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KANYE WEST is not from the hood. The son of an English professor at Chicago State University, he struggled to be taken seriously as a rapper until he realized that [...]

Target Tehran By YONAH JEREMY BOB AND ILAN EVYATAR Simon & Schuster, 368 pages I FINISHED READING Target Tehran, an excellent new book on the Mossad's shadow war to keep [...]

Boomers: The Men and Women Who Promised Freedom and Delivered Disaster BY HELEN ANDREWS Penguin Publishing Group, 256 pages THE baby boomers are a popular target, but Helen Andrews goes [...]

BYMY SECOND WEEK at Stanford, I knew only three things: I was unable to drink alcohol responsibly, I didn't really like my new friends, and supporting Israel was going to [...]

The article offers a perspective on the history and radical potential of the anti-semitism of billionaire rapper Kanye West. It looks into West's accusation of the Jewish villain as the local landlord or shopkeeper and argues that Jews were made as scapegoat for the failure of African Americans to adopt solutions to their problems. It contends that focusing on Jews allows West to make African American grievances coterminous with white grievances.

Letters to My Palestinian Neighbor BY YossI KLEIN HALEVI Harper, 224 pages THE following passage appears at the outset of Yossi Klein Halevi's new book: 'The muezzin's call to prayer [...]

Free Speech on Campus BY ERWIN CHEMERINSKY AND HOWARD GILLMAN Yale University Press, 197 pages 'OUR POSITION is absolute,' declare Erwin Chemerinsky and Howard Gillman in the beginning of their [...]

Abstract: A mutant cell line, called don801, was isolated from a wild-type population of V79.5 Chinese hamster cells by its ability to grow in the presence of the glutamine analog 6-diazo-5-oxo-l-norleucine (DON), which is toxic for V79.5 cells. The don801 cells were found not to be cross-resistant to another glutamine analog, O-diazoacetyl-l-serine (azaserine, AS). It was shown that guanine but neither hypoxanthine nor adenine protected V79.5 cells from the toxic effects of DON, while hypoxanthine and adenine, but not guanine protected them against AS toxicity. Exposure of wild-type cells to DON was shown to result in a specific reduction of intracellular GTP pools, while in the mutant cells there was no effect on GTP levels. These results strongly suggested that DON was specifically inhibiting guanylate synthetase (GMP synthetase;xanthosine-5prime-phosphate:l-glutamine amidoligase, EC 6.3.5.2) in V79.5 cells and that the enzyme in don801 cells was resistant to inhibition. In vitro assays of GMP synthetase activities from V79.5 and don801 cells confirmed this hypothesis. The mutant phenotype was also found to be dominant in intraspecific cell hybrids.