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1. Immunomolecular and reactivity landscapes of gut IgA subclasses in homeostasis and inflammatory bowel disease.

2. Cutting Edge: Neutrophil Complement Receptor Signaling Is Required for BAFF-Dependent Humoral Responses in Mice.

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3. Good intentions gone wrong: The B cell block to epithelial repair.

4. Ulcerative colitis is characterized by a plasmablast-skewed humoral response associated with disease activity.

5. SARS-CoV-2 sculpts the immune system to induce sustained virus-specific naïve-like and memory B-cell responses.

6. Mutations make gut antibodies promiscuous.

7. Gut T cell-independent IgA responses to commensal bacteria require engagement of the TACI receptor on B cells.

8. Rethinking mucosal antibody responses: IgM, IgG and IgD join IgA.

9. Fecal IgA Levels Are Determined by Strain-Level Differences in Bacteroides ovatus and Are Modifiable by Gut Microbiota Manipulation.

10. BAFF-driven B cell hyperplasia underlies lung disease in common variable immunodeficiency.

11. Secreted IgD Amplifies Humoral T Helper 2 Cell Responses by Binding Basophils via Galectin-9 and CD44.

12. mTOR intersects antibody-inducing signals from TACI in marginal zone B cells.

13. Human Secretory IgM Emerges from Plasma Cells Clonally Related to Gut Memory B Cells and Targets Highly Diverse Commensals.

14. Deletion of WASp and N-WASp in B cells cripples the germinal center response and results in production of IgM autoantibodies.

15. Sterile inflammation in the spleen during atherosclerosis provides oxidation-specific epitopes that induce a protective B-cell response.

16. Immunology. Retroviral help for B cells.

17. BLyS-mediated modulation of naive B cell subsets impacts HIV Env-induced antibody responses.

18. The inflammatory cytokine IL-18 induces self-reactive innate antibody responses regulated by natural killer T cells.