19 results on '"Hagen, Evelin"'
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2. Fetal origin of allergic asthma: insights on mechanistic cues and therapeutic targets arising from a mouse model of prenatal stress challenge
3. Is there a ‘gut–brain–skin axis’?
4. Topical minoxidil counteracts stress-induced hair growth inhibition in mice
5. Patterns of Proliferation and Apoptosis during Murine Hair Follicle Morphogenesis
6. A new role for neurotrophins: involvement of brain-derived neurotrophic factor and neurotrophin-4 in hair cycle control
7. Do Hair Bulb Melanocytes Undergo Apoptosis During Hair Follicle Regression (Catagen)?
8. Generation and Cyclic Remodeling of the Hair Follicle Immune System in Mice
9. Stress-Induced Neurogenic Inflammation in Murine Skin Skews Dendritic Cells Towards Maturation and Migration : Key Role of Intercellular Adhesion Molecule-1/Leukocyte Function-Associated Antigen Interactions
10. Stress-Induced Neurogenic Inflammation in Murine Skin Skews Dendritic Cells Towards Maturation and Migration
11. Further Exploring the Brain–Skin Connection: Stress Worsens Dermatitis via Substance P-dependent Neurogenic Inflammation in Mice
12. Probing the Effects of Stress Mediators on the Human Hair Follicle
13. Intercellular Adhesion Molecule-1/LFA-1 Cross Talk Is a Proximate Mediator Capable of Disrupting Immune Integration and Tolerance Mechanism at the Feto-Maternal Interface in Murine Pregnancies
14. Neurogenic Inflammation in Stress-Induced Termination of Murine Hair Growth Is Promoted by Nerve Growth Factor
15. Stress Inhibits Hair Growth in Mice by Induction of Premature Catagen Development and Deleterious Perifollicular Inflammatory Events via Neuropeptide Substance P-Dependent Pathways
16. Increased Expression of Chemokine Receptor 5 (CCR5) in Blood and Decidual Cells in A Mouse Model for Preeclampsia (PE)
17. Upregulation of Decidual P-Selectin Expression Is Associated with an Increased Number of Th1 Cell Populations in Patients Suffering from Spontaneous Abortions
18. Indications for a brain‐hair follicle axis: inhibition of keratinocyte proliferation and up‐regulation of keratinocyte apoptosis in telogen hair follicles by stress and substance P
19. Hair bulb melanocytes undergo apotosis during normal and pathologic hair follicle regression (catagen)
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