46 results on '"Guerreiro-Cacais, Andre"'
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2. Disease-associated oligodendroglia: a putative nexus in neurodegeneration
3. Cellular architecture of evolving neuroinflammatory lesions and multiple sclerosis pathology
4. IL-4 controls activated neutrophil FcγR2b expression and migration into inflamed joints
5. C-type lectin receptors Mcl and Mincle control development of multiple sclerosis-like neuroinflammation
6. A pan-cancer signature for dysfunctional T cells
7. Functional genomics analysis of vitamin D effects on CD4+ T cells in vivo in experimental autoimmune encephalomyelitis
8. Cross-reactive EBNA1 immunity targets alpha-crystallin B and is associated with multiple sclerosis
9. Anoctamin 2 identified as an autoimmune target in multiple sclerosis
10. Strain influences on inflammatory pathway activation, cell infiltration and complement cascade after traumatic brain injury in the rat
11. Identification of four novel T cell autoantigens and personal autoreactive profiles in multiple sclerosis
12. A pan-cancer signature for dysfunctional T cells
13. Microglial autophagy-associated phagocytosis is essential for recovery from neuroinflammation
14. Syntaxin 11 marks a distinct intracellular compartment recruited to the immunological synapse of NK cells to colocalize with cytotoxic granules
15. Microglial autophagy–associated phagocytosis is essential for recovery from neuroinflammation
16. C-type lectin receptors Mcl and Mincle control development of multiple sclerosis–like neuroinflammation
17. IL-22 Binding Protein Promotes the Disease Process in Multiple Sclerosis
18. IL-22 binding protein regulates murine skin inflammation
19. Additional file 6: Figure S4. of MOG-induced experimental autoimmune encephalomyelitis in the rat species triggers anti-neurofascin antibody response that is genetically regulated
20. Additional file 4: Figure S2. of MOG-induced experimental autoimmune encephalomyelitis in the rat species triggers anti-neurofascin antibody response that is genetically regulated
21. Genomes and phenomes of a population of outbred rats and its progenitors
22. Translational utility of experimental autoimmune encephalomyelitis: recent developments
23. Rat bone marrow-derived dendritic cells generated with GM-CSF/IL-4 or FLT3L exhibit distinct phenotypical and functional characteristics
24. MOG-induced experimental autoimmune encephalomyelitis in the rat species triggers anti-neurofascin antibody response that is genetically regulated
25. Variability in C-type lectin receptors regulates neuropathic pain-like behavior after peripheral nerve injury
26. VAV1 controls inflammation in Experimental Autoimmune Encephalomyelitis (EAE)
27. Variability in C-Type Lectin Receptors Regulates Neuropathic Pain-Like Behavior after Peripheral Nerve Injury
28. Immunological consequences of Epstein-Barr virus replication
29. Translational utility of experimental autoimmune encephalomyelitis: recent developments.
30. Syntaxin 11 marks a distinct intracellular compartment recruited to the immunological synapse of NK cells to colocalize with cytotoxic granules
31. B cell receptor triggering sensitizes human B cells to TRAIL-induced apoptosis
32. Inhibition of Heavy Chain and β 2 -Microglobulin Synthesis as a Mechanism of Major Histocompatibility Complex Class I Downregulation during Epstein-Barr Virus Replication
33. Capacity of Epstein–Barr virus to infect monocytes and inhibit their development into dendritic cells is affected by the cell type supporting virus replication
34. Rat bone marrow‐derived dendritic cells generated with GM‐CSF/IL‐4 or FLT3L exhibit distinct phenotypical and functional characteristics
35. Combined sequence-based and genetic mapping analysis of complex traits in outbred rats.
36. Additional file 1: Table S1. of MOG-induced experimental autoimmune encephalomyelitis in the rat species triggers anti-neurofascin antibody response that is genetically regulated
37. Additional file 5: Figure S3. of MOG-induced experimental autoimmune encephalomyelitis in the rat species triggers anti-neurofascin antibody response that is genetically regulated
38. Additional file 5: Figure S3. of MOG-induced experimental autoimmune encephalomyelitis in the rat species triggers anti-neurofascin antibody response that is genetically regulated
39. Additional file 3: Table S2. of MOG-induced experimental autoimmune encephalomyelitis in the rat species triggers anti-neurofascin antibody response that is genetically regulated
40. Additional file 1: Table S1. of MOG-induced experimental autoimmune encephalomyelitis in the rat species triggers anti-neurofascin antibody response that is genetically regulated
41. Additional file 3: Table S2. of MOG-induced experimental autoimmune encephalomyelitis in the rat species triggers anti-neurofascin antibody response that is genetically regulated
42. Additional file 2: Figure S1. of MOG-induced experimental autoimmune encephalomyelitis in the rat species triggers anti-neurofascin antibody response that is genetically regulated
43. Additional file 2: Figure S1. of MOG-induced experimental autoimmune encephalomyelitis in the rat species triggers anti-neurofascin antibody response that is genetically regulated
44. Inhibition of Heavy Chain and β2-Microglobulin Synthesis as a Mechanism of Major Histocompatibility Complex Class I Downregulation during Epstein-Barr Virus Replication.
45. Rat bone marrow-derived dendritic cells generated with GM-CSF/IL-4 or FLT3L exhibit distinct phenotypical and functional characteristics.
46. Inhibition of heavy chain and beta2-microglobulin synthesis as a mechanism of major histocompatibility complex class I downregulation during Epstein-Barr virus replication.
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