49 results on '"Herrlich, Andreas"'
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2. Sterile kidney tissue injury induces neutrophil swarming in lung alveolar capillaries
3. Functional Genomics Approach Identifies Novel Signaling Regulators of TGFα Ectodomain Shedding
4. Acute Kidney Injury-Induced Circulating TNFR1/2 Elevations Correlate with Persistent Kidney Injury and Progression to Fibrosis
5. Interorgan communication networks in the kidney–lung axis
6. FOXM1 drives proximal tubule proliferation during repair from acute ischemic kidney injury
7. Role of Proneuregulin 1 Cleavage and Human Epidermal Growth Factor Receptor Activation in Hypertonic Aquaporin Induction
8. Data from Tumor Suppressor NF2 Blocks Cellular Migration by Inhibiting Ectodomain Cleavage of CD44
9. Supplementary Methods from Functional Genomics Approach Identifies Novel Signaling Regulators of TGFα Ectodomain Shedding
10. Supplemental Material and Methods from Tumor Suppressor NF2 Blocks Cellular Migration by Inhibiting Ectodomain Cleavage of CD44
11. Supplemental Figure Legends from Tumor Suppressor NF2 Blocks Cellular Migration by Inhibiting Ectodomain Cleavage of CD44
12. Data from Functional Genomics Approach Identifies Novel Signaling Regulators of TGFα Ectodomain Shedding
13. Supplementary Tables from Functional Genomics Approach Identifies Novel Signaling Regulators of TGFα Ectodomain Shedding
14. Supplemental Figures S1-S7 from Tumor Suppressor NF2 Blocks Cellular Migration by Inhibiting Ectodomain Cleavage of CD44
15. Supplementary Figures from Functional Genomics Approach Identifies Novel Signaling Regulators of TGFα Ectodomain Shedding
16. ADAM Metalloprotease-Released Cancer Biomarkers
17. Ligand-Independent Activation of Platelet-Derived Growth Factor Receptor is a Necessary Intermediate in Lysophosphatidic, Acid-Stimulated Mitogenic Activity in L Cells
18. TNF or EGFR inhibition equally block AKI-to-CKD transition: opportunities for etanercept treatment.
19. TNF or EGFR inhibition equally block AKI-to-CKD transition: opportunities for etanercept treatment
20. Mechanisms of interorgan crosstalk in health and disease
21. Interorgan crosstalk mechanisms in disease: the case of acute kidney injury‐induced remote lung injury
22. Identification of kidney injury released circulating osteopontin as causal agent of respiratory failure
23. Regulated ADAM17-dependent EGF family ligand release by substrate-selecting signaling pathways
24. Circulating osteopontin released by injured kidneys causes pulmonary inflammation and edema
25. EGFR-driven profibrotic signals in AKI-to-CKD transition are blocked by TNF inhibition: Opportunities for etanercept treatment
26. Proximal Tubule–Derived Amphiregulin Amplifies and Integrates Profibrotic EGF Receptor Signals in Kidney Fibrosis
27. iRhoms: A Potential Path to More Specific Therapeutic Targeting of Lupus Nephritis
28. Growth factor and co-receptor release by structural regulation of substrate metalloprotease accessibility
29. ADAM17 substrate release in proximal tubule drives kidney fibrosis
30. Proteolytic Activity Matrix Analysis (PrAMA) for simultaneous multiple protease activities
31. Signaling regulators of TGF cleavage predict mechanisms for RTK re‐activation and therapeutic resistance in gastric cancer
32. Abstract B1-33: Network analyses identify hidden regulators of TGFa shedding
33. Distinct Intracellular Domain Substrate Modifications Selectively Regulate Ectodomain Cleavage of NRG1 or CD44
34. Inside-out Regulation of Ectodomain Cleavage of Cluster-of-Differentiation-44 (CD44) and of Neuregulin-1 Requires Substrate Dimerization
35. PI3kα and STAT1 Interplay Regulates Human Mesenchymal Stem Cell Immune Polarization
36. Tumor Suppressor NF2 Blocks Cellular Migration by Inhibiting Ectodomain Cleavage of CD44
37. Abstract 1577: Inside-out regulation of ectodomain protease accessibility in the release of cytokines
38. Loss of Trop2 causes ErbB3 activation through a neuregulin-1-dependent mechanism in the mesenchymal subtype of HNSCC
39. Who decides when to cleave an ectodomain?
40. Epidermal Growth Factor (EGF) Ligand Release by Substrate-specific A Disintegrin and Metalloproteases (ADAMs) Involves Different Protein Kinase C (PKC) Isoenzymes Depending on the Stimulus
41. Proteolytic Activity Matrix Analysis (PrAMA) for simultaneous determination of multiple protease activities
42. Ectodomain cleavage of the EGF ligands HB‐EGF, neuregulin1‐β, and TGF‐α is specifically triggered by different stimuli and involves different PKC isoenzymes
43. Epidermal Growth Factor Receptor Tyrosine Kinase Mediates Ras Activation by Gonadotropin-releasing Hormone
44. Gonadotropin-releasing Hormone Receptor Initiates Multiple Signaling Pathways by Exclusively Coupling to Gq/11Proteins
45. Involvement of Gs and Gi Proteins in Dual Coupling of the Luteinizing Hormone Receptor to Adenylyl Cyclase and Phospholipase C
46. Ectodomain cleavage of the EGF ligands HB-EGF, neuregulin 1-β, and TGF-α is specifically triggered by different stimuli and involves different PKC isoenzymes.
47. Signal characteristics of G protein-transactivated EGF receptor.
48. Involvement of Gsand GiProteins in Dual Coupling of the Luteinizing Hormone Receptor to Adenylyl Cyclase and Phospholipase C*
49. Sterile kidney tissue injury induces neutrophil swarming in lung alveolar capillaries.
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