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4,895 results on '"HU, Guang"'

4. The rise of baobab trees in Madagascar

Author

Wan, Jun-Nan, Wang, Sheng-Wei, Leitch, Andrew R., Leitch, Ilia J., Jian, Jian-Bo, Wu, Zhang-Yan, Xin, Hai-Ping, Rakotoarinivo, Mijoro, Onjalalaina, Guy Eric, Gituru, Robert Wahiti, Dai, Can, Mwachala, Geoffrey, Bai, Ming-Zhou, Zhao, Chen-Xi, Wang, Hong-Qi, Du, Sheng-Lan, Wei, Neng, Hu, Guang-Wan, Chen, Si-Chong, Chen, Xiao-Ya, Wan, Tao, and Wang, Qing-Feng

Published
2024
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12. The cotton MYB33 gene is a hub gene regulating the trade-off between plant growth and defense in Verticillium dahliae infection

Author

Hu Guang, Ge Xiaoyang, Wang Zhian, Wang Ye, Wang Peng, Shi Linfang, Wang Bingting, Zhang Anhong, Li Fuguang, and Wu Jiahe

Subjects
GhMYB33, ghr-miR319c, Trade-off, Gossypium hirsutum, Verticillium dahliae, Medicine (General), R5-920, Science (General), Q1-390
Abstract

Introduction: Sessile plants engage in trade-offs between growth and defense capacity in response to fluctuating environmental cues. MYB is an important transcription factor that plays many important roles in controlling plant growth and defense. However, the mechanism behind how it keeps a balance between these two physiological processes is still largely unknown. Objectives: Our work focuses on the dissection of the molecular mechanism by which GhMYB33 regulates plant growth and defense. Methods: The CRISPR/Cas9 technique was used to generate mutants for deciphering GhMYB33 functions. Yeast two-hybrid, luciferase complementary imaging, and co-immunoprecipitation assays were used to prove that proteins interact with each other. We used the electrophoretic mobility shift assay, yeast one-hybrid, and luciferase activity assays to analyze GhMYB33 acting as a promoter. A β-glucuronidase fusion reporter and 5′ RNA ligase mediated amplification of cDNA ends analysis showed that ghr-miR319c directedly cleaved the GhMYB33 mRNA. Results: Overexpressing miR319c-resistant GhMYB33 (rGhMYB33) promoted plant growth, accompanied by a significant decline in resistance against Verticillium dahliae. Conversely, its knockout mutant, ghmyb33, demonstrated growth restriction and concomitant augmentation of V. dahliae resistance. GhMYB33 was found to couple with the DELLA protein GhGAI1 and bind to the specific cis-elements of GhSPL9 and GhDFR1 promoters, thereby modulating internode elongation and plant resistance in V. dahliae infection. The ghr-miR319c was discovered to target and suppress GhMYB33 expression. The overexpression of ghr-miR319c led to enhanced plant resistance and a simultaneous reduction in plant height. Conclusion: Our findings demonstrate that GhMYB33 encodes a hub protein and controls the expression of GhSPL9 and GhDFR1, implicating a pivotal role for the miR319c-MYB33 module to regulate the trade-offs between plant growth and defense.

Published
2024
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14. Design and Research of the Control and Management System of Photovoltaic Cell

Author

Zhou, Youjie, Chen, Jinmao, Xiong, Chunhua, Wang, Xudong, Wen, Liang, Ren, Lianling, Huang, Yongcheng, Wang, Yaohui, Hu, Guang, Angrisani, Leopoldo, Series Editor, Arteaga, Marco, Series Editor, Chakraborty, Samarjit, Series Editor, Chen, Jiming, Series Editor, Chen, Shanben, Series Editor, Chen, Tan Kay, Series Editor, Dillmann, Rüdiger, Series Editor, Duan, Haibin, Series Editor, Ferrari, Gianluigi, Series Editor, Ferre, Manuel, Series Editor, Jabbari, Faryar, Series Editor, Jia, Limin, Series Editor, Kacprzyk, Janusz, Series Editor, Khamis, Alaa, Series Editor, Kroeger, Torsten, Series Editor, Li, Yong, Series Editor, Liang, Qilian, Series Editor, Martín, Ferran, Series Editor, Ming, Tan Cher, Series Editor, Minker, Wolfgang, Series Editor, Misra, Pradeep, Series Editor, Mukhopadhyay, Subhas, Series Editor, Ning, Cun-Zheng, Series Editor, Nishida, Toyoaki, Series Editor, Oneto, Luca, Series Editor, Panigrahi, Bijaya Ketan, Series Editor, Pascucci, Federica, Series Editor, Qin, Yong, Series Editor, Seng, Gan Woon, Series Editor, Speidel, Joachim, Series Editor, Veiga, Germano, Series Editor, Wu, Haitao, Series Editor, Zamboni, Walter, Series Editor, Zhang, Junjie James, Series Editor, Tan, Kay Chen, Series Editor, Wang, Wei, editor, Liu, Xin, editor, Na, Zhenyu, editor, and Zhang, Baoju, editor

Published
2024
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22. Laboratory observation of ion acceleration via reflection off laser-produced magnetized collisionless shocks

Author

Tang, Hui-bo, Yu-fei, Hao, Hu, Guang-yue, Lu, Quan-ming, Ren, Chuang, Zhang, Yu, Guo, Ao, Hu, Peng, Wang, Yu-lin, Wang, Xiang-bing, Zhang, Zhen-chi, Yuan, Peng, Liu, Wei, Si, Hua-chong, Yu, Chun-kai, Zhao, Jia-yi, Wang, Jin-can, Zhang, Zhe, Yuan, Xiao-hui, Yuan, Da-wei, Xie, Zhi-yong, Xiong, Jun, Fang, Zhi-heng, Xu, Jian-cai, Ju, Jing-Jing, Guo-qiang, Zhang, Zhu, Jian-Qiang, Shen, Bai-fei, Li, Ru-xin, and Xu, Zhi-zhan

Subjects
Physics - Plasma Physics, Physics - Space Physics
Abstract

Fermi acceleration by collisionless shocks is believed to be the primary mechanism to produce high energy charged particles in the Universe,where charged particles gain energy successively from multiple reflections off the shock front.Here,we present the first direct experimental evidence of ion energization from reflection off a supercritical quasi perpendicular collisionless shock,an essential component of Fermi acceleration in a laser produced magnetized plasma. We observed a quasi monoenergetic ion beam with 2,4 times the shock velocity in the upstream flow using time of flight method. Our related kinetic simulations reproduced the energy gain and showed that these ions were first reflected and then accelerated mainly by the motional electric field associated with the shock. This mechanism can also explain the quasi monoenergetic fast ion component observed in the Earth's bow shock.

Published
2022

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