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3. Pseudomonas aeruginosa utilizes host polyunsaturated phosphatidylethanolamines to trigger theft-ferroptosis in bronchial epithelium.

6. PEBP1 Wardens Ferroptosis by Enabling Lipoxygenase Generation of Lipid Death Signals

7. Phospholipase iPLA2β averts ferroptosis by eliminating a redox lipid death signal

8. Redox lipid reprogramming commands susceptibility of macrophages and microglia to ferroptotic death

10. Phosphorylation of Cytochrome c Threonine 28 Regulates Electron Transport Chain Activity in Kidney: IMPLICATIONS FOR AMP KINASE

18. Disruption of the M80-Fe Ligation Stimulates the Translocation of Cytochrome c to the Cytoplasm and Nucleus in Nonapoptotic Cells

22. Peroxidase activity and structural transitions of the cytochrome c bound to cardiolipin-containing membranes

27. Translocation of Cytochrome C to the Cytoplasm and Nucleus in Non-Apoptotic Cells: 57

29. Serine‐47 phosphorylation of cytochrome c in the mammalian brain regulates cytochrome c oxidase and caspase‐3 activity

30. Redox (phospho)lipidomics of signaling in inflammation and programmed cell death

31. Oxidized Arachidonic/Adrenic Phosphatidylethanolamines Navigate Cells to Ferroptosis

33. Lipid bodies containing oxidatively truncated lipids block antigen cross-presentation by dendritic cells in cancer

34. Tocopherols and tocotrienols prevent lipoxygenase‐driven phospholipid oxidation in ferroptosis

36. Oxidized arachidonic and adrenic PEs navigate cells to ferroptosis

37. Cytochrome c is phosphorylated by AMP kinase in mammalian kidney, leading to controlled electron transport chain flux

38. Impaired Clearance and Enhanced Pulmonary Inflammatory/Fibrotic Response to Carbon Nanotubes in Myeloperoxidase-Deficient Mice

39. Mitochondrial NM23-H4/NDPK-D Supports Cardiolipin Signaling to Eliminate Depolarized Mitochondria by Mitophagy

40. Mitochondrial Redox Opto-Lipidomics Reveals Mono-Oxygenated Cardiolipins as Pro-Apoptotic Death Signals

41. Peroxidase Mechanism of Lipid-dependent Cross-linking of Synuclein with Cytochrome c: PROTECTION AGAINST APOPTOSIS VERSUS DELAYED OXIDATIVE STRESS IN PARKINSON DISEASE*

45. Lung Macrophages “Digest” Carbon Nanotubes Using a Superoxide/Peroxynitrite Oxidative Pathway

46. Impaired Clearance And Enhanced Pulmonary Inflammatory/Fibrotic Response To Carbon Nanotubes In Myeloperoxidase-Deficient Mice

47. The hydrogen-peroxide-induced radical behaviour in human cytochrome c–phospholipid complexes: implications for the enhanced pro-apoptotic activity of the G41S mutant

48. Cardiolipin externalization to the outer mitochondrial membrane acts as an elimination signal for mitophagy in neuronal cells

49. Carbon Nanotubes: Biodegradation of Single-Walled Carbon Nanotubes by Eosinophil Peroxidase (Small 16/2013)

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