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4. β-Arrestin pathway activation by selective ATR1 agonism promotes calcium influx in podocytes, leading to glomerular damage

6. List of Contributors

9. Rare variants in tenascin genes in a cohort of children with primary vesicoureteric reflux

12. Steroid-sensitive nephrotic syndrome candidate gene CLVS1 regulates podocyte oxidative stress and endocytosis

15. IL-1 receptor signaling in podocytes limits susceptibility to glomerular damage.

16. Inhibitor-[kappa]B kinase-[beta] regulates LPS-induced TNF-[alpha] production in cardiac myocytes through modulation of NF-[kappa]B p65 subunit phosphorylation

18. Endotoxin stress-response in cardiomyocytes: NF-[kappa]B activation and tumor necrosis factor-[alpha] expression

19. TRPC6 Enhances Angiotensin II-induced Albuminuria

23. Pregnancy and estradiol modulate myometrial G-protein pathways in the guinea pig

24. SP001A Novel Heterozygous Missense Mutation of Wilms’ Tumor 1 May Cause FSGS Through Dysregulated Expression of ARHGAP24

25. FO068The LMX1βR246Q Mutation Induces Podocyte Injury Through Dysregulation of Cholesterol Transport Gene Expression

26. Genetic Testing for Steroid-Resistant-Nephrotic Syndrome in an Outbred Population

27. TRPC Channels in Proteinuric Kidney Diseases.

29. The Human FSGS-Causing ANLN R431C Mutation Induces Dysregulated PI3K/AKT/mTOR/Rac1 Signaling in Podocytes

33. Rare variants in tenascin genes in a cohort of children with primary vesicoureteric reflux

36. HLA-DQA1 and PLCG2 Are Candidate Risk Loci for Childhood-Onset Steroid-Sensitive Nephrotic Syndrome

37. A Novel Missense Mutation of Wilms’ Tumor 1 Causes Autosomal Dominant FSGS

38. Rare hereditary COL4A3/COL4A4 variants may be mistaken for familial focal segmental glomerulosclerosis

40. Mutations in the Gene That Encodes the F-Actin Binding Protein Anillin Cause FSGS

41. Phosphodiesterase 5 inhibition ameliorates angiontensin II-induced podocyte dysmotility via the protein kinase G-mediated downregulation of TRPC6 activity

42. TNXB Mutations Can Cause Vesicoureteral Reflux

43. COLABORADORES

44. Inverted formin 2 mutations with variable expression in patients with sporadic and hereditary focal and segmental glomerulosclerosis

48. Translating genetic findings in hereditary nephrotic syndrome: the missing loops.

49. Human mesenchymal stem cells exert potent antitumorigenic effects in a model of Kaposi's sarcoma

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