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We evaluated the effects of different exercise types suitable for a home/work setting on the postprandial glucose response. Twenty-three healthy, active, young individuals performed one of two studies (12 in Study 1 and 11 in Study 2), with four randomized protocols each. After a meal high in carbohydrate content (1 g of carbohydrate per kg of body weight), in Study 1, participants performed 30 min of either walking (WALK), bench stepping exercise (STEP) or isometric wall squat (SQUAT); in Study 2, participants performed 30 min of either walking (WALK), neuromuscular electrical stimulation alone (P_NMES) or superimposed on voluntary muscle contraction (VC_NMES). In both studies, participants performed a prolonged sitting condition (CON) that was compared to the exercise sessions. In Study 1, WALK and STEP significantly reduced the glucose peak compared to CON (p < 0.011). In Study 2, the peak was significantly reduced in WALK compared to CON, P_NMES and VC_NMES (p < 0.011) and in VC_NMES compared to CON and P_NMES (p < 0.011). A significant reduction of 3 h glucose iAUC was found for WALK and VC_NMES compared to CON and P_NMES (p < 0.033). In conclusion, WALK is the most effective strategy for improving the postprandial glycemic response. However, STEP and VC_NMES can also be used for reducing postprandial glycemia.

BackgroundProlonged or unaccustomed eccentric exercise may cause muscle damage and depending from its extent, this event negatively affects physical performance.ObjectivesThe aim of the present investigation was to evaluate, in humans, the effect of the flavonoid quercetin on circulating levels of the anabolic insulin-like growth factor 1 (IGF-I) and insulin-like growth factor 2 (IGF-II), produced during the recovery period after an eccentric-induced muscle damage (EIMD).MethodsA randomized, double-blind, crossover study has been performed; twelve young men ingested quercetin (1 g/day) or placebo for 14 days and then underwent an eccentric-induced muscle damaging protocol. Blood samples were collected, and cell damage markers [creatine kinase (CK), lactate dehydrogenase (LDH) and myoglobin (Mb)], the inflammatory responsive interleukin 6 (IL-6), IGF-I and IGF-II levels were evaluated before the exercise and at different recovery times from 24 hours to 7 days after EIMD.ResultsWe found that, in placebo treatment the increase in IGF-I (72 h) preceded IGF-II increase (7 d). After Q supplementation there was a more marked increase in IGF-I levels and notably, the IGF-II peak was found earlier, compared to placebo, at the same time of IGF-I (72 h). Quercetin significantly reduced plasma markers of cell damage [CK (pConclusionsOur data are encouraging about the use of quercetin as dietary supplementation strategy to adopt in order to mitigate and promote a faster recovery after eccentric exercise as suggested by the increase in plasma levels of the anabolic factors IGF-I and IGF-II.

Purpose Despite their widespread use in exercise physiology, time-to-exhaustion (TTE) tests present an often-overlooked challenge to researchers, which is how to computationally deal with between- and within-subject differences in exercise duration. We aimed to verify the best analysis method to overcome this problem. Methods Eleven cyclists performed an incremental test and three TTE tests differing in workload as preliminary tests. The TTEs were used to derive the individual power–duration relationship needed to set the workload (corresponding to an estimated TTE of 1200 s) for four identical experimental TTE tests. Within individuals, the four tests were subsequently rank ordered by performance. Physiological and psychological variables expected to change with performance were analysed using different methods, with the main aim being to compare the traditional “group isotime” method and a less-used “individual isotime” method. Results The four tests, ranked from the best to the worst, had a TTE of 1526 ± 332, 1425 ± 313, 1295 ± 325, and 1026 ± 265 s. Ratings of perceived exertion, minute ventilation, respiratory frequency, and affective valence were sensitive to changes in performance when their responses were analysed with the “individual isotime” method (P ηp2 > 0.144) but not when using the “group isotime” method, because the latter resulted in partial data loss. Conclusions The use of the “individual isotime” method is strongly encouraged to avoid the misinterpretation of the phenomenon under study. Important implications are not limited to constant-workload exercise, but extend to incremental exercise, which is another commonly used test of exercise tolerance.

Postprandial exercise represents an important tool for improving the glycemic response to a meal. This study evaluates the effects of the combination and sequence of different exercise types on the postprandial glycemic response in patients with type 2 diabetes. In this repeated-measures crossover study, eight patients with type 2 diabetes performed five experimental conditions in a randomized order: (i) uninterrupted sitting (CON); (ii) 30 min of moderate intensity aerobic exercise (walking) (A); (iii) 30 min of combined aerobic and resistance exercise (AR); (iv) 30 min of combined resistance and aerobic exercise (RA); and (v) 15 min of resistance exercise (R). All the exercise sessions started 30 min after the beginning of a standardized breakfast. All the exercise conditions showed a significant attenuation of the post-meal glycemic excursion (P < 0.003) and the glucose incremental area under the curve at 0–120 min (P < 0.028) and 0–180 min (P < 0.048) compared with CON. A greater reduction in the glycemic peak was observed in A and AR compared to RA (P < 0.02). All the exercise types improved the post-meal glycemic response in patients with type 2 diabetes, with greater benefits when walking was performed alone or before resistance exercise.

Purpose We systematically investigated the effects of different exercise strategies on postprandial glycemia. Methods Six randomized repeated-measures crossover studies were performed. Study 1 compared the effects of 60 min of brisk walking started at 30, 60, or 90 min after breakfast on postbreakfast and postlunch glycemic responses. Study 2 investigated the effects of 30 min of different exercise types (aerobic vs resistance vs combined). Study 3 compared the effects of 30 min of different aerobic exercise types (walking vs cycling vs elliptical). Study 4 evaluated the effects of 30 min of brisk walking performed 45 min before or 15 and 30 min after breakfast. Study 5 compared 30 with 45 min of postprandial brisk walking. Study 6 compared the effects of a total of 30 min brisk walking exercise fragmented in bouts of 15, 5, or 2.5 min performed every 15 min. Results Postprandial but not preprandial exercise improved glycemic response (studies 1 and 4). The glycemic peak was attenuated only when exercise started 15 min after the meal (study 4). A similar reduction of the postprandial glycemic response was observed with different exercise types (studies 2 and 3). Thirty and 45 min of brisk walking provided a similar reduction of the postprandial glucose response (study 5). When performing activity breaks, 10 and 20 min of cumulative exercise were sufficient to attenuate postprandial glycemia in the first hour postmeal (study 6). Conclusion Our findings provide insight into how to choose timing, type, duration, and modality for postprandial exercise prescription in healthy individuals.

Exercise plays an important role in preventing and treating postprandial dysmetabolism. We investigated the postprandial metabolic responses to a standard lunch when a session of aerobic exercise is performed in the early postprandial phase or divided between the pre- and postprandial period. Nine healthy volunteers consumed a standardised mixed lunch and rested for the following 3 h (Con) or performed 40 min of cycling at 65% VO2max after lunch (CPPEx), or two 20-min sessions, one before (SplitEx1) and the other after lunch (SplitEx2), at the same intensity. At 1-h post-lunch, a significant reduction (P

We evaluated the effect of postprandial walking on the post-meal glycemic response after meals with different characteristics. Twenty-one healthy young volunteers participated in one of two randomized repeated measures studies. Study 1 (10 participants) assessed the effects of 30 min of brisk walking after meals with different carbohydrate (CHO) content (0.75 or 1.5 g of CHO per kg/body weight). Study 2 (11 participants) evaluated the effects of 30 min of brisk walking after consuming a mixed meal or a CHO drink matched for absolute CHO content (75 g). Postprandial brisk walking substantially reduced (p < 0.009) the glucose peak in both studies, with no significant differences across conditions. When evaluating the glycemic response throughout the two hours post-meal, postprandial walking was more effective after consuming a lower CHO content (Study 1), and similarly effective after a mixed meal or a CHO drink (Study 2), although higher glucose values were observed when consuming the CHO drink. Our findings show that a 30 min postprandial brisk walking session improves the glycemic response after meals with different CHO content and macronutrient composition, with implications for postprandial exercise prescription in daily life scenarios.

This study was aimed at investigating whether quercetin (Q) may improve the recovery of neuromuscular function and biochemical parameters in the 7 days following an eccentric exercise-induced muscle damage (EEIMD). Sixteen men (25.9 &plusmn, 3.3 y) ingested Q (1000 mg/day) or placebo (PLA) for 14 days following a double-blind crossover study design. A neuromuscular (NM) test was performed pre&ndash, post, 24 h, 48 h, 72 h, 96 h and 7 days after an intense eccentric exercise. The force&ndash, velocity relationship of the elbow flexor muscles and their maximal voluntary isometric contraction (MVIC) were recorded simultaneously to the electromyographic signals (EMG). Pain, joint angle, arm circumference, plasma creatine kinase (CK) and lactate-dehydrogenase (LDH) were also assessed. The results showed that Q supplementation significantly attenuated the strength loss compared to PLA. During the recovery, force&ndash, velocity relationship and mean fibers conduction velocity (MFCV) persisted significantly less when participants consumed PLA rather than Q, especially at the highest angular velocities (p &lt, 0.02). A greater increase in biomarkers of damage was also evident in PLA with respect to Q. Q supplementation for 14 days seems able to ameliorate the recovery of eccentric exercise-induced weakness, neuromuscular function impairment and biochemical parameters increase probably due to its strong anti-inflammatory and antioxidant action.

Pedalling cadence influences respiratory frequency (fR) during exercise, with group III/IV muscle afferents possibly mediating its effect. However, it is unclear how exercise intensity affects the link between cadence and fR. We aimed to test the hypothesis that the effect of cadence on fR is moderated by exercise intensity, with interest in the underlying mechanisms. Ten male cyclists performed a preliminary ramp incremental test and three sinusoidal experimental tests on separate visits. The experimental tests consisted of 16 min of sinusoidal variations in cadence between 115 and 55 rpm (sinusoidal period of 4 min) performed during passive exercise (PE), moderate exercise (ME) and heavy exercise (HE). The amplitude (A) and phase lag (φ) of the dependent variables were calculated. During PE, fR changed in proportion to variations in cadence (r = 0.85, P

To examine the effect of acute quercetin (Q) ingestion on neuromuscular function, biomarkers of muscle damage, and rate of perceived exertion (RPE) in response to an acute bout of resistance training. 10 young men (22.1 ± 1.8 years, 24.1 ± 3.1 BMI) participated in a randomized, double-blind, crossover study. Subjects consumed Q (1 g/day) or placebo (PLA) 3 h prior to a resistance training session which consisted of 3 sets of 8 repetitions at 80% of the one repetition maximum (1RM) completed bilaterally for eight different resistance exercises. Electromyographic (EMG) signals were recorded from the knee extensor muscles during maximal isometric (MVIC) and isokinetic voluntary contractions, and during an isometric fatiguing test. Mechanical and EMG signals, biomarkers of cell damage, and RPE score were measured PRE, immediately POST, and 24 h (blood indices only) following the resistance exercise. After a single dose of Q, the torque–velocity curve of knee extensors was enhanced and after the resistance exercise, subjects showed a lower MVIC reduction (Q: 0.91 ± 6.10%, PLA: 8.66 ± 5.08%) with a greater rate of torque development (+ 10.6%, p

The polyphenolic flavonoid quercetin has been shown to be a powerful antioxidant, in vitro and in murine models. However, its effect on redox status has been poorly examined in humans, particularly in combination with strenuous exercise. We hypothesized that quercetin supplementation would beneficially affect redox homeostasis in healthy individuals undergoing eccentric exercise. To test this hypothesis, the effects of chronic consumption of quercetin on glutathione system (reduced, oxidized, and reduced to oxidized glutathione ratio), oxidative damage [thiobarbituric acid reactive substances (TBARs)], antioxidant enzymatic network (catalase, glutathione peroxidase, superoxide dismutase) and resistance to lysis, were investigated in erythrocytes, a traditional model widely used to study the effects of oxidative stress as well as the protective effects of antioxidants. In a two weeks controlled, randomized, crossover, intervention trial, 14 individuals ingested 2 caps (1 g/d) of quercetin or placebo. Blood samples were collected before, after 2 weeks of supplementation and after a bout of eccentric exercise. Quercetin, reduced significantly erythrocytes lipid peroxidation levels and the susceptibility to hemolysis induced by the free radical generator AAPH, while no differences in antioxidant enzyme activities and glutathione homeostasis were found between the two groups. After a single bout of eccentric exercise, quercetin supplementation improved redox status as assessed by reduced/oxidized glutathione ratio analysis and reduced TBARs levels both in erythrocytes and plasma. In conclusion, our study provides evidences that chronic quercetin supplementation has antioxidant potential prior to and after a strenuous eccentric exercise thus making the erythrocytes capable to better cope with an oxidative insult.

Purpose:To determine whether repeated carbohydrate (CHO) mouth rinsing would improve neuromuscular performance during high-intensity fatiguing contractions.Methods:Eighteen young men (age 26.1 ± 5.0 y, BMI 22.9 ± 1.9) performed 3 maximal voluntary isometric contractions (MVICPRE). Immediately after, they completed 10-second mouth rinse with 6.4% maltodextrin solution (MAL), 7.1% glucose solution (GLU), water (W), artificially sweetened solution (PLA), or a control trial with no rinse (CON) in a crossover protocol. Subjects performed 5 sets of 30 isokinetic fatiguing contractions at 180°/s, and an MVICPOST with their elbow flexors was performed after each mouth rinse. Mechanical and electromyographic (EMG) signals were recorded from the biceps brachii and parameters of interest analyzed.Results:When rinsing the mouth with a solution containing CHO, independently of the sweetness, isokinetic performance was enhanced as shown by the greater total work achieved in comparison with CON. The decay of torque and mean fiber-conduction velocity (MFCV) recorded at the end of the fatiguing task was lower when rinsing the mouth with GLU than with CON. The torque recorded during the MVICPOST was greater with CHO with respect to CON, and this was associated to a lower decay of MFCV.Conclusions:CHO mouth rinse counteracts fatigue-induced decline in neuromuscular performance, supporting the notion that CHO rinse may activate positive afferent signals able to modify motor output. Repeated mouth rinsing with sweet and nonsweet CHO-containing solutions can improve neuromuscular performance during an isokinetic intermittent fatiguing task.

Since respiratory frequency (fR) has been proposed as a good marker of physical effort, furthering the understanding of how fR is regulated during exercise is of great importance. We manipulated recovery intensity and exercise duration during high-intensity interval training (HIIT) to test the hypothesis that fast inputs (including central command) contribute more than metabolic stimuli to fR regulation. Seven male cyclists performed an incremental test, a 10 min and a 20 min continuous time trial (TT) as preliminary tests. Subsequently, recovery intensity and exercise duration were manipulated during HIIT (30 s work and 30 s active recovery) by performing four 10 min and one 20 min trial (recovery intensities: 85%, 70%, 55% and 30% of the 10 min TT mean workload; 85% of the 20 min TT mean workload). The work intensity of the HIIT sessions was self-paced by participants to achieve the best performance possible. When manipulating recovery intensity, fR, but not tidal volume (VT), showed a fast response to the alternation of the work and recovery phases, proportional to the extent of workload variations. No association between fR and gas exchange responses was observed. When manipulating exercise duration, fR and Rating of Perceived Exertion (RPE) were dissociated from VT, VCO2 and VO2 responses. Overall, RPE was strongly correlated with fR (r = 0.87; P

Although it is widely accepted that diabetic polyneuropathy (DPN) is linked to a marked decline in neuromuscular performance, information on the possible impact of type 1 diabetes (T1D) on muscle strength and fatigue remains unclear. The purpose of this study was to investigate the effects of T1D and DPN on strength and fatigability in knee extensor muscles. Thirty-one T1D patients (T1D), 22 T1D patients with DPN (DPN) and 23 matched healthy control participants (C) were enrolled. Maximal voluntary contraction (MVC) and endurance time at an intensity level of 50% of the MVC were assessed at the knee extensor muscles with an isometric dynamometer. Clinical characteristics of diabetic patients were assessed by considering a wide range of vascular and neurological parameters. DPN group had lower knee extensor muscles strength than T1D (−19%) and the C group (−37.5%). T1D group was 22% weaker when compared to the C group. Lower body muscle fatigability of DPN group was 22 and 45.5% higher than T1D and C group, respectively. T1D group possessed a higher fatigability (29.4%) compared to C group. A correlation was found between motor and sensory nerve conduction velocity and muscle strength and fatigability. Patients with T1D are characterised by both a higher fatigability and a lower muscle strength, which are aggravated by DPN. Our data suggest that factors other than nervous damage play a role in the pathogenesis of such defect.

PURPOSE Motor unit conduction velocity (MUCV) represents the propagation velocity of action potentials along the muscle fibers innervated by individual motor neurons and indirectly reflects the electrophysiological properties of the sarcolemma. In this study, we investigated the effect of a 4-wk strength training intervention on the peripheral properties (MUCV and motor unit action potential amplitude, RMSMU) of populations of longitudinally tracked motor units (MU). METHODS The adjustments exhibited by 12 individuals who participated in the training (INT) were compared with 12 controls (CON). Strength training involved ballistic (4 × 10) and sustained (3 × 10) isometric ankle dorsiflexions. Measurement sessions involved the recordings of maximal voluntary isometric force and submaximal isometric ramp contractions, whereas high-density surface EMG was recorded from the tibialis anterior. High-density surface EMG signals were decomposed into individual MU discharge timings, and MU was tracked across the intervention. RESULTS Maximal voluntary isometric force (+14.1%, P = 0.003) and average MUCV (+3.0%, P = 0.028) increased in the INT group, whereas normalized MU recruitment threshold (RT) decreased (-14.9%, P = 0.001). The slope (rate of change) of the regression between MUCV and MU RT increased only in the INT group (+32.6%, P = 0.028), indicating a progressive greater increase in MUCV for higher-threshold MU. The intercept (initial value) of MUCV did not change after the intervention (P = 0.568). The association between RMSMU and MU RT was not altered by the training. CONCLUSION The increase in the rate of change in MUCV as a function of MU RT, but not the initial value of MUCV, suggests that short-term strength training elicits specific adaptations in the electrophysiological properties of the muscle fiber membrane in high-threshold MU.

Purpose: Variables currently used in soccer training monitoring fail to represent the physiological demand of the player during movements like accelerations, decelerations, and directional changes performed at high intensity. We tested the hypothesis that respiratory frequency (fR) is a marker of physical effort during soccer-related high-intensity exercise. Methods: A total of 12 male soccer players performed a preliminary intermittent incremental test and 2 shuttle-run high-intensity interval training (HIIT) protocols, in separate visits. The 2 HIIT protocols consisted of 12 repetitions over 9 minutes and differed in the work-to-recovery ratio (15:30 vs 30:15 s). Work rate was self-paced by participants to achieve the longest possible total distance in each HIIT protocol. Results: Work-phase average metabolic power was higher (P −1) compared with the 30:15-second protocol (22.8 [2.0] W·kg−1). Unlike heart rate and oxygen uptake, fR showed a fast response to the work–recovery alternation during both HIIT protocols, resembling changes in metabolic power even at supramaximal intensities. Large correlations (P fR and rating of perceived exertion during both 15:30-second (r = .87) and 30:15-second protocols (r = .85). Conclusions: Our findings suggest that fR is a good marker of physical effort during shuttle-run HIIT in soccer players. These findings have implications for monitoring training in soccer and other team sports.

The aim of the present investigation was to test the hypothesis that quercetin (Q) may prevent the strength loss and neuromuscular impairment associated with eccentric exercise-induced muscle damage (EEIMD). Twelve young men (26.1 &plusmn, 3.1 years) ingested either Q (1000 mg/day) or placebo (PLA) for 14 days using a randomized, double-blind, crossover study design. Participants completed a comprehensive neuromuscular (NM) evaluation before, during and after an eccentric protocol able to induce a severe muscle damage (10 sets of 10 maximal lengthening contractions). The NM evaluation comprised maximal voluntary isometric contraction (MVIC) and force&ndash, velocity relationship assessments with simultaneous recording of electromyographic signals (EMG) from the elbow flexor muscles. Soreness, resting arm angle, arm circumference, plasma creatine kinase (CK) and lactate dehydrogenase (LDH) were also assessed. Q supplementation significantly increased the isometric strength recorded during MVIC compared to baseline (+4.7%, p &lt, 0.05). Moreover, the torque and muscle fiber conduction velocity (MFCV) decay recorded during the eccentric exercise was significant lower in Q compared to PLA. Immediately after the EEIMD, isometric strength, the force&ndash, velocity relationship and MFCV were significantly lower when participants were given PLA rather than Q. Fourteen days of Q supplementation seems able to attenuate the severity of muscle weakness caused by eccentric-induced myofibrillar disruption and sarcolemmal action potential propagation impairment.

KEY POINTS Previous studies have indicated that several weeks of strength training is sufficient to elicit significant adaptations in the neural drive sent to the muscles. There are few data, however, on the changes elicited by strength training in the recruitment and rate coding of motor units during voluntary contractions. We show for the first time that the discharge characteristics of motor units in the tibialis anterior muscle tracked across the intervention are changed by 4 weeks of strength training with isometric voluntary contractions. The specific adaptations included significant increases in motor unit discharge rate, decreases in the recruitment-threshold force of motor units and a similar input-output gain of the motor neurons. The findings suggest that the adaptations in motor unit function may be attributable to changes in synaptic input to the motor neuron pool or to adaptations in intrinsic motor neuron properties. ABSTRACT The strength of a muscle typically begins to increase after only a few sessions of strength training. This increase is usually attributed to changes in the neural drive to muscle as a result of adaptations at the cortical or spinal level. We investigated the change in the discharge characteristics of large populations of longitudinally tracked motor units in tibialis anterior before and after 4 weeks of strength training the ankle-dorsiflexor muscles with isometric contractions. The adaptations exhibited by 14 individuals were compared with 14 control subjects. High-density electromyogram grids with 128 electrodes recorded the myoelectric activity during isometric ramp contractions to the target forces of 35%, 50% and 70% of maximal voluntary force. The motor unit recruitment and derecruitment thresholds, discharge rate, interspike intervals and estimates of synaptic inputs to motor neurons were assessed. The normalized recruitment-threshold forces of the motor units were decreased after strength training (P

Purpose: To indirectly evaluate the effect of androgens on neuromuscular system in humans we analyzed if an induced short-term hypogonadal state (serum total testosterone-TT.

Muscle fibre conduction velocity (MFCV) is a basic physiological parameter biophysically related to the diameter of muscle fibres and properties of the sarcolemma. The aim of this study was to assess the intersession reproducibility of the relation between voluntary force and estimates of average muscle fibre conduction velocity (MFCV) from multichannel high-density surface electromyographic recordings (HDsEMG). Ten healthy men performed six linearly increasing isometric ankle dorsiflexions on two separate experimental sessions, 4 weeks apart. Each session involved the recordings of voluntary force during maximal isometric (MViF) and submaximal ramp contractions at 35–50–70% of MViF. Concurrently, the HDsEMG activity was detected from the tibialis anterior muscle and MFCV estimates were derived in 250-ms epochs. Absolute and relative reproducibility of MFCV initial value (intercept) and rate of change (regression slope) as a function of force were assessed by within-subject coefficient of correlation (CVw) and with intraclass correlation coefficient (ICC). MFCV was positively correlated with voluntary force (R2 = 0.75 ± 0.12) in all individuals and test conditions (P Overall, MFCV regression coefficients showed a high degree of intersession reproducibility in both absolute and relative terms. These results may have important practical implications in the tracking of training-induced neuromuscular changes and/or in the monitoring of the progress of neuromuscular disorders when a full sEMG signal decomposition is problematic or not possible.

Introduction Walking on vibrating floor causes a complex exposure pattern and the superimposition of walk and vibration may induce early muscular fatigue.1 The problem is relevant is many field, as sea platform or railway transports. The present study studies the leg muscular activation and stride phases during walking under vibration to derive a muscle model in these circumstances. Methods Subjects walked on a treadmill positioned on a 6-DOF vibrating table. Vibration was imposed at four frequencies (4, 8, 12, 16 Hz) along vertical and transversal direction. The walking speed was set at 1.25 m/s. Surface electromyography (sEMG) of four muscles was recorded. Stride phases were recorded using accelerometers and stride length was calculated. Acceleration signals were acquired in several body districts (foot, knee and hip). All measurements were related to the walking condition without the vibration. Result Preliminary results showed that vibration does not affect stride length and step phases. The muscular activation patterns exhibit frequency related modification, in terms of sEMG bursts amplitude and timing. There is a linear correlation between 8 Hz frequency and muscular activation. Discussion Transmitted vibration triggers a tonic vibration reflex (TVR) that is related to mechanical frequencies.2 TVR is also related to the motor task because of the mechanical coupling between vibrator and biological apparatus.3 These facts could explain the modifications in leg muscle activation revealed with sEMG. References . Fattorini L, Tirabasso A, Lunghi A, Di Giovanni R, Sacco F, Marchetti E. Muscular forearm activation in hand-grip tasks with superimposition of mechanical vibrations. J Electromyogr Kinesiol2016;26:143–148. . Eklund G, Hagbarth KE. Normal variability of tonic vibration reflexes in man. Exp Neurol1966;16:80–92. . Fattorini L, Tirabasso A, Lunghi A, Di Giovanni R, Sacco F, Marchetti E. Muscular synchronisation and hand-arm fatigue. Int J Ind Ergon2017.

The force developed by the human neuromuscular system can change very rapidly (15–50 ms). When processing EMG signals for inferring neural control strategies, it is therefore necessary to extract estimates from short time intervals. In this study, we investigate the relation between joint torque and estimates of average muscle fibre conduction velocity (MFCV) and amplitude (RMS) from surface EMG signals, when varying the duration of the processing interval. Moreover, we assessed the inter-subject variability in RMS and MFCV estimates. Ten healthy subjects performed isometric linearly increasing ankle dorsiflexion contractions up to 70% MVC at a rate of 5%MVCs−1. High-density EMG signals were recorded from the tibialis anterior muscle and MFCV and RMS were estimated in eight time-intervals ranging from 15 to 2000 ms. MFCV and RMS were significantly correlated with force in all subjects and when using all time-intervals (MFCV = 0.77 ± 0.07, RMS = 0.79 ± 0.06 (R2), Pearson-P

Diabetic patients are at higher risk of developing physical disabilities than non-diabetic subjects. Physical disability appears to be related, at least in part, to muscle dysfunction. Several studies have reported reduced muscle strength and power under dynamic and static conditions in both the upper and lower limbs of patients with type 2 diabetes. Additional effects of diabetes include a reduction in muscle mass, quality, endurance and an alteration in muscle fibre composition, though the available data on these parameters are conflicting. The impact of diabetes on neuromuscular function has been related to the co-existence of long-term complications. Peripheral neuropathy has been shown to affect muscle by impairing motor nerve conduction. Also, vascular complications may contribute to the decline in muscle strength. However, muscle dysfunction occurs early in the course of diabetes and affects also the upper limbs, thus suggesting that it may develop independently of micro and macrovascular disease. A growing body of evidence indicates that hyperglycaemia may cause an alteration of the intrinsic properties of the muscle to generate force, via several mechanisms. Recently, resistance exercise has been shown to be an effective strategy to counteract the deterioration of muscular performance. High-intensity exercise seems to provide greater benefits than moderate-intensity training, whereas the effect of a power training is yet unknown. This article reviews the available literature on the impairment of muscle function induced by diabetes, the underlying mechanisms, and the effect of resistance training on this defect. Copyright © 2015 John Wiley & Sons, Ltd.

The present study tested the possibility of a localized fat mass (FM) reduction by means of training programmes focusing on specific bodily regions.Sixteen physically inactive women (age: 31±4; BMI: 27.5±2.1), randomly allocated to two groups, completed an 8 week training programme. In one group (UpBdResist) training sessions consisted of upper body resistance exercises followed by 30-minute cycling at 50%VO2max, while the other group (LwBdResist) performed lower body resistance exercises followed by 30 minutes on an arm-ergometer. Regional body composition was assessed by DEXA and skin fold measures.Regardless of a similar reduction in both groups, UpBdResist training elicited a greater reduction of the upper limbs (UL) FM as compared to the lower limbs (LL) (Δ% UL vs. LL: -12.1±3.4 vs. -4.0±4.7; P=0.02). Conversely, in the LwBdResist group, FM loss was more pronounced in the LL as compared to the UL (Δ% UL vs. LL: -2.3±7.0 vs. -11.5±8.2, P=0.02). Likewise, LwBdResist elicited a larger effect on lean mass (LM) of the LL as compared to UL (Δ% LL vs. UL: +8.4±5.8 vs. -2.7±5.0, P0.01), yet no differences between upper and lower limb LM changes were detected in UpBdResist group.The present data suggest that a training programme entailing localized explosive resistance exercise, prior to an endurance exercise bout, may target specific adipose tissue sites eliciting localised fat mass loss in the upper and lower limbs.

What is the central question of this study? By manipulating recovery intensity and exercise duration during high-intensity interval training (HIIT), we tested the hypothesis that fast inputs contribute more than metabolic stimuli to respiratory frequency (f

BACKGROUND The bulk of research investigating soccer player's performance has been concentrated on the metabolic demand, while only few studies focused on the neuromuscular activation. The present study aimed at investigating the activation profile of the leg muscles throughout a 90- minute soccer match. METHODS Fifteen football players (18.3±0.7 years) performed: 1) an isometric maximal voluntary contraction (MVC) before the game [MVCpre]; 2) a 90-minute soccer match (composed of two 45-minute periods separated by a 15-minute rest); 3) a second MVC after the match [MVCpost]. Electromyographic (EMG) activity of the Vastus Lateralis (VL) muscle of the dominant leg was recorded during the match. The root mean square (RMS) of the EMG signals was normalized for the maximal RMS obtained during the MVCpre (100%RMSmax) and six intensity classes were created in order to represent the %RMS distribution during the match (1st: 0-20%RMSmax; 2nd: 20-40%RMSmax; 3rd: 40-60%RMSmax; 4th: 60-80%RMSmax; 5th: 80-100%RMSmax; 6th: 100-120%RMSmax). RESULTS After the 90-minute soccer match, knee extensor MVC failed to show any statistical difference from pre-game values (-4.2%; P>0.05) whilst the neuromuscular activation demonstrated a significant reduction (-26.3%, P

Purpose:To investigate the effects of work-to-rest-ratio manipulation on neuromuscular and metabolic responses during 2 high-intensity intermittent training (HIT) protocols to exhaustion. Since different exercise durations were expected, the authors hypothesized that the protocol registering a longer duration would have a more pronounced effect on neuromuscular responses, while the other would challenge the cardiopulmonary system more.Methods:Thirteen competitive cyclists (age 19 ± 2 y) performed a preliminary incremental test to identify their maximal power output and 2 intermittent protocols to exhaustion (40:20s and 30:30s) at a fixed work rate of 135%Pmax interspersed by passive recovery. Surface electromyographic (sEMG) parameters (including muscle-fiber conduction velocity), cardiopulmonary parameters, and blood lactate concentration [La−] were recorded.Results:Time to exhaustion and total work were significantly higher for the 30:30s (38 ± 13 min, 495 ± 161 kJ) than for the 40:20s (10 ± 3 min, 180 ± 51 kJ). No differences were found in sEMG parameters for the 2 protocols. Mean and peak values of VO2, heart rate, ventilatory parameters (except for the peak value of respiratory frequency), and [La−] were significantly higher in the 40:20s than in the 30:30s.Conclusions:These results do not support the hypothesis that a longer time spent at high intensity has a more pronounced effect on neuromuscular responses, as no differences in EMG parameters were found in the 2 HIT protocols. Regarding metabolic responses, while the 40:20s led to maximal values of VO2, [La−], and ventilatory parameters within a few minutes, the 30:30s allowed maintenance of moderately high values for a considerably longer period, especially for [La−] and ventilatory parameters.

Purpose:To verify the accuracy of predicting performance in the severe-intensity domain by means of end-test power output (EP) and the work performed above EP (WEP) obtained from a 3-min all-out test in competitive cyclists.Methods:Ten welltrained cyclists performed a ramp incremental test and a 3-min all-out familiarization test. Subsequently, they performed a 3-min all-out experimental test to obtain EP and WEP and a 10-min time trial (TT). The actual 10-min-TT mean power output was then compared with the power output predicted as P = WEP/Tlim + EP, where Tlim corresponds to 600 s. The ramp-test peak power output (PPO) was compared with PPO predicted as , where S represents the ramp slope (0.5 W/s).Results:The actual (347 ± 30 W) and predicted (376 ± 48 W) 10-min TT mean power output were correlated (r = .87, P = .001) but significantly different (P < .01). The coefficient of variation (CV) between the predicted and actual performance was 5.6% ± 4.4%. The error of prediction was positively correlated to EP (r = .80, P = .005) and negatively correlated to WEP (r = –.71, P = .021). No significant difference was found between the 10-min-TT mean power output and EP (351 ± 53 W). The actual (438 ± 30 W) and predicted (472 ± 41 W) ramp PPO were correlated (r = .88, P < .001) but significantly different (P < .001). The CV between the predicted and actual PPO was 5.2% ± 3%. The error of prediction was positively correlated to EP (r = .63, P = .051).Conclusions:EP and WEP obtained from a 3-min all-out test overestimate severe-intensity performance in competitive cyclists.

Diabetic patients are at higher risk of developing physical disabilities than non-diabetic subjects. Physical disability appears to be related, at least in part, to muscle dysfunction. Several studies have reported reduced muscle strength and power under dynamic and static conditions in both the upper and lower limbs of patients with type 2 diabetes. Additional effects of diabetes include a reduction in muscle mass, quality, endurance and an alteration in muscle fibre composition, though the available data on these parameters are conflicting. The impact of diabetes on neuromuscular function has been related to the co-existence of long-term complications. Peripheral neuropathy has been shown to affect muscle by impairing motor nerve conduction. Also, vascular complications may contribute to the decline in muscle strength. However, muscle dysfunction occurs early in the course of diabetes and affects also the upper limbs, thus suggesting that it may develop independently of micro and macrovascular disease. A growing body of evidence indicates that hyperglycaemia may cause an alteration of the intrinsic properties of the muscle to generate force, via several mechanisms. Recently, resistance exercise has been shown to be an effective strategy to counteract the deterioration of muscular performance. High-intensity exercise seems to provide greater benefits than moderate-intensity training, whereas the effect of a power training is yet unknown. This article reviews the available literature on the impairment of muscle function induced by diabetes, the underlying mechanisms, and the effect of resistance training on this defect. Copyright © 2015 John WileySons, Ltd.

Introduction: The effect of recovery time on neuromuscular function after a fatiguing task was compared in both the upper and lower limbs between patients with Charcot–Marie–Tooth type 1A (CMT1A) and healthy individuals. Methods: Torque of elbow flexors and knee extensors and surface electromyography (sEMG) data of biceps brachii and vastus lateralis were recorded from 8 CMT1A patients and 8 matched, healthy individuals during maximal voluntary contraction (MVC) before (pre-fatigue MVC), 10 s after (10-s post-fatigue MVC), and 10 min after (10-min post-fatigue MVC) a fatiguing task at 80% MVC until exhaustion. Results: Only in the lower limb, torque and root mean square of sEMG (RMS) during pre-fatigue MVC were lower (P < 0.05) in patients (91.93 ± 45.95 Nm, 0.11 ± 0.07 mV) than in controls (161.06 ± 75.5 Nm, 0.24 ± 0.16 mV). In the 10-min post-fatigue MVC, muscle-fiber conduction velocity (MFCV) and RMS, expressed as a percentage of pre-fatigue MVC, were lower (P < 0.05) in patients (MFCV 90.3 ± 6.91%, RMS 84.50 ± 9.89%) than in controls (MFCV 100.87 ± 5.1%, RMS 92.71 ± 11.84%). Conclusions: CMT1A patients are not only weaker than healthy individuals in the knee extensors, but they also have impaired neuromuscular recovery after fatigue. Muscle Nerve 46: 434–439, 2012

Introduction: In this study we tested the hypothe- sis that caffeine supplementation improves neuromuscular func- tion, which has both nutritional and clinical relevance. Methods: Fourteen male subjects (mean 6 SD: 23.8 6 2.8 years) volun- teered in a double-blind, repeated-measures study with placebo (PLA) or caffeine (CAFF) (6 mg kg � 1 ). Maximal voluntary iso- metric contractions (MVCs), evoked maximal twitch, and maxi- mal isokinetic contractions during elbow flexion were assessed. Mechanical and electromyographic (EMG) signals from the biceps brachii muscle were recorded, and muscle fiber conduc- tion velocity (CV) was calculated to evaluate changes in the muscle force-velocity relationship and muscle fiber recruitment. Results: The torque-angular velocity curve was enhanced after CAFF supplementation. This was supported by a concomitant increase of CV values (8.7% higher in CAFF). Conclusions: Caffeine improves muscle performance during short-duration maximal dynamic contractions. The concomitant improvement of mean fiber CV supports the hypothesis of an effect of caf- feine on motor unit recruitment. Muscle Nerve 000: 000-000, 2011

Differentiating between respiratory frequency (fR) and tidal volume (V T) may improve our understanding of exercise hyperpnoea because fR and V T seem to be regulated by different inputs. We designed a series of exercise manipulations to improve our understanding of how fR and V T are regulated during exercise. Twelve cyclists performed an incremental test and three randomized experimental sessions in separate visits. In two of the three experimental visits, participants performed a moderate‐intensity sinusoidal test followed, after recovery, by a moderate‐to‐severe‐intensity sinusoidal test. These two visits differed in the period of the sinusoid (2 min vs. 8 min). In the third experimental visit, participants performed a trapezoidal test where the workload was self‐paced in order to match a predefined trapezoidal template of rating of perceived exertion (RPE). The results collectively reveal that fR changes more with RPE than with workload, gas exchange, V T or the amount of muscle activation. However, fR dissociates from RPE during moderate exercise. Both V T and minute ventilation (V˙E) showed a similar time course and a large correlation with V˙CO2in all the tests. Nevertheless, V˙CO2 was associated more with V˙E than with V T because V T seems to adjust continuously on the basis of fR levels to match V˙E with V˙CO2. The present findings provide novel insight into the differential control of fR and V T – and their unbalanced interdependence – during exercise. The emerging conceptual framework is expected to guide future research on the mechanisms underlying the long‐debated issue of exercise hyperpnoea.

BAZZUCCHI, I., F. FELICI, and M. SACCHETTI. Effect of Short-Term Creatine Supplementation on Neuromuscular Function. Med. Sci. Sports Exerc., Vol. 41, No. 10, pp. 1934–1941, 2009. Purpose: The purpose of the present investigation was to determine whether short-term creatine (Cr) supplementation would affect 1) muscle contractile properties assessed by evoked and voluntary contractions, 2) force–velocity relationship, and 3) mean muscle fiber conduction velocity (CV). Methods: Using a double-blind random design, 16 moderately trained men (25.2 T 5.1 yr) were assigned to a Cr (CRE) or a placebo (PLA) group. Subjects supplemented their diet four times a day for 5 d with 5 g of Cr + 15 g maltodextrin (CRE) or 20 g maltodextrin (PLA). Isometric maximal voluntary contraction, maximal twitch, force–velocity relationship, and dynamic fatiguing contractions were assessed in the elbow flexors. Mechanical and EMG signals were recorded and analyzed. CV was estimated from the EMG and used as a parameter of interest. Results: After supplementation, peak torque (PT) of maximal twitch was 33.4% higher, and the time to reach the PT was 54.7% lower in CRE than in PLA (P G 0.05). Torque–angular velocity curve was enhanced after Cr supplementation, especially at the higher velocities. Mean fiber CV was, on average, 8.9% higher in CRE at all angular velocities after supplementation (P G 0.05). EMG and mechanical parameters during the fatiguing exercise protocol did not show significant differences in muscle fatigue between the two groups after supplementation. Conclusions: The present study shows that oral Cr supplementation improves neuromuscular

Previous studies have suggested that muscle coactivation could be reduced by a recurrent activity (training, daily activities). If this was correct, skilled athletes should show a specific muscle activation pattern with a low level of coactivation of muscles which are typically involved in their discipline. In particular, the aim of this study was to verify the hypothesis that the amount of antagonist activation of biceps brachii (BB) and triceps brachii (TB) is different between tennis players and non-players individuals during maximal isokinetic contractions.Ten young healthy men and eight male tennis players participated in the study. The surface electromyographic signals (sEMG) were recorded from the BB and TB muscles during three maximal voluntary isometric contractions (MVC) of elbow flexors and extensors and a set of three maximal elbow flexions and extensions at 15 degrees , 30 degrees , 60 degrees , 120 degrees , 180 degrees and 240 degrees /s. Normalized root mean square (RMS) of sEMG was calculated as an index of sEMG amplitude.Antagonist activation (%RMSmax) of TB was significantly lower in tennis players (from 14.0+/-7.9% at MVC to 16.3+/-8.9% at 240 degrees /s) with respect to non-players (from 27.7+/-19.7% at MVC to 38.7+/-17.6% at 240 degrees /s) at all angular velocities. Contrary to non-players, tennis players did not show any difference in antagonist activation between BB and TB muscles.Tennis players, with a constant practice in controlling forces around the elbow joint, learn how to reduce coactivation of muscles involved in the control of this joint. This has been shown by the lower antagonist muscular activity of triceps brachii muscle during isokinetic elbow flexion found in tennis players with respect to non-players.

Purpose: The effects of exercise training on neuromuscular function of arm and leg muscles in type 2 diabetic patients (T2D) was investigated. \ud \ud Methods: Eight T2D sedentary male patients (61.0 ± 2.3 years) and eight sedentary healthy age matched control subjects (H, 63.9 ± 3.8 years) underwent a 16-week supervised combined endurance and resistance exercise program. Before and after training, maximal isometric (MVIC), isokinetic (15, 30, 60, 120, 180, 240° s−1) torque and muscle endurance of the elbow flexors (EF) and knee extensors (KE) were assessed. Simultaneously, surface electromyographic signals from biceps brachii (BB) and vastus lateralis (VL) muscles were recorded and muscle fiber conduction velocity (MFCV) estimated. \ud \ud Results: Following training, maximal torque of the KE increased during MVIC and isokinetic contractions at 15 and 30° s−1 in the T2D (+19.1 ± 2.7% on average; p < 0.05) but not in the H group (+7 ± 0.9%; p > 0.05). MFCV recorded from the VL during MVIC and during isokinetic contractions at 15 and 30° s−1 increased (+11.2 ± 1.6% on average; p < 0.01), but in the diabetic group only. Muscular endurance was lower in T2D (20.1 ± 0.7 s) compared to H (26.9 ± 1.3 s), with an associated increase in the MFCV slope after training in the KE muscles only. \ud \ud Conclusion: The effect of a combined exercise training on muscle torque appears to be angular velocity-specific in diabetic individuals, with a more pronounced effect on KE muscles and at slow contraction velocities, along with an associated increase in the MFCV. MFCV appears to be a more sensitive marker than torque in detecting the early signs of neuromuscular function reconditioning.

In the present study the influence of speed of contraction on the interplay between recruitment and firing rate of motor units (MUs) was assessed. The surface electromyographic (sEMG) signal was recorded in nine healthy subjects from the right biceps brachii using a linear electrode array during ramp isometric contractions (from 0 to 100% of the maximal voluntary force, MVC) at 5, 10, and 20% MVC s(-1) (ramp phase), followed by 10 s of sustained MVC (hold phase). The median frequency (MDF), Root Mean Square (RMS) and conduction velocity (CV) of sEMG, were computed on adjacent epochs covering a force range of 5% MVC each. Full motor unit recruitment (FMUR) point was assessed as the force level at which MDF reached its maximum value; the MDF decay during the hold phase was taken as an index of localized muscle fatigue. At 5% MVC s(-1), FMUR was reached at 52.3% MVC. At 10%MVC s(-1) FMUR was achieved at 58% MVC; while at 20% MVC s(-1) FMUR point was located at 77% MVC, being statistically different from 5 and 10% MVCs(-1) ramps (p

Quercetin (Q) functions as antioxidant in vitro, but its effect have been minimally examined in combination with exercise in humans. The purpose of this investigation was to determine the effects of a diet supplemented with 1 g per day of Q for 2 weeks on the erythrocytes oxidative balance before and after an acute bout of eccentric exercise (EE). Fourteen volunteer males were randomly assigned, in a double-blind crossover design, to a placebo or experimental supplemented groups. Blood samples were taken before and after 2 weeks of supplementation under resting and post-exercise conditions. Erythrocytes glutathione (GSH, GSSG, GSH/GSSG), malonaldehyde (TBARs), enzymes antioxidant activities as well as time of hemolysis were evaluated in ex vivo. Quercertin per se did not affect redox homeostasis but increased the time of hemolysis and decreased TBARs levels. Following the EE the Q group displayed a higher GSH/GSSG ratio and a less pronounced increase in TBARs, compared to placebo group. Moreover, we found that GPx enzyme activity were induced after EE only in Q group, while any significant modification of this parameter was detected in placebo group. In conclusion, the Q supplementation may be used as a countermeasure against oxidative stress inducing, in erythrocytes, a cellular adaptation allowing subjects to better cope with the oxidative stress induced by an acute exercise.

The present study proposes an alternative way of comparing performance and acute physiological responses to continuous exercise with those of intermittent exercise, ensuring similar between-protocol overall effort (isoeffort) and the same total duration of exercise (isotime). This approach was expected to overcome some drawbacks of traditional methods of comparison. Fourteen competitive cyclists (20±3 yrs) performed a preliminary incremental test and four experimental 30-min self-paced protocols, i.e. one continuous and three passive-recovery intermittent exercise protocols with different work-to-rest ratios (2 = 40∶20s, 1 = 30∶30s and 0.5 = 20∶40s). A “maximal session effort” prescription was adopted for this experimental design. As expected, a robust perceived exertion template was observed irrespective of exercise protocol. Similar between-protocol pacing strategies further support the use of the proposed approach in competitive cyclists. Total work, oxygen uptake and heart rate mean values were significantly higher (P

The aim of this study was to evaluate the influence of exposure to altitude on muscle endurance during isometric contractions. Six sedentary subjects were studied. Surface electromyograph (sEMG) activity was recorded from the right biceps brachii (BB) during exhausting isometric exercise at 80% maximal voluntary contraction. Experiments were performed before, during and 6 months after a 12 day stay at the EV-K2 laboratory (Nepal, 5,050 m above sea level). From the sEMG signals from BB, the median frequencies (fmed) were computed for consecutive 1 s epochs. The sEMG was also analysed using a non-linear tool, the recurrence quantification analysis, and the percentage of determinism (%DET) was then calculated. The haemoglobin saturation significantly decreased at altitude. The mean (SD) BB endurance time decreased from 22.4 (4) s to 18.3 (4.7) s (P < 0.05). After exposure to altitude a significant variation in fmed and %DET slopes was observed. We concluded that during the first period of acclimatisation at altitude there was an impairment of isometric muscle endurance performance and there was also evidence of a modified myoelectric activity pattern suggesting a greater fatigability of the neuromuscular system.

Background Patients with type 2 diabetes (T2D) may be subject to premature muscle fatigue. However, the effect of diabetes on muscle fatigability has not yet been thoroughly examined. The purpose of this study was to investigate the effect of T2D on muscle fatigability at the upper and lower body. Methods Thirty-three T2D patients (18 men and 15 women; mean age, 59.3 ± 5.3 years) and 34 matched healthy control participants (17 men and 17 women; mean age, 60.1 ± 6.1 years) were recruited. Clinical characteristics of diabetic patients were assessed by considering a wide range of vascular and neurological parameters in order to exclude the presence of micro- and macro-vascular complications. Gender-specific muscle function was evaluated measuring the maximal voluntary isometric contraction (MVIC), and the endurance time at 50% of the MVIC at the shoulder and at the knee extensor muscles. Results Muscle strength in the upper body was similar among groups, whereas in the lower body, it was significantly reduced in T2D men (-16%) and women (-22%) compared with the controls. Additionally, the endurance time in both upper and lower body was significantly lower in T2D men (-18% and -29%) and women (-19% and -25%, respectively) than controls. Conclusions Besides the reduction in strength, muscle dysfunction in T2D is characterized by a higher fatigability that affects both upper and lower body muscles. This effect is independent to the presence of diabetic complications and may represent a more sensitive marker of muscular dysfunction than muscle strength. Copyright © 2016 John Wiley & Sons, Ltd.

The aim of this study was to compare muscle activation and cardio-respiratory response during ergometer and on-water rowing. Nine internationally competitive rowers (five Olympic Games medal winners, age 25.6 ± 4.8 years) were requested to perform a 1,000 m race simulation test in the two conditions. Surface electromyographic (sEMG) signals from trapezius superior (TRS), latissimus dorsi (LD), biceps brachii (BB), rectus femoris (RF), vastus medialis (VAM), vastus lateralis (VAL), biceps femoris (BF) and tibialis anterior (TA) muscles were recorded continuously during the tests together with other cardio-respiratory parameters: heart rate (HR), ventilation (VE), oxygen consumption (VO₂). On-water, subjects covered the same distance in a longer time (218.4 ± 3.8 s vs. 178.1 ± 5.6 s during ergometer test). TRS, LD, BB, RF, VAM and VAL muscle activation on-water was lower than off-water during the rowing race. VO₂ and VE responses were similar between the two conditions even if the time to complete the 1,000 m race simulation test was higher on-water. The results indicate that for most of the analyzed muscles EMG activation on the ergometer is higher than on-water with the maximal activity at the beginning of the on-water test due reasonably to overcome the forces opposing the forward motion, while the ergometer task elicited increasing muscle activation over time. The present data may be considered by coaches when choosing a rowing ergometer in substitution for the training on-water or when relying on the indoor tests to select the crew.