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8. Poster presentations

Author

Aksu, Funda, Topacoglu, Hakan, Arman, Candan, Atac, Aytul, Tetik, Suleyman, Hasanovic, Aida, Kulenovic, Amela, Mornjakovic, Zakira, Pikula, Branko, Sarac-Hadzihalilovic, Aida, Voljevica, Alma, Bamac, Belgin, Colak, Tuncay, Alemdar, Murat, Dundar, Gulmine, Selekler, Macit, Dincer, Ozgur, Colak, Enis, Ozbek, Aydin, Kilic, Cenk, Kamburoglu, Kivanc, Ozen, Tuncer, Kavak, Vatan, Kirici, Yalcin, Oztas, Emin, Soysal, Handan Altinkaya, Unur, Erdogan, Ekinci, Nihat, Karaca, Omur, Malakhova, Olga, Kocaoglu, Murat, Toker, Serdar, Taser, Figen, Kilincoglu, Volkan, Yurtgun, Mustafa Fahri, Dalcik, Cannur, Zeybek, Ali, Baroncini, Marc, Peltier, Johann, Jissendi, Patrice, Pruvo, Jean-Pierre, Francke, Jean-Paul, Prevot, Vincent, Kosif, Rengin, Arifoglu, Yasin, Diramali, Murat, Sarsilmaz, Mustafa, Kose, Evren, Ogeturk, Murat, Akpinar, Burhan, Kus, Ilter, Meydan, Sedat, Kara, Alev, Kurtoglu, Zeliha, Tekdemir, Ibrahim, Elhan, Alaittin, Bas, Orhan, Odaci, Ersan, Mollaoglu, Hakan, Ucok, Kagan, Kaplan, Suleyman, Senoglu, Mehmet, Nacitarhan, Vedat, Kurutas, Ergul Belge, Senoglu, Nimet, Altun, Idris, Atli, Yalcin, Ozbag, Davut, Karakas, Sacide, Bilgin, M. Dincer, Tellioglu, Ayfer Metin, Ozlem, Sercin, Akcanal, Betul, Yildiz, Yuksel, Gunes, Hakki, Kose, Hayrullah, Uzum, Ibrahim, Gundogmus, Umit Naci, Caglayan, Cigdem, Pavlova, Velichka, Dimitrova, Mashenka, Georgieva, Lilia, Nikolova, Elena, Uzmansel, Deniz, Ozturk, Nail Can, Saylam, Canan Yurttas, Ozgiray, Erkin, Orhan, Mustafa, Cagli, Sedat, Zileli, Mehmet, Ozkan, Derya, Akkaya, Taylan, Comert, Ayhan, Balikci, Nilgun, Ozdemir, Esra, Gumus, Haluk, Ergul, Zafer, Kaya, Oskay, Altun, Serdar, Unlu, R. Erkin, Orbay, Hakan, Kim, Deog-Im, Han, Seung-Ho, Kim, Yi-Suk, Kim, Ho-Jeong, Lee, Kyu-Seok, Elcioglu, Omur, Ozden, Hilmi, Guven, Gul, Imre, Nurcan, Yalcin, Bulent, Ozan, Hasan, Akyer, Pinar, Guvencer, Mustafa, Karatosun, Vasfi, Sagoo, Mandeep Gill, Aland, Rachel Claire, Ustuner, Derya, Ustuner, M. Cengiz, Ai, Jafar, Ghazi, Seyed Reza, Mansouri, Seyed Hadi, Tuncer, Mehmet Cudi, Aluclu, Mehmet Ufuk, Karabulut, Ozlen, Hatipoglu, Eyup Savas, Nazaroglu, Hasan, Icke, Cigdem, Akbay, Emrah, Gunay, Turkan, Icke, Suleyman, Yildiz, Selda, Yazar, Fatih, Barlas, Barcin Orhan, Zahoi, Delia Elena, Kavakli, Ahmet, Tas, Ufuk, Dabak, Durrin Ozlem, Sapmaz, Hilal Irmak, Kocabiyik, Necdet, Ozer, Cenk Murat, Ozcan, Ayhan, Elevli, Levent, Desdicioglu, Kadir, Alanbay, Ibrahim, Govsa, Figen, Saylam, Canan Y., Akdogan, Ilgaz, Kiroglu, Yilmaz, Onur, Sule, Evcil, Emine Hilal, Cankara, Neslihan, Malas, Mehmet Ali, Kalcioglu, M. Tayyar, Duman, Serdar, Ulcay, Tufan, Uzun, Ahmet, Karabulut, Zulfu, Barut, Cagatay, Sevinc, Ozdemir, Yurdakan, Gamze, Kacar, Dundar, Erdogan, Ali Riza, Kurt, Hulyam, Demir, Bunyamin, Saltan, Mustafa, Burukoglu, Dilek, Ustuner, Mehmet Cengiz, Degirmenci, Irfan, Erdogan, Aliriza, Damar, Ozlem, Is, Merih, Bayramoglu, Gokhan, Kabay, Sahin, Uysal, Onur, Senturk, Hakan, Bayramoglu, Aysegul, Ozbayar, Cansu, Kutlu, Ali, Canbek, Mediha, Cevli, Salih Cenap, Hancerlioglu, Oguz, Koplay, Mustafa, Aksakalli, Elif, Dikici, Fatih, Kale, Aysin, Gayretli, Ozcan, Gurses, Ilke Ali, Ozdemir, Senem Turan, Ercan, Ilker, Baskan, Emel Bulbul, Yilmaz, Mediha, Ozkaya, Guven, Saricaoglu, Hayriye, Erturk, Mete, Kayalioglu, Gulgun, Uzel, Mehmet, Kahraman, Guler, Tanyeli, Ercan, Soyluoglu, Ali Ihsan, Tacar, Orhan, Demirant, Ayda, Bilgin, Murat, Karadede, Aziz, Aktas, Ayfer, Evcil, E. Hilal, Koyuncu, Esra, Sulak, Osman, Albay, Soner, Ozguner, Gulnur, Ozbek, Ahmet, Ozbek, Elvan, Ozturk, A. Hakan, Demirci, Tuba, Ciftcioglu, Engin, Demir, Mehmet Tevfik, Kopuz, Cem, Eroglu, Esra, Gedikli, Semin, Ozyurek, Hamit, Nural, Mehmet Selim, Incesu, Lutfi, Ogur, Gonul, Kara, Engin, Celebi, Baris, Yildiz, Altan, Altunkaynak, B. Zuhal, Kuvat, Samet Vasfi, Tagil, Suleyman Murat, Ertekin, Cumhur, Uysal, Hilmi, Bademkiran, Fikret, Albayrak, Nural, Esmer, Ali Firat, Coskun, Nigar Keles, Sindel, Muzaffer, Kizilay, Ferah, Yalin, Sevket, Karapinar, Nevin, Tokdemir, Mehmet, Karakurt, Lokman, Tumkaya, Levent, Korkmaz, Adnan, Ayas, Bulent, Ciftci, Nusret, Terzi, Yuksel, Baran, Ozlem, Nergiz, Yusuf, Akkus, Murat, Aluclu, Ufuk, Topal, Askin Ender, Yuksel, Dilek, Acar, Halil Ibrahim, Kendir, Simel, Hekimoglu, Emre, Basman, Deniz, Duman, Sunay, Ozener, Baris, Pelin, Can, Zagyapan, Ragiba, Kurkcuoglu, Ayla, Koc, Mustafa, Erdinc, Meral, Erdinc, Levent, Kelle, Ilker, Sancakdar, Enver, Cetin, Nil, Tunik, Selcuk, Yildirim, Ayse, Kaplanoglu, Iskender, Ayaz, Ercan, Ilhan, Necip, Okumus, Mehmet, Yuksel, Kasim Zafer, Ciralik, Harun, Yilmaz, Zeki, Gumusalan, Yakup, Gamsizkan, Mehmet, Kazkayasi, Mustafa, Dogan, Nadire Unver, Uysal, Ismihan Ilknur, Karalezli, Aylin, Fazliogullari, Zeliha, Buyukmumcu, Mustafa, Bozkurt, Mehmet Cem, Cicekcibasi, Aynur Emine, Demiryurek, Deniz, Ozsoy, M. Hakan, Bayramoglu, Alp, Tuccar, Eray, Baran, Ozlem Pamukcu, Soker, Sevda, Bahceci, Selen, Nasir, Yasemin, Yilmaz, Mehmet Tugrul, Cicekcibasi, Emine Aynur, Ulusoy, Mahinur, Gunaslan, Pervin, Bilge, Nuray, Akkaya, Muzaffer, Genc, Abdurrahman, Akcer, Sezer, Gonul, Yucel, Cosar, Emine, Koken, Gulengul, Ari, Ilknur, Bakirci, Sinan, Kafa, Ilker Mustafa, Uysal, Murat, Karabulut, Ahmet Kagan, Keles, Bahar, Emlik, Dilek, Uyar, Yavuz, Ozturk, Kayhan, Yilmaz, Neslihan Altuntas, Salbacak, Ahmet, Kacira, Burkay Kutluhan, Arazi, Mehmet, Demirci, Serafettin, Kiresi, Demet, Gumus, Serter, Seker, Muzaffer, Uyar, Mehmet, Astaneh, Mohammad Ebrahim, Khorshid, Alireza, Uygur, Ramazan, Songur, Ahmet, Sonmez, Osman Fikret, Dogan, Kamil Hakan, Kolcu, Giray, Iliescu, Madalina, Bordei, Petru, Iliescu, Dan, Ciobotaru, Camelia, Lucescu, Viorel, Covaleov, Anatoli, Ionescu, Constantin, Guirao, Miguel, Páramo, E., Mutuberria, R., Sánchez-Montesinos, I., Roda, O., Girón, F., Lopez-Soler, Miguel, Roda, Olga, Campos-López, Raúl, Guirao-Piñeiro, Miguel, Pascual-Morenilla, Maria Teresa, Sanchez-Montesinos, Indalecio, Pascual, Maria Teresa, Garzon, I., Serrato, D., Nieto-Aguilar, R., Sanchez-Montesinos, I., Sanchez-Quevedo, M., Ozdemir, M. Bulent, Ozean, R. Hakan, Bagdatli, Dilek, Adiguzel, Esat, Dogan, Zumrut, Aycan, Ozlem, Vardi, Nigar, Erkal, Haldun Sukru, Ozturk, Hakan, Mocanu, S., Stefanescu, C., Ionescu, A., Talpes, Raluca, Sapte, Elena, Dina, Constantin, Surdu, Loredana, Bulbuc, Ionut, Medina, M. T., Medina, J., López-Soler, M., Martin-Oviedo, Carlos, Lowy-Benoliel, Alejandro, Maranillo, Eva, Martinez-Guirado, Tomas, Sañudo, Jose, Scola, Bartolome, Vazquez, Teresa, Arráez-Aybar, L. A., Conejo-Menor, J. L., Gonzáles-Gómez, C. C., Torres-García, A. J., Nasu, Hisayo, Chiba, Shoji, Gutierrez-Semillera, M., Paksoy, Yahya, Kalaycioglu, Ahmet, Yildirim, Mehmet, Ozyasar, Ali, Ozdogmus, Omer, Cakmak, Yusuf Ozgur, Verimli, Ural, Cavdar, Safiye, Yildizhan, Begum, Aktan Ikiz, Z. Asli, Ucerler, Hulya, Ozgur, Zuhal, Yilmaz, Seher, Demirtas, Abdullah, Mavili, Ertugrul, Hacialiogullari, Mehtap, Susar, Hatice, Arslan, Seda, Aycan, Kenan, Ozkaya, Vecihi, Pilmane, Mara, Boka, Sarmite, Ortug, Gursel, Ramirez, Carlos, Pascual-Font, Aran, Valderrama-Canales, Francisco, Kucukalic, Abdulah, Kapur, Eldan, Talovic, Elvira, Baca, Vaclav, Grill, Robert, Horak, Zdenek, Kachlik, David, Dzupa, Valer, Konarik, Marek, Knize, Jakub, Veleminsky, Petr, Smrzova, Tereza, Otcenasek, Michal, Chmelova, Jana, Kheck, Michal, Kheck, Sr., Michal, Cupka, Tomas, Hnatek, Lukas, van der Meijs, Floris, Cech, Pavel, Musil, Vladimir, Ozkan, H. Mustafa, Muratli, S. Kivanc, Tayefi, Hamid, Ergur, Ipek, Kiray, Amac, Toktas, Muhsin, Alkoc, Ozan, Acar, Tolgahan, Uzun, Ibrahim, Ozen, Oguz Asian, Aycicek, Abdullah, Alkoc, Ozan Alper, Unlu, Mehmet, Corumlu, Ufuk, Ikiz, Ihsaniye Coskun, Oygucu, I. Hakan, Sendemir, Erdogan, Kaner, Tuncay, Caglar, Veli, Eser, Olcay, Demir, Mehmet T., Iyigun, Omer, Pirzirenli, Gokhan, Kaya, Ahmet Hilmi, Aydin, Mennan Ece, Celik, Fahrettin, True, Hakan, Ozkaya, Sevket, Ergur, Bekir Ugur, Zeybek, Gulsah, Bacakoglu, Kadir, Tadjalli, Mina, Poostpasand, Aghdas, Mansouiri, Seid Hadi, Allahvaisi, Ozra, Soleimanirad, Jafar, Nikkhoo, Bahram, Nagato, Yasukazu, Haruki, Yasuo, Yazawa, Komazo, Okazaki, Tutomu, Haida, Munetaka, Imai, Yutaka, Peirouvi, Thmineh, Mahzad-Sadaghiani, Mehrzad, Noroozinia, Farahnaz, Siamak, Salami, Farjah, Gholamhosseine, Mola, Sima, Biegaj, Ewa, Skadorwa, Tymon, Pawlewicz, Konrad, Kapolka, Robert, Chachulska, Agata, Zabicka, Joanna, Krasowska, Aleksandra, Prusik, Alicja, Jaczewski, Grzegorz, Kolesnik, Adam, Taghavi, M. Mohsen, Alavi, S. Hasan, Moallem, S. Adel, Safikhani, Zahed, Panahi, Marzieh, Dabiri, Shahriar, Shekaari, Majid Asadi, Latorre, Rafael, Soria, Federico, Lopez-Albors, Octavio, Sarria, Ricardo, Ayala, Inacio, Serrano, Inma, Perez-Cuadrado, Enrique, Musienko, Vladimir, Tkachenko, Dmitry, Colakoglu, Neriman, Kus, Murat Abdulgani, Jalali, Mahdi, Nikravesh, Mohammad Reza, Moeen, Abbas Ali, Karimfar, Mohammad Hassan, Rafighdoost, Houshang, Mohammadi, Shabnam, Korneeva, Marina, Rafighdoust, Houshang, Lovasova, Kvetuse, Bolekova, Adriana, Kluchova, Darina, Sulla, Igor, Kapitonova, Marina Yurievna, Syed Ahmad Fuad, Syed Baharom, Jayakaran, Flossie, Shams, Ali Reza, Aghaee, Fereshteh, Baqer, Zohreh, Faroki, Mohamad, Das, Srijit, Kassim, Normadiah, Latiff, Azian, Suhaimi, Frihah, Ghafar, Norzana, Hlaing, Khin Pa Pa, Maatoq, Israa, Othman, Faizah, Kiray, Muge, Bagriyanik, Husnu Alper, Pekcetin, Cetin, Ozogul, Candan, Fidan, Mustafa, Suhaimi, Farihah, Sun, Fei, Sanchez-Margallo, Francisco, Gil, Francisco, Crisostomo, Verónica, Uson, Jesus, Ramirez, Gegorio, Turamanlar, Ozan, Kirpiko, Oguz, Haktanir, Alpay, Climent, Salvador, Losilla, Sergio, Climent, Maria, Sarikcioglu, Levent, Senol, Yesim, Yildirim, Fatos B., Utuk, Arzu, Kunicki, Jacek, Pasbakhsh, Parichehr, Omidi, Negar, Omidi, Hamed, Nazhvani, Fatemeh Dehghani, Ghalebi, Seyed Razi, Javan, Nima, Mohagery, Akrami, Bideskan, Ali Reza Ebrahimzadeh, Taheri, Mohammad Mehdi Hassanzadeh, Fazel, Ali Reza, Tiengo, Cesare, Macchi, Veronica, Stecco, Carla, Porzionato, Andrea, Mazzoleni, Franco, De Caro, Raffaele, Clemente, Alberto, Morra, Aldo, Greco, Pietro, Pavan, Piero, Natali, Arturo, Demir, Mehmet, Dokur, Mehmet, Acer, Niyazi, Mavi, Ayfer, Matveeva, Niki, Lazarova, Dobrila, Korneti, Kostandina, Jovevska, Svetlana, Jurkovik, Dragica, Papazova, Meri, Havasi, Masoumeh, Alboghobeish, Naeim, Savari, Ahmad, Salamat, Negin, Sharifi, Mozafar, Kwak, Hyun-Ho, Hu, Kyung-Seok, Kim, Gyoo-Cheon, Park, Bong-Soo, Kim, Hee-Jin, Sinav, Ahmet, Gulati, Adarsh K., Gulati, Nidhi K., Alshammary, Hussien, Nazhvani, Seifollah Dehghani, Vafafar, Amir, Esmaeilpour, Tahereh, Bahmanpour, Soghra, Elyasi, Leila, Monabbati, Ahmad, Ghanadi, M., Paryani, Mohammad Reza, Gilanpour, Hassan, Amirsam, Banino, Omaña, Rodrigo Elizondo, López, Santos Guzmán, De la Garza Castro, Oscar, Vega, Edgar Urrutia, Lopez, Santos Guzman, Talebpour, Freshteh, Golmohammadi, Rahim, Dashti, Golamreza, Atlasi, Mohammad Ali, Mehdizadeh, Mehdi, Bahadori, Mohammad Hadi, Joghataei, Mohammad Taghi, Hatami, Leili, Boroujeni, Mandana Beigi, Estakhr, Jasem, Esfandiary, Ebrahim, Marzban, Mohsen, Bakhtiary, Mehrdad, Modiry, Navid, Jafarpur, Mokhtar, Mofidpur, Hassan, Alavi, S. Hassan, Mahmoudian, Alareza, Taghavi, Mohmmad Mohsen, Jafarpour, Mokhtar, Mahmoudian, Ali Reza, Sanjarmousavi, Nasrin, Doassans, Ines, Sorrenti, Natalia, Decuadro, German, Saibene, Andres, Poumayrac, Marie, Laza, Sebastian, Almiron, Carina, Vergara, Maria Elena, Soria, Victor, Lasa, Sebastian, Perez, Adolfo, Castro, Gabriela, Maria, Ana Santa, Soleimani, Mansoureh, Katebi, Majid, Bakhshayesh, Masoomeh, Oner, Mithat, Halici, Mehmet, Yikilmaz, Ali, Guney, Ahmet, Turk, Yildirim, Edizer, Mete, Beden, Umit, Icten, Nihal, Afshar, Mohammad, Hasanzadeh Taheri, Mohammad Mehdi, Moalem, Adel, Golalipour, Mohammad Jafar, Tamizi, Azadeh, Ahi, Mohammad, Mohammadpour, Shahram, Maiery, Ardeshir, Acikel, Cengiz, Ulkur, Ersin, Karagoz, Huseyin, Celikoz, Bahattin, Bedi, Kuldip, Ginus, Partadiredja, Golalipoor, Mohammad Jafar, Mohammadi, Mohammad Reza, Jhand, Poya, Mansourian, Azad Reza, Hosseinpoor, Kanizreza, Keshtkar, Abbas Ali, Alsaffar, Raith, Balajadeh, Babak Kabiri, Ghafari, Soraya, Azarhosh, Ramin, Fazeli, Seyyed Amirhossein, Jahanshahi, Mehrdad, Gharravi, Annen Mohammad, Alicioglu, Banu, Karakas, Hakki Muammer, Harma, Ahmet, Yang, Hun-Mu, Won, Sung-Yoon, Lee, Jae-Gi, Lee, Ju-Young, Lee, Jeong-Yong, Kim, Yoo-Ri, Song, Wu-Chul, Koh, Ki-Seok, Hwang, Eu-Na, Choi, Hyun-Gon, Kim, Soon-Heum, Kim, Soo-Young, Hur, Mi-Sun, Ulucam, Enis, Celbis, Osman, Kim, Da-Hye, Hong, Hee-Suk, Kim, Hyun-Joo, Choi, Jong-Hoon, Park, Jong-Tae, Kim, Hyeon-Cheol, Abbasi, Hamed, Hosseinipanah, Seyed Mohammad, Hosseini, Mohammad, Amani, A., Ashrafi, H. R., Sadeghimehr, Mohsen, Kim, Hyun-Ju, Sheverdin, Vadim, Amani, Zahra, Ashrafi, Alireza, Ashrafi, Ali Reza, Javad, Hami, Kachap, Mokhtar Jafarpoor, Laza, Sebastián, Poumayrac, Marie Catherine, Doassans, Inés, Vergara, María Elena, Almirón, Carina, Soria, Víctor, Rivara, Alvaro, Sirilo, Angela, Freire, Diego, Cirillo, Angela, Veragara, Maria Elena, Krmek, Vlado, Krmek, Nikola, Jo-Osvatic, Ana, Nikolic, Vasilije, Radic, Radivoje, Tubbs, R. Shane, Loukas, Marios, Fogg, Quentin, Ashwood, Neil, Cilingiroglu, Serpil, Ozbakir, Cemal, Mazoochi, Tahereh, Sabanciogullari, Vedat, Gumus, Cesur, Erdil, F. Hayat, Cimen, Mehmet, Moodi, Hesam, Ghiasi, Fateme, Akbari, Asghar, Hami, Javad, Khazei, Majid, Haghparast, Elham, Mitsakis, Ioannis, Anastasiou, Aikaterini, Mitsakis, Menelaos, Sianou, Kyriaki, Hainoglou, Roxani, Francisco, Margarida, Mitsaki, Charikleia, Konstantinidi, Maria, Prapa, Stamatia, Leksan, Igor, Mrcela, Tomislav, Selthofer, Robert, Kermanian, Fatemeh, Mahmoudian, Alireza, Ahmadpoor, Mahmood Erfanian, Dalili, Naser, Elian, Amir Hossein, Moaiery, Ardesheer, Jamalpour, Zahra, Nourani, Mohammad Reza, Asgari, Alireza, Hassanzadeh Taheri, Mohammad Mehdi, Ebrahimzadeh, Alireza, Eftekharvaghefi, Seyed Hasan, Mohammadi, Abbas, Sheibani, Vahid, Nematollahi-Mahani, Seyed Noureddin, Latifpour, Mastafa, Deilami, Masood, Soroure-Azimzadeh, Behzad, Nabipour, Fatemeh, Najafipour, Hamid, Nakhaee, Nouzar, Yaghoobi, Mohammad, Eftekharvaghefi, Rana, Salehinejad, Parvin, Azizi, Hasan, Riasi, Hamid Reza, Nobakht, Maliheh, Asalgoo, Sara, Rahbar, Roshanak, Najafzadeh, Norooz, Moosavizadeh, Kazem, Ezzatabadypour, Massood, Majidi, Masoud, Malekpor-Afshar, Reza, Karimzade, Fariba, Hoseini, Mahmood, Bayat, Mohamad, Gorgi, Ali, Nezhadi, Akram, Bakhtiari, Mehrdad, Jazi, Homa Rasooli, Jafaryan, Maryam, Haghir, Hosein, Hosseini, Mahmood, Rahimi, Sadegh, Rassouli, Fatemeh Behnam, Gorji, Ali, Habibi, Aliasghar, Pouya, Fatemeh, Dabiri, Shahryar, Mousavi, A., Rajabalian, Saeed, Abolidokht, A., Khanlarkhani, Neda, Naderian, Homayoun, Berjis, Nezamedin, Namavar, Mohamad Reza, Talaei, Tahereh, Mazaheri, Zohreh, Monabati, Ahmad, Kosar, Mehmet Ilkay, Karacan, Kezban, Chegini, Hamidreza, Nikzad, Hossein, Ayhan, Egemen, Ustundag, Sinan, Akkin, Salih Murat, Ogut, Tahir, Rayegan, Parviz, Meibodi, Mohamad Ali Emami, Ghaem, Reza Montazer, Zargarpoor, Rosa, Eftekhar Vaghefi, Seyd Hasan, Moshkdanian, Ghazale, Poya, Fateme, Kohestani, Hamid, Abarghoeai, Roozbeh Rayegan, Abarghoeai, Parviz Rayegan, Eftekhar Vaghefi, Seyed Hasan, Mahmodi, Abolghasem Amir, Poraboli, Ali, Kohestani, Hamid Reza, Vaghefi, Raena Eftekhar, Eftekhar Vaghefy, Seyed Hasan, Vaghefy, Raena Eftekhar, Abarghoeai, Parviz Raygan, Saba, Mohamad, Gharravi, Anneh Mohammad, Javadnia, Fatemeh, Zhaleh, Mohsen, Nezhad, Dariush Bijan, Gholami, Mohammad Reza, Piagkou, Maria, Aikaterini, Vassiliki Kouki, Piagkos, Giannoulis, Douvetzemis, Stergios, Skandalakis, Panagiotis, Anagnostopoulou, Sophia, Papadopoulos, Nikolaos, Celik, H. Hamdi, Tatar, Ilkan, Tatar, Emel Cadalli, Mocan, Burce Ozgen, Sargon, Mustafa F., Denk, C. Cem, Rasoolijazi, Homa, Joghataie, Mohammad Taghi, Roghani, Mehrdad, Akkin, Salin Murat, Dinc, Gulten, Kurklu, Mustafa, Ozboluk, Sener, Komurcu, Mahmut, Koebke, Jürgen, Balioglu, Mehmet Bulent, Kaygusuz, Mehmet Akif, Bozkus, Ferdi Sefa, Korkmaz, Ozgur, Bayram, Sule Biyik, Can, Mehmet Ali, Nasiri, Ebrahim, Jafar-Kazemi, Koroush, Hosseini, Melina, Maghoul, Shahin, Soleimani, Mansooreh, Amini, Abdollah, Hassanzade, Mohamad Mahdi, Davari, Mohammad Hossein, Van Hoof, Tom, Gomes, Germano T., Audenaert, Emmanuel, Verstraete, Koenraad, Kerckaert, Ingrid, D’Herde, Katharina, Benninger, Brion, Hedley, Gil, Filipoiu, Florin Mihail, Tarta, Eugen, Enyedi, Mihali, Pantu, Cosmin, Stanciulescu, Razvan, Skobowiat, Cezary, Calka, Jaroslaw, Majewski, Mariusz, Rezaian, Maryam, Yaghoobfar, Akbar, Hamedi, Somayeh, and Shomali, T.

Published
2009
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9. Metabolic risk factors of ovarian cancer: a review

Author

Khanlarkhani, Neda, primary, Azizi, Elham, additional, Amidi, Fardin, additional, Khodarahmian, Mahshad, additional, Salehi, Ensieh, additional, Pazhohan, Azar, additional, Farhood, Bagher, additional, Mortezae, Keywan, additional, Goradel, Nasser Hashemi, additional, and Nashtaei, Maryam Shabani, additional

Published
2021
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13. Macrophage polarity in cancer: A review

Author

Najafi, Masoud, primary, Hashemi Goradel, Nasser, additional, Farhood, Bagher, additional, Salehi, Eniseh, additional, Nashtaei, Maryam Shabani, additional, Khanlarkhani, Neda, additional, Khezri, Zahra, additional, Majidpoor, Jamal, additional, Abouzaripour, Morteza, additional, Habibi, Mohsen, additional, Kashani, Iraj Ragerdi, additional, and Mortezaee, Keywan, additional

Published
2018
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23. Comparison of the effects of progesterone and 17 β-estradiol on Schwann cell markers expression in rat adipose-derived stem cells.

Author

Naderain, Homayoun, Khanlarkhani, Neda, Kashani, Iraj Ragerdi, Atlasi, Amirabbas, and Atlasi, Mohammad Ali

Subjects
PROGESTERONE, ESTRADIOL, SCHWANN cells, MESENCHYMAL stem cells, ADIPOSE tissues, PLATELET-derived growth factor
Abstract

Steroids promote the myelination and regeneration in the peripheral nervous system. Whereas, little is known about the inducing effects by which the hormones exert their effects on Schwann cells differentiation. This could be revealed by the expression of Schwann cell markers in adipose-derived stem cells (ADSCs). The purpose of this study was to present the effects of progesterone and 17 β-estradiol on the Schwann cell markers in rat ADSCs. The mesenchymal stem cell markers (CD73, and CD90) were assayed by flow cytometry. Rat ADSCs were sequentially treated with β-mercaptoethanol, and all-trans-retinoic acid, followed by a mixture of basic fibrobroblast growth factor, platelet-derived growth factor, forskolin and heregulin. In experimental groups, forskolin and heregulin were substituted by progesterone and 17 β-estradiol. After induction, the expression of Schwann cell markers P0, and S-100 and the cellular immunocytochemical staining positive rate of anti-S100 and antiglial fibrillary acidic protein (GFAP) antibodies were compared in the experimental and control groups. Progesterone and 17 β-estradiol triggered P0 and S-100 genes expression and induced a cellular immunocytochemical staining positive rate of S-100 and GFAP in rats ADSCs. Progesterone induced these changes stronger than 17 β-estradiol. Thus, progesterone may induce rat ADSCs toward Schwann-like cells by expression of Schwann cell markers and is more potent than 17 β-estradiol in the expression of these markers. [ABSTRACT FROM AUTHOR]

Published
2019
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24. Curcumin as an anti‐inflammatory agent: Implications to radiotherapy and chemotherapy.

Author

Farhood, Bagher, Mortezaee, Keywan, Goradel, Nasser Hashemi, Khanlarkhani, Neda, Salehi, Ensieh, Nashtaei, Maryam Shabani, Najafi, Masoud, and Sahebkar, Amirhossein

Subjects
CURCUMIN, ANTI-inflammatory agents, RADIOTHERAPY, CANCER chemotherapy, SKIN inflammation, TUMOR growth
Abstract

Cancer is the second cause of death worldwide. Chemotherapy and radiotherapy are the most common modalities for the treatment of cancer. Experimental studies have shown that inflammation plays a central role in tumor resistance and the incidence of several side effects following both chemotherapy and radiotherapy. Inflammation resulting from radiotherapy and chemotherapy is responsible for adverse events such as dermatitis, mucositis, pneumonitis, fibrosis, and bone marrow toxicity. Chronic inflammation may also lead to the development of second cancer during years after treatment. A number of anti‐inflammatory drugs such as nonsteroidal anti‐inflammatory agents have been proposed to alleviate chronic inflammatory reactions after radiotherapy or chemotherapy. Curcumin is a well‐documented herbal anti‐inflammatory agents. Studies have proposed that curcumin can help management of inflammation during and after radiotherapy and chemotherapy. Curcumin targets various inflammatory mediators such as cyclooxygenase‐2, inducible nitric oxide synthase, and nuclear factor κB (NF‐κB), thereby attenuating the release of proinflammatory and profibrotic cytokines, and suppressing chronic production of free radicals, which culminates in the amelioration of tissue toxicity. Through modulation of NF‐κB and its downstream signaling cascade, curcumin can also reduce angiogenesis, tumor growth, and metastasis. Low toxicity of curcumin is linked to its cytoprotective effects in normal tissues. This protective action along with the capacity of this phytochemical to sensitize tumor cells to radiotherapy and chemotherapy makes it a potential candidate for use as an adjuvant in cancer therapy. There is also evidence from clinical trials suggesting the potential utility of curcumin for acute inflammatory reactions during radiotherapy such as dermatitis and mucositis. [ABSTRACT FROM AUTHOR]

Published
2019
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25. Melatonin and cancer: From the promotion of genomic stability to use in cancer treatment.

Author

Farhood, Bagher, Goradel, Nasser Hashemi, Mortezaee, Keywan, Khanlarkhani, Neda, Najafi, Masoud, and Sahebkar, Amirhossein

Subjects
CANCER treatment, DNA damage, DISEASE incidence, MELATONIN, OXIDATIVE stress, ANTICARCINOGENIC agents
Abstract

Cancer remains among the most challenging human diseases. Several lines of evidence suggest that carcinogenesis is a complex process that is initiated by DNA damage. Exposure to clastogenic agents such as heavy metals, ionizing radiation (IR), and chemotherapy drugs may cause chronic mutations in the genomic material, leading to a phenomenon named genomic instability. Evidence suggests that genomic instability is responsible for cancer incidence after exposure to carcinogenic agents, and increases the risk of secondary cancers following treatment with radiotherapy or chemotherapy. Melatonin as the main product of the pineal gland is a promising hormone for preventing cancer and improving cancer treatment. Melatonin can directly neutralize toxic free radicals more efficiently compared with other classical antioxidants. In addition, melatonin is able to regulate the reduction/oxidation (redox) system in stress conditions. Through regulation of mitochondrial nction and inhibition of pro‐oxidant enzymes, melatonin suppresses chronic oxidative stress. Moreover, melatonin potently stimulates DNA damage responses that increase the tolerance of normal tissues to toxic effect of IR and may reduce the risk of genomic instability in patients who undergo radiotherapy. Through these mechanisms, melatonin attenuates several side effects of radiotherapy and chemotherapy. Interestingly, melatonin has shown some synergistic properties with IR and chemotherapy, which is distinct from classical antioxidants that are mainly used for the alleviation of adverse events of radiotherapy and chemotherapy. In this review, we describe the anticarcinogenic effects of melatonin and also its possible application in clinical oncology. In this review, we describe the anticarcinogenic effects of melatonin and also its possible application in clinical oncology. [ABSTRACT FROM AUTHOR]

Published
2019
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27. Macrophage polarity in cancer: A review.

Author

Najafi, Masoud, Hashemi Goradel, Nasser, Farhood, Bagher, Salehi, Eniseh, Nashtaei, Maryam Shabani, Khanlarkhani, Neda, Khezri, Zahra, Majidpoor, Jamal, Abouzaripour, Morteza, Habibi, Mohsen, Kashani, Iraj Ragerdi, and Mortezaee, Keywan

Published
2019
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28. Adjuvant chemotherapy with melatonin for targeting human cancers: A review.

Author

Najafi, Masoud, Salehi, Eniseh, Farhood, Bagher, Nashtaei, Maryam Shabani, Hashemi Goradel, Nasser, Khanlarkhani, Neda, Namjoo, Zeinab, and Mortezaee, Keywan

Subjects
CANCER chemotherapy, MELATONIN, DRUG resistance, CANCER invasiveness
Abstract

Melatonin is a multifunctional hormone that has long been known for its antitumoral effects. An advantage of the application of melatonin in cancer therapy is its ability to differentially influence tumors from normal cells. In this review, the roles of melatonin adjuvant therapy in human cancer are discussed. Combination of melatonin with chemotherapy could provide synergistic antitumoral outcomes and resolve drug resistance in affected patients. This combination reduces the dosage for chemotherapeutic agents with the subsequent attenuation of side effects related to these drugs on normal cells around tumor and on healthy organs. The combination therapy increases the rate of survival and improves the quality of life in affected patients. Cancer cell viability is reduced after application of the combinational melatonin therapy. Melatonin does all these functions by adjusting the signals involved in cancer progression, re‐establishing the dark/light circadian rhythm, and disrupting the redox system for cancer cells. To achieve effective therapeutic outcomes, melatonin concentration along with the time of incubation for this indoleamine needs to be adjusted. Importantly, a special focus is required to be made on choosing an appropriate chemotherapy agent for using in combination with melatonin. Because of different sensitivities of cancer cells for melatonin combination therapy, cancer‐specific targeted therapy is also needed to be considered. For this review, the PubMed database was searched for relevant articles based on the quality of journals, the novelty of articles published by the journals, and the number of citations per year focusing only on human cancers. Combination of melatonin with chemotherapy for targeting human cancer. Combination of melatonin with chemotherapy could provide synergistic antitumoral outcomes and resolve drug resistance in affected patients. This combination reduces the dosage for chemotherapeutic agents with the subsequent attenuation of side effects related to these drugs on normal cells around tumor and on healthy organs. The combination therapy increases the rate of survival and improves the quality of life in affected patients. Cancer cell viability is reduced after application of the combinational melatonin therapy. [ABSTRACT FROM AUTHOR]

Published
2019
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29. Fusobacterium nucleatum and colorectal cancer: A mechanistic overview.

Author

Hashemi Goradel, Nasser, Heidarzadeh, Siamak, Jahangiri, Samira, Farhood, Bagher, Mortezaee, Keywan, Khanlarkhani, Neda, and Negahdari, Babak

Subjects
FUSOBACTERIUM, COLON cancer, TUMOR microenvironment, CARCINOGENESIS, MICRORNA
Abstract

Colorectal cancer (CRC) is the third most prevalent cancer in the world. There are many risk factors involved in CRC. According to recent findings, the tumor microenvironment and feces samples of patients with CRC are enriched by Fusobacterium nucleatum. Thus, F. nucleatum is proposed as one of the risk factors in the initiation and progression of CRC. The most important mechanisms of Fusobacterium nucleatum involved in CRC carcinogenesis are immune modulation (such as increasing myeloid‐derived suppressor cells and inhibitory receptors of natural killer cells), virulence factors (such as FadA and Fap2), microRNAs (such as miR‐21), and bacteria metabolism. The aim of this review was to evaluate the mechanisms underlying the action of F. nucleatum in CRC. One of the probabilities in colorectal cancer (CRC) development is Fusobacterium. F. nucleatum could contribute in CRC development through virulence factors, microRNAs (miRNA), immune evasion, and increasing metastasis. [ABSTRACT FROM AUTHOR]

Published
2019
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31. Effect of brain-derived neurotrophic factor (BDNF) on sperm quality of normozoospermic men.

Author

Safari, Hassan, Khanlarkhani, Neda, Sobhani, Aligholi, Najafi, Atefeh, and Amidi, Fardin

Subjects
*ALKALOIDS, *CLINICAL trials, *ORGAN donation, *ENZYME-linked immunosorbent assay, *FLOW cytometry, *INSULIN, *LIPID peroxidation (Biology), *MICROSCOPY, *MITOCHONDRIA, *NITRIC oxide, *SPERMATOZOA, *SPERM motility, *STAINS & staining (Microscopy), *LEPTIN, *BRAIN-derived neurotrophic factor
Abstract

The neurotrophin family of proteins and their receptors act as important proliferative and pro-survival factors in differentiation of nerve cells and are thought to play key roles in the development of reproductive tissues and normal function of spermatozoa. The objective of the present study was to evaluate the effect of Brain-Derived Neurotrophic Factor (BDNF) on the sperm viability and motility, lipid peroxidation (LPO), mitochondrial activity and concentration of leptin, nitric oxide (NO) and insulin in normozoospermic men. Semen samples from 20 normozoospermic men were divided into three groups: (i) control, (ii) BDNF and (iii) BDNF + K252a. BDNF and K252a were added in the dose of 0.133 and 0.1 nM, respectively. Viability was assessed by eosin-nigrosin staining technique, and motility was observed by microscopy. NO concentration and mitochondrial activity were measured with flow cytometry, and LPO was analyzed using enzyme-linked immunosorbent assay (ELISA) kits. Results showed that exogenous BDNF at 0.133 nM could significantly (p < 0.05) influence viability, motility, NO concentration, mitochondrial activity and LPO content. Secretions of insulin and leptin by human sperm were increased in cells exposed to the exogenous BDNF, whereas viability, mitochondrial activity and insulin and leptin secretions were decreased in cells exposed to the K252. [ABSTRACT FROM AUTHOR]

Published
2018
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32. Comparison of the effects of progesterone and 17 β-estradiol on Schwann cell markers expression in rat adipose-derived stem cells.

Author

Naderain, Homayoun, Khanlarkhani, Neda, Kashani, Iraj Ragerdi, Atlasi, Amirabbas, and Atlasi, Mohammad Ali

Subjects
SCHWANN cells, MESENCHYMAL stem cells, ADIPOSE tissues, CELL proliferation, GENE expression
Abstract

Steroids promote the myelination and regeneration in the peripheral nervous system. Whereas, little is known about the inducing effects by which the hormones exert their effects on Schwann cells differentiation. This could be revealed by the expression of Schwann cell markers in adipose-derived stem cells (ADSCs). The purpose of this study was to present the effects of progesterone and 17 β-estradiol on the Schwann cell markers in rat ADSCs. The mesenchymal stem cell markers (CD73, and CD90) were assayed by flow cytometry. Rat ADSCs were sequentially treated with β-mercaptoethanol, and all-trans-retinoic acid, followed by a mixture of basic fibrobroblast growth factor, platelet-derived growth factor, forskolin and heregulin. In experimental groups, forskolin and heregulin were substituted by progesterone and 17 β-estradiol. After induction, the expression of Schwann cell markers P0, and S-100 and the cellular immunocytochemical staining positive rate of anti-S100 and antiglial fibrillary acidic protein (GFAP) antibodies were compared in the experimental and control groups. Progesterone and 17 β-estradiol triggered P0 and S-100 genes expression and induced a cellular immunocytochemical staining positive rate of S-100 and GFAP in rats ADSCs. Progesterone induced these changes stronger than 17 β-estradiol. Thus, progesterone may induce rat ADSCs toward Schwann-like cells by expression of Schwann cell markers and is more potent than 17 β-estradiol in the expression of these markers. [ABSTRACT FROM AUTHOR]

Published
2018
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33. Melatonin application in targeting oxidative‐induced liver injuries: A review.

Author

Mortezaee, Keywan and Khanlarkhani, Neda

Subjects
*LIVER injuries, *MELATONIN, *OXIDATIVE stress, *LIVER physiology, *FATTY liver
Abstract

It is believed that oxidative stress is a key causing factor of liver damage induced by a variety of agents, and it is a major contributing factor in almost all conditions compromising liver function, including ischemia–reperfusion injury (IRI), nonalcoholic fatty liver disease (NAFLD), nonalcoholic steatohepatitis (NASH), liver fibrosis, liver cirrhosis, and hepatocellular carcinoma (HCC). Liver is the organ that high concentration of melatonin (N‐acetyl‐5‐methoxytryptamine) accumulates, and it is the sole organ where circulating melatonin is metabolized. Melatonin is one of the best antioxidants that protects liver, and its metabolites also have antioxidative function. Melatonin exerts its antioxidative function directly through its radical scavenging ability and indirectly through stimulation of antioxidant enzymes. The antioxidative response from melatonin in liver affects from various factors, including its dosage, route, time and duration of administration, the type of oxidative‐induced agent and species aging. This indoleamine is also an effective and promising antioxidative choice for targeting liver IRI, NAFLD, NASH, fibrosis, cirrhosis, and HCC. [ABSTRACT FROM AUTHOR]

Published
2018
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34. Role of spermatogonial stem cells extract in transdifferentiation of 5-Aza-2′-deoxycytidine-treated bone marrow mesenchymal stem cells into germ-like cells

Author

Kharizinejad, Ebrahim, primary, Minaee Zanganeh, Bagher, additional, Khanlarkhani, Neda, additional, Mortezaee, Keywan, additional, Rastegar, Tayebeh, additional, Baazm, Maryam, additional, Abolhassani, Farid, additional, Sajjadi, Seyed Mehdi, additional, Hajian, Mahdieh, additional, Aliakbari, Fereshte, additional, and Barbarestani, Mohammad, additional

Published
2016
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36. Multipotent Stem Cell and Current Application.

Author

Sobhani, Aligholi, Khanlarkhani, Neda, Baazm, Maryam, Mohammadzadeh, Farzaneh, Najafi, Atefeh, Mehdinejadiani, Shayesteh, and Aval, Fereydoon Sargolzaei

Subjects
*MULTIPOTENT stem cells, *PLURIPOTENT stem cells, *SPINAL cord injuries, *THERAPEUTICS, *RHEUMATOID arthritis treatment, *TREATMENT of fractures
Abstract

Stem cells are self-renewing and undifferentiated cell types that can be differentiate into functional cells. Stem cells can be classified into two main types based on their source of origin: Embryonic and Adult stem cells. Stem cells also classified based on the range of differentiation potentials into Totipotent, Pluripotent, Multipotent, and Unipotent. Multipotent stem cells have the ability to differentiate into all cell types within one particular lineage. There are plentiful advantages and usages for multipotent stem cells. Multipotent Stem cells act as a significant key in procedure of development, tissue repair, and protection. Multipotent Stem cells have been applying in treatment of different disorders such as spinal cord injury, bone fracture, autoimmune diseases, rheumatoid arthritis, hematopoietic defects, and fertility preservation. [ABSTRACT FROM AUTHOR]

Published
2017

37. Multipotent Stem Cell and Reproduction.

Author

Khanlarkhani, Neda, Baazm, Maryam, Mohammadzadeh, Farzaneh, Najafi, Atefeh, Mehdinejadiani, Shayesteh, and Sobhani, Aligholi

Subjects
MULTIPOTENT stem cells, PLURIPOTENT stem cells, TOTIPOTENCY (Cytology), DNA repair, INFERTILITY treatment
Abstract

Stem cells are self-renewing and undifferentiated cell types that can be differentiate into functional cells. Stem cells can be classified into two main types based on their source of origin: Embryonic and Adult stem cells. Stem cells also classified based on the range of differentiation potentials into Totipotent, Pluripotent, Multipotent, and Unipotent. Multipotent stem cells have the ability to differentiate into all cell types within one particular lineage. There are plentiful advantages and usages for multipotent stem cells. Multipotent Stem cells act as a significant key in procedure of development, tissue repair, and protection. The accessibility and adaptability of these amazing cells create them a great therapeutic choice for different part of medical approaches, and it becomes interesting topic in the scientific researches to found obvious method for the most advantageous use of MSC-based therapies. Recent studies in the field of stem cell biology have provided new perspectives and opportunities for the treatment of infertility disorders. [ABSTRACT FROM AUTHOR]

Published
2016

38. The effects of progestrone on the in-vitro expression of P0, S100 and Krox20 genes in adipose-derived stem cells.

Author

Khanlarkhani, Neda, Atlasi, Mohammad Ali, Kashani, Iraj Ragerdi, Naderian, Homayoun, Taherian, Ali Akbar, and Nikzad, Hossein

Subjects
*PROGESTERONE, *STEM cells, *CELL lines, *CELL separation, *FLOW cytometry, *CELL culture
Abstract

Background: Adipose-derived stem cells (ADSCs) have noticeable self-renewal ability and can differentiate into several cell lines such as adipocytes, osteoblasts, chondrocytes, and myocytes. Progesterone plays a significant role in the myelination of peripheral nerves. Regarding the role of progesterone on the myelination of peripheral nervous system, we evaluated its effects on the in-vitro expression of P0, S100 and Krox20 mRNA in adipose-derived stem cells. Methods: In this experimental study, rat adipose-derived stem cells were isolated from the inguinal region of the animals and were evaluated by flow cytometry before culture. In preinduction phase, the cells were sequentially treated with various factors such as β- mercaptoethanol and all-trans-retinoic acid, followed by different induction mixtures. The cells were divided into four groups including two control groups (receiving either fibroblast and platelet derived-growth factors, or fibroblast growth factor, platelet derived-growth factor, forskolin and heregulin) and two experimental groups (receiving either fibroblast growth factor, platelet derived-growth factor, forskolin and progesterone, or fibroblast growth factor, platelet derived-growth factor, heregulin and progesterone). Expression of Schwann cell markers, S-100, P0 and Krox20 mRNA, was determined by semi-quantitative RT-PCR. Results: ADSCs expressed CD90, CD73, and CD31 but showed lack of CD45, and VEGFR2 expression. After the induction stage, S-100, P0 and Krox20 mRNA were expressed in the progesterone receiving experimental groups, but expression of S-100 and Krox20 mRNA were less than the control group which was receiving forskolin and heregulin (P<0.0001). Conclusion: Progesterone can promote the in-vitro expression of S-100, P0, and Krox20 genes in adipose-derived stem cells. [ABSTRACT FROM AUTHOR]

Published
2011

39. Metabolic risk factors of ovarian cancer: a review.

Author

Khanlarkhani N, Azizi E, Amidi F, Khodarahmian M, Salehi E, Pazhohan A, Farhood B, Mortezae K, Goradel NH, and Nashtaei MS

Subjects
Female, Humans, Incidence, Obesity complications, Obesity epidemiology, Risk Factors, Diabetes Mellitus epidemiology, Ovarian Neoplasms epidemiology, Ovarian Neoplasms etiology
Abstract

Ovarian cancer continues to be the leading cause of death from gynecological cancers. Despite inconsistent results, patients with metabolic abnormalities, including obesity and diabetes mellitus (DM), have poorer outcomes, showing a correlation with ovarian cancer incidence and ovarian cancer survival. Since ovarian cancer is the most common cancer in women, and considering the increasing prevalence of obesity and DM, this paper reviews the literature regarding the relationship between the aforementioned metabolic derangements and ovarian cancer, with a focus on ovarian cancer incidence, mortality, and likely mechanisms behind them. Several systematic reviews and meta-analyses have shown that obesity is associated with a higher incidence and poorer survival in ovarian cancer. Although more studies are required to investigate the etiological relation of DM and ovarian cancer, sufficient biological evidence indicates poorer outcomes and shorter survival in DM women with ovarian cancer. A variety of pathologic factors may contribute to ovarian cancer risk, development, and survival, including altered adipokine expression, increased levels of circulating growth factors, altered levels of sex hormones, insulin resistance, hyperinsulinemia, and chronic inflammation. Thus, obesity and DM, as changeable risk factors, can be targeted for intervention to prevent ovarian cancer and improve its outcomes.

Published
2022
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