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1. Rad5 and Its Human Homologs, HLTF and SHPRH, Are Novel Interactors of Mismatch Repair

2. Effects of diet and hyperlipidemia on levels and distribution of circulating lysophosphatidic acid

3. Rad5 and Its Human Homologs, HLTF and SHPRH, Are Novel Interactors of Mismatch Repair

4. Effects of diet and hyperlipidemia on levels and distribution of circulating lysophosphatidic acid

5. Regulation of PLPP3 gene expression by NF-κB family transcription factors

6. Author Correction: Identification of novel genetic variants predisposing to familial oral squamous cell carcinomas

7. Abstract 799: HLTF and SHPRH in mismatch repair and cancer

8. Cancer-driving H3G34V/R/D mutations block H3K36 methylation and H3K36me3–MutSα interaction

9. Coronary Artery Disease Risk-Associated

10. Identification of novel genetic variants predisposing to familial oral squamous cell carcinomas

11. J. Lipid Res.-2019-Kraemer-jlr.M093096.pdf

12. Modulation of microRNA processing by mismatch repair protein MutLα

13. Evidence That a Mutation in the MLH1 3′-Untranslated Region Confers a Mutator Phenotype and Mismatch Repair Deficiency in Patients with Relapsed Leukemia

14. Identification and characterization of OGG1 mutations in patients with Alzheimer's disease

15. Coordinated Processing of 3′ Slipped (CAG)n/(CTG)n Hairpins by DNA Polymerases β and δ Preferentially Induces Repeat Expansions*

16. Cancer-driving H3G34V/R/D mutations block H3K36 methylation and H3K36me3-MutSα interaction.

17. The histone mark H3K36me3 regulates human DNA mismatch repair through its interaction with MutSα

18. Specific sequence determinants of miR-15/107 microRNA gene group targets

19. The miR-15/107 group of microRNA genes: evolutionary biology, cellular functions, and roles in human diseases

20. Altered 8-oxoguanine glycosylase in mild cognitive impairment and late-stage Alzheimer’s disease brain

22. Preferential loss of mismatch repair function in refractory and relapsed acute myeloid leukemia: potential contribution to AML progression

23. [Changes of phosphorylation states of Cx43 protein in CCL-64 cells induced by SiO2]

24. [Changes of phosphorylation states of Cx43 protein in Chinese hamster lung cells induced by SiO2]

25. [Downregulation of gap junctional intercellular communication induced by silicon dioxide in the pulmonary alveolar epithelial cell]

26. [Abnormal shift of connexin 43 gap-junction protein induced by 50 Hz electromagnetic fields in Chinese hamster lung cells]

27. Preferential loss of mismatch repair function in refractory and relapsed acute myeloid leukemia: potential contribution to AML progression.

28. Coordinated Processing of 3′ Slipped (CAG)n/(CTG)n Hairpins by DNA Polymerases β and δ Preferentially Induces Repeat Expansions.

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