α6β1 in Glioblastoma
20. Supplementary Table 2 from Endothelial Expression of TNF Receptor-1 Generates a Proapoptotic Signal Inhibited by Integrin α6β1 in Glioblastoma
21. Supplementary Figure 3 from Endothelial Expression of TNF Receptor-1 Generates a Proapoptotic Signal Inhibited by Integrin α6β1 in Glioblastoma
22. Data from Plasminogen Kringle 5 Induces Apoptosis of Brain Microvessel Endothelial Cells: Sensitization by Radiation and Requirement for GRP78 and LRP1
23. Supplementary Figure 3 from Plasminogen Kringle 5 Induces Apoptosis of Brain Microvessel Endothelial Cells: Sensitization by Radiation and Requirement for GRP78 and LRP1
24. Supplementary Figure 6 from Endothelial Expression of TNF Receptor-1 Generates a Proapoptotic Signal Inhibited by Integrin α6β1 in Glioblastoma
25. Supplementary Figure 1 from Plasminogen Kringle 5 Induces Apoptosis of Brain Microvessel Endothelial Cells: Sensitization by Radiation and Requirement for GRP78 and LRP1
26. Supplementary Figure 2 from Plasminogen Kringle 5 Induces Apoptosis of Brain Microvessel Endothelial Cells: Sensitization by Radiation and Requirement for GRP78 and LRP1
27. Supplementary Table 1 from Endothelial Expression of TNF Receptor-1 Generates a Proapoptotic Signal Inhibited by Integrin α6β1 in Glioblastoma
28. Supplementary Figure 7 from Endothelial Expression of TNF Receptor-1 Generates a Proapoptotic Signal Inhibited by Integrin α6β1 in Glioblastoma
29. The αv Integrins
30. Correlation of higher levels of soluble TNF-R1 with a shorter survival, independent of age, in recurrent glioblastoma
31. Glioblastoma Cell Motility: The Role of FAK and Cellular SRC
32. FAK-Related Nonkinase Is a Multifunctional Negative Regulator of Pulmonary Fibrosis
33. Urokinase receptor mediates lung fibroblast attachment and migration toward provisional matrix proteins through interaction with multiple integrins
34. Overexpression of FAK promotes Ras activity through the formation of a FAK/p120RasGAP complex in malignant astrocytoma cells
35. Oncostatin-M induction of vascular endothelial growth factor expression in astroglioma cells
36. TGF-β1 up-regulates paxillin protein expression in malignant astrocytoma cells: requirement for a fibronectin substrate
37. Laminin alpha 2 enables glioblastoma stem cell growth
38. Transcriptional regulation of the human osteopontin promoter: functional analysis and DNA-protein interactions
39. Downregulation of FAK-related non-kinase mediates the migratory phenotype of human fibrotic lung fibroblasts
40. A Phase 1 Trial of ABT-510 Concurrent With Standard Chemoradiation for Patients With Newly Diagnosed Glioblastoma
41. The Role of the Extracellular Matrix in Angiogenesis in Malignant Glioma Tumors
42. Adenovirally Mediated Expression of Urokinase Receptor Binding Site on Integrin α-Chain Blocks Adhesion and Migration of Human Lung Fibroblasts*
43. The Extracellular Matrix of Gliomas: Modulation of Cell Function
44. Increased Injury Following Intermittent Fetal Hypoxia-Reoxygenation Is Associated with Increased Free Radical Production in Fetal Rabbit Brain
45. Sustained Hypoxia-lschemia Results in Reactive Nitrogen and Oxygen Species Production and Injury in the Premature Fetal Rabbit Brain
46. Expression of Integrin avß3 in Small Blood Vessels of Glioblastoma Tumors
47. Changes in Procoagulant and Fibrinolytic Gene Expression during Bleomycin-induced Lung Injury in the Mouse
48. Macropinocytosis of Bevacizumab by Glioblastoma Cells in the Perivascular Niche Affects their Survival
49. Selenoprotein P neutralizes lipopolysaccharide and participates in hepatic cell endoplasmic reticulum stress response
50. Correlation of higher levels of soluble TNF-R1 with a shorter survival, independent of age, in recurrent glioblastoma
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