1,029 results on '"Kaina, Bernd"'
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2. PARP-1 selectively impairsKRAS-driven phenotypic and molecular features in intrahepatic cholangiocarcinoma
3. Auswirkungen von DNA‐Reparaturfaktoren auf die Schädigung von Darmzellen und Entzündungen, ausgelöst durch diätisches Häm‐Eisen
4. Genotoxic and Cytotoxic Activity of Fisetin on Glioblastoma Cells
5. Dose response to methylating agents in the γH2AX, SCE and colony formation assays: Effect of MGMT and MPG overexpression
6. Do Carcinogens Have a Threshold Dose? The Pros and Cons
7. Artesunate in glioblastoma therapy: Case reports and review of clinical studies
8. From cultivation to cancer: formation of N -nitrosamines and other carcinogens in smokeless tobacco and their mutagenic implications
9. Chronic intestinal inflammation drives colorectal tumor formation triggered by dietary heme iron in vivo
10. Genotoxic and cytotoxic potential of methacrylate-based orthodontic adhesives
11. Methadone-mediated sensitization of glioblastoma cells is drug and cell line dependent
12. Comparison of DNA repair and radiosensitivity of different blood cell populations
13. Strong apoptotic response of testis tumor cells following cisplatin treatment
14. Temozolomide, Procarbazine and Nitrosoureas in the Therapy of Malignant Gliomas: Update of Mechanisms, Drug Resistance and Therapeutic Implications.
15. Positive effects of PARP-1 inhibition in KRAS-mutated intrahepatic cholangiocarcinoma is mediated by CHK1 kinase
16. PARP-1 protects against colorectal tumor induction, but promotes inflammation-driven colorectal tumor progression
17. DNA Damage-Induced Apoptosis
18. Immunological and mass spectrometry-based approaches to determine thresholds of the mutagenic DNA adduct O6-methylguanine in vivo
19. A genome-wide screening for DNA repair genes: much more players than hitherto known
20. The impact of the IGF-1 system of cancer cells on radiation response – An in vitro study
21. Chloroethylating nitrosoureas in cancer therapy: DNA damage, repair and cell death signaling
22. Impact of DNA repair on the dose-response of colorectal cancer formation induced by dietary carcinogens
23. Supplementary Data, Figure S1 - S3 from The SIAH1–HIPK2–p53ser46 Damage Response Pathway is Involved in Temozolomide-Induced Glioblastoma Cell Death
24. Data from Targeting Homologous Recombination by Pharmacological Inhibitors Enhances the Killing Response of Glioblastoma Cells Treated with Alkylating Drugs
25. Data from The SIAH1–HIPK2–p53ser46 Damage Response Pathway is Involved in Temozolomide-Induced Glioblastoma Cell Death
26. Supplementary Table 3 from Contribution of ATM and ATR to the Resistance of Glioblastoma and Malignant Melanoma Cells to the Methylating Anticancer Drug Temozolomide
27. Supplementary Figure S3 from Targeting Homologous Recombination by Pharmacological Inhibitors Enhances the Killing Response of Glioblastoma Cells Treated with Alkylating Drugs
28. Supplementary Table 1 from Contribution of ATM and ATR to the Resistance of Glioblastoma and Malignant Melanoma Cells to the Methylating Anticancer Drug Temozolomide
29. Supplementary Table 2 from Contribution of ATM and ATR to the Resistance of Glioblastoma and Malignant Melanoma Cells to the Methylating Anticancer Drug Temozolomide
30. Data from Contribution of ATM and ATR to the Resistance of Glioblastoma and Malignant Melanoma Cells to the Methylating Anticancer Drug Temozolomide
31. Supplementary Table1+2 from Temozolomide Induces Senescence and Repression of DNA Repair Pathways in Glioblastoma Cells via Activation of ATR–CHK1, p21, and NF-κB
32. Supplementary Figure5 from Temozolomide Induces Senescence and Repression of DNA Repair Pathways in Glioblastoma Cells via Activation of ATR–CHK1, p21, and NF-κB
33. Supplementary Data from Extent and Patterns of MGMT Promoter Methylation in Glioblastoma- and Respective Glioblastoma-Derived Spheres
34. Supplementary figure legends from Enhanced Histone Deacetylase Activity in Malignant Melanoma Provokes RAD51 and FANCD2-Triggered Drug Resistance
35. Supplementary Table 1, Figures 1 - 3 from Translesion Polymerase η Is Upregulated by Cancer Therapeutics and Confers Anticancer Drug Resistance
36. Supplementary Table S1 from Enhanced Histone Deacetylase Activity in Malignant Melanoma Provokes RAD51 and FANCD2-Triggered Drug Resistance
37. Supplementary Figure Legend from Translesion Polymerase η Is Upregulated by Cancer Therapeutics and Confers Anticancer Drug Resistance
38. Supplementary Figure S3 from Enhanced Histone Deacetylase Activity in Malignant Melanoma Provokes RAD51 and FANCD2-Triggered Drug Resistance
39. Data from Cisplatin-Induced Apoptosis Involves Membrane Fluidification via Inhibition of NHE1 in Human Colon Cancer Cells
40. Data from Translesion Polymerase η Is Upregulated by Cancer Therapeutics and Confers Anticancer Drug Resistance
41. Supplementary Materials from Cisplatin-Induced Apoptosis Involves Membrane Fluidification via Inhibition of NHE1 in Human Colon Cancer Cells
42. Human primary endothelial cells are impaired in nucleotide excision repair and sensitive to benzo[a]pyrene compared with smooth muscle cells and pericytes
43. Senescence and death induced by radiation and alkylating drugs in cancer therapy
44. Preferential effect of PARP-1 inhibition in KRAS-mutated intrahepatic cholangiocarcinomas is mediated by CHK1/2 kinases
45. Do Carcinogens Have a Threshold Dose? Pro and Contra
46. Sensitivity of CD3/CD28-stimulated versus non-stimulated lymphocytes to ionizing radiation and genotoxic anticancer drugs: key role of ATM in the differential radiation response
47. Abrogation of Cellular Senescence Induced by Temozolomide in Glioblastoma Cells: Search for Senolytics
48. Analysis of the interplay between all-trans retinoic acid and histone deacetylase inhibitors in leukemic cells
49. The Brassica-derived phytochemical indolo[3,2-b]carbazole protects against oxidative DNA damage by aryl hydrocarbon receptor activation
50. Biodosimetry Based on γ-H2AX Quantification and Cytogenetics after Partial- and Total-Body Irradiation during Fractionated Radiotherapy
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