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2. Single-cell multiomic analysis identifies a HOX-PBX gene network regulating the survival of lymphangioleiomyomatosis cells

3. Data from The Codon 72 TP53 Polymorphism Contributes to TSC Tumorigenesis through the Notch–Nodal Axis

4. Supplementary Figure, Figure Legends, Tables and Material and Methods from The Codon 72 TP53 Polymorphism Contributes to TSC Tumorigenesis through the Notch–Nodal Axis

5. Data from Complement C5a Receptor Facilitates Cancer Metastasis by Altering T-Cell Responses in the Metastatic Niche

6. Supplementary Materials and Methods from Complement C5a Receptor Facilitates Cancer Metastasis by Altering T-Cell Responses in the Metastatic Niche

7. Supplementary Figure Legends from Complement C5a Receptor Facilitates Cancer Metastasis by Altering T-Cell Responses in the Metastatic Niche

8. Supplementary Figures 1 - 5 from Complement C5a Receptor Facilitates Cancer Metastasis by Altering T-Cell Responses in the Metastatic Niche

9. Upregulation of acid ceramidase contributes to tumor progression in Tuberous Sclerosis Complex

11. Integrative Single Cell Multiomic Profiling Analysis Reveals HOX-PBX Gene Regulatory Network Contributing to the Survival of mTOR Hyperactive Cells

15. The evolutionarily conserved TSC/Rheb pathway activates Notch in tuberous sclerosis complex and Drosophila external sensory organ development

17. Complement as Prognostic Biomarker and Potential Therapeutic Target in Renal Cell Carcinoma

20. Therapeutic Targeting of Vasculature in the Premetastatic and Metastatic Niches Reduces Lung Metastasis

23. The Codon 72 TP53 Polymorphism Contributes to TSC Tumorigenesis through the Notch–Nodal Axis

27. Notch transactivates Rheb to maintain the multipotency of TSC-null cells

32. Complement C5a Receptor Facilitates Cancer Metastasis by Altering T-Cell Responses in the Metastatic Niche

33. The complement C5a receptor facilitates cancer metastasis by altering T cell responses in metastases-targeted organs (TUM2P.883)

40. Tumorigenesis in tuberous sclerosis complex is autophagy and p62/sequestosome 1 (SQSTM 1)-dependent.

41. Constitutive mTOR activation in TSC mutants sensitizes cells to energy starvation and genomic damage via p53.

42. Diet and exercise as potential anticonvulsant therapies in mouse seizure models

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