28 results on '"Homma, Hidenori"'
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2. Dynamic molecular network analysis of iPSC-Purkinje cells differentiation delineates roles of ISG15 in SCA1 at the earliest stage
3. AAV-mediated editing of PMP22 rescues Charcot-Marie-Tooth disease type 1A features in patient-derived iPS Schwann cells
4. PQBP5/NOL10 maintains and anchors the nucleolus under physiological and osmotic stress conditions
5. Necrosis Links Neurodegeneration and Neuroinflammation in Neurodegenerative Disease
6. Identification of hepta-histidine as a candidate drug for Huntington's disease by in silico-in vitro- in vivo-integrated screens of chemical libraries.
7. Prediction and verification of the AD-FTLD common pathomechanism based on dynamic molecular network analysis
8. Tau activates microglia via the PQBP1-cGAS-STING pathway to promote brain inflammation
9. HMGB1 signaling phosphorylates Ku70 and impairs DNA damage repair in Alzheimer’s disease pathology
10. PQBP3/NOL7 is an intrinsically disordered protein
11. Mutant α-synuclein propagates via the lymphatic system of the brain in the monomeric state
12. YAP-dependent necrosis occurs in early stages of Alzheimer’s disease and regulates mouse model pathology
13. The intellectual disability gene PQBP1 rescues Alzheimer’s disease pathology
14. HMGB1 facilitates repair of mitochondrial DNA damage and extends the lifespan of mutant ataxin‐1 knock‐in mice
15. Comprehensive phosphoproteome analysis unravels the core signaling network that initiates the earliest synapse pathology in preclinical Alzheimerʼs disease brain
16. Hepta-Histidine Inhibits Tau Aggregation
17. DNA damage in embryonic neural stem cell determines FTLDs’ fate via early-stage neuronal necrosis
18. Ser46-Phosphorylated MARCKS Is a Marker of Neurite Degeneration at the Pre-aggregation Stage in PD/DLB Pathology
19. Targeting Tyro3 ameliorates a model of PGRN-mutant FTLD-TDP via tau-mediated synaptic pathology
20. Developmental YAPdeltaC determines adult pathology in a model of spinocerebellar ataxia type 1
21. Targeting TEAD/YAP-transcription-dependent necrosis, TRIAD, ameliorates Huntington’s disease pathology
22. HMGB1, a pathogenic molecule that induces neurite degeneration via TLR4-MARCKS, is a potential therapeutic target for Alzheimer’s disease
23. RpA1 ameliorates symptoms of mutant ataxin-1 knock-in mice and enhances DNA damage repair
24. Fasting activates macroautophagy in neurons of Alzheimer’s disease mouse model but is insufficient to degrade amyloid-beta
25. HMGB 1 facilitates repair of mitochondrial DNA damage and extends the lifespan of mutant ataxin‐1 knock‐in mice
26. Comprehensive phosphoproteome analysis unravels the core signaling network that initiates the earliest synapse pathology in preclinical Alzheimer's disease brain
27. An Automatic Creation Method of the Query Vector for the Semantic Associative Search Corresponding to the Impression of Sound of Arbitrary Words
28. Loss of function of VCP/TER94 causes neurodegeneration in the Drosophila central nervous system.
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