36 results on '"Handjiski, Bori"'
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2. High Expression of Survivin and Down-Regulation of Stat-3 Characterize the Feto-Maternal Interface in Failing Murine Pregnancies During the Implantation Period
3. A pivotal role for galectin-1 in fetomaternal tolerance
4. Mast cell deficient and neurokinin-1 receptor knockout mice are protected from stress-induced hair growth inhibition
5. Is there a ‘gut–brain–skin axis’?
6. Zinc as an ambivalent but potent modulator of murine hair growth in vivo– preliminary observations
7. Reduced Uterine Indoleamine 2,3-Dioxygenase Versus Increased Th1/Th2 Cytokine Ratios as a Basis for Occult and Clinical Pregnancy Failure in Mice and Humans
8. A Guide to Assessing Damage Response Pathways of the Hair Follicle: Lessons From Cyclophosphamide-Induced Alopecia in Mice
9. Hair Cycle Control by Estrogens: Catagen Induction via Estrogen Receptor (ER)-α Is Checked by ERβ Signaling
10. Topical Estrogen Accelerates Hair Regrowth in Mice After Chemotherapy-Induced Alopecia by Favoring the Dystrophic Catagen Response Pathway to Damage
11. Topical minoxidil counteracts stress-induced hair growth inhibition in mice
12. A Comprehensive Guide for the Accurate Classification of Murine Hair Follicles in Distinct Hair Cycle Stages
13. Active Hair Growth (Anagen) is Associated with Angiogenesis
14. Control of murine hair follicle regression (catagen) by TGF-β1 in vivo
15. Involvement of hepatocyte growth factor/scatter factor and Met receptor signaling in hair follicle morphogenesis and cycling
16. A Comprehensive Guide for the Recognition and Classification of Distinct Stages of Hair Follicle Morphogenesis
17. Pharmacological Disruption of Hair Follicle Pigmentation by Cyclophosphamide as a Model for Studying the Melanocyte Response to and Recovery from Cytotoxic Drug Damage In Situ
18. Stress-Induced Neurogenic Inflammation in Murine Skin Skews Dendritic Cells Towards Maturation and Migration : Key Role of Intercellular Adhesion Molecule-1/Leukocyte Function-Associated Antigen Interactions
19. Erratum: Corrigendum: A pivotal role for galectin-1 in fetomaternal tolerance
20. Stress-Induced Neurogenic Inflammation in Murine Skin Skews Dendritic Cells Towards Maturation and Migration
21. Electron paramagnetic resonance (EPR) spectroscopy for investigating murine telogen skin after spontaneous or depilation-induced hair growth
22. Towards a “free radical theory of graying”: melanocyte apoptosis in the aging human hair follicle is an indicator of oxidative stress induced tissue damage
23. Splenic eumelanin differs from hair eumelanin in C57BL/6 mice.
24. Neurogenic Inflammation in Stress-Induced Termination of Murine Hair Growth Is Promoted by Nerve Growth Factor
25. Stress Inhibits Hair Growth in Mice by Induction of Premature Catagen Development and Deleterious Perifollicular Inflammatory Events via Neuropeptide Substance P-Dependent Pathways
26. Indications for a brain‐hair follicle axis: inhibition of keratinocyte proliferation and up‐regulation of keratinocyte apoptosis in telogen hair follicles by stress and substance P
27. Overexpression of Bcl-2 Protects from Ultraviolet B-Induced Apoptosis but Promotes Hair Follicle Regression and Chemotherapy-Induced Alopecia
28. Control of murine hair follicle regression (catagen) by TGF‐β1in vivo
29. Intercellular Adhesion Molecule-1 and Hair Follicle Regression
30. A Comprehensive Guide for the Recognition and Classification of Distinct Stages of Hair Follicle Morphogenesis
31. A Murine Model for Inducing and Manipulating Hair Follicle Regression (Catagen): Effects of Dexamethasone and Cyclosporin A
32. A new model for cyclophosphamide-induced alopecia and its manipulation by cyclosporine a and topical dexamethasone
33. Control of murine hair follicle regression (catagen) by TGF‐β1 in vivo
34. Corrigendum: A pivotal role for galectin-1 in fetomaternal tolerance.
35. Pharmacological Disruption of Hair Follicle Pigmentation by Cyclophosphamide as a Model for Studying the Melanocyte Response to and Recovery from Cytotoxic Drug Damage <em>In Situ</em>.
36. Hair cycle control by estrogens: catagen induction via estrogen receptor (ER)-alpha is checked by ER beta signaling.
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