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1. Combination of dual JAK/HDAC inhibitor with regorafenib synergistically reduces tumor growth, metastasis, and regorafenib-induced toxicity in colorectal cancer

2. Molecular Characterization of KRAS Wild-type Tumors in Patients with Pancreatic Adenocarcinoma.

4. PBRM1 mutations might render a subtype of biliary tract cancers sensitive to drugs targeting the DNA damage repair system

7. Molecular Differences With Therapeutic Implications in Early-Onset Compared With Average-Onset Biliary Tract Cancers.

9. Allele-specific KRAS mutations as prognostic biomarkers in mucinous adenocarcinoma of the appendix and colorectal cancer.

11. Contributors

15. The adherence to the American Association for the Study of Liver Diseases 2018 guidelines in the management of hepatocellular carcinoma and its impact on survival

16. Molecular profile of BRCA-mutated biliary tract cancers

17. Molecular differences between lymph nodes and distant metastases compared with primaries in colorectal cancer patients

19. Association of class II and III BRAF mutations with EGFR blockade therapy response and representation of molecularly distinct subgroups of BRAF mutations in MMR proficient CRC.

20. Genomic analysis of oesophageal carcinoma (EC) to identify recurrent mutations in histone methyltransferases as a distinctive subset.

21. Molecular differences with therapeutic implications in early-onset compared to average-onset cholangiocarcinoma.

22. Characterization of the cachexia pathway in pancreatic ductal adenocarcinoma.

23. APC as a high-utility mutational biomarker that may identify subpopulations of patients with mutant RAS/BRAF and right-sided colorectal cancer (CRC) who derive benefit from EGFR inhibitors (EGFRi).

27. Advances in Immunotherapy for Transplant Oncology.

32. Supplementary Figure 6 from BZW2 Inhibition Reduces Colorectal Cancer Growth and Metastasis

33. Supplementary Figure 7 from BZW2 Inhibition Reduces Colorectal Cancer Growth and Metastasis

34. Supplementary Figure 1 from BZW2 Inhibition Reduces Colorectal Cancer Growth and Metastasis

35. Supplementary Figure 5 from BZW2 Inhibition Reduces Colorectal Cancer Growth and Metastasis

36. Supplementary Figure 4 from BZW2 Inhibition Reduces Colorectal Cancer Growth and Metastasis

37. Supplementary Figure 8 from BZW2 Inhibition Reduces Colorectal Cancer Growth and Metastasis

38. Supplementary Data from BZW2 Inhibition Reduces Colorectal Cancer Growth and Metastasis

39. Supplementary Figure 3 from BZW2 Inhibition Reduces Colorectal Cancer Growth and Metastasis

40. Supplementary Figure 2 from BZW2 Inhibition Reduces Colorectal Cancer Growth and Metastasis

41. BZW2 Inhibition Reduces Colorectal Cancer Growth and Metastasis

42. Data from BZW2 Inhibition Reduces Colorectal Cancer Growth and Metastasis

43. Abstract 1003: The prognostic significance of prolyl 4-hydroxylase subunit alpha 1 and the therapeutic efficacy of diethyl-pythiDC, its small molecule inhibitor, in pancreatic ductal adenocarcinoma

44. Abstract 1297: BZW2 a potential target to inhibit colorectal cancer growth and metastasis

45. Figure S5 from The Landscape of Alterations in DNA Damage Response Pathways in Colorectal Cancer

46. Supplementary Methods from The Landscape of Alterations in DNA Damage Response Pathways in Colorectal Cancer

47. Data from Molecular Characterization of KRAS Wild-type Tumors in Patients with Pancreatic Adenocarcinoma

48. Supplementary Table from Molecular Characterization of KRAS Wild-type Tumors in Patients with Pancreatic Adenocarcinoma

49. Supplementary Figure from Molecular Characterization of KRAS Wild-type Tumors in Patients with Pancreatic Adenocarcinoma

50. Supplementary Tables from The Landscape of Alterations in DNA Damage Response Pathways in Colorectal Cancer

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