37 results on '"Kim, Kook Hwan"'
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2. An autophagy enhancer ameliorates diabetes of human IAPP-transgenic mice through clearance of amyloidogenic oligomer
3. Phosphorylation of EIF2S1 (eukaryotic translation initiation factor 2 subunit alpha) is indispensable for nuclear translocation of TFEB and TFE3 during ER stress
4. Cell non-autonomous effect of hepatic growth differentiation factor 15 on the thyroid gland
5. Snail acetylation by autophagy‐derived acetyl‐coenzyme A promotes invasion and metastasis of KRAS ‐ LKB1 co‐mutated lung cancer cells
6. Fibroblast growth factor 21 participates in adaptation to endoplasmic reticulum stress and attenuates obesity-induced hepatic metabolic stress
7. Amyloidogenic peptide oligomer accumulation in autophagy-deficient β cells induces diabetes
8. Autophagy as a crosstalk mediator of metabolic organs in regulation of energy metabolism
9. Age-dependent gait abnormalities in mice lacking the Rnf170 gene linked to human autosomal-dominant sensory ataxia
10. Autophagy deficiency leads to protection from obesity and insulin resistance by inducing Fgf21 as a mitokine
11. Autophagy—a key player in cellular and body metabolism
12. GDF15 as a central mediator for integrated stress response and a promising therapeutic molecule for metabolic disorders and NASH
13. Targeting of Secretory Proteins as a Therapeutic Strategy for Treatment of Nonalcoholic Steatohepatitis (NASH)
14. Bile acid induces expression of COX-2 through the homeodomain transcription factor CDX1 and orphan nuclear receptor SHP in human gastric cancer cells
15. Pathogenesis of Nonalcoholic Steatohepatitis and Hormone-Based Therapeutic Approaches
16. Growth Differentiation Factor 15 Ameliorates Nonalcoholic Steatohepatitis Associated with Metabolic Syndrome in Mice
17. Growth differentiation factor 15 ameliorates nonalcoholic steatohepatitis and related metabolic disorders in mice
18. A novel autophagy enhancer as a therapeutic agent against metabolic syndrome and diabetes
19. Ezetimibe ameliorates steatohepatitis via AMP activated protein kinase-TFEB-mediated activation of autophagy and NLRP3 inflammasome inhibition
20. Tumor suppressor protein VHL inhibits Hedgehog–Gli activation through suppression of Gli1 nuclear localization
21. Age-dependent gait abnormalities in mice lacking theRnf170gene linked to human autosomal-dominant sensory ataxia
22. FGF21 as a mediator of adaptive responses to stress and metabolic benefits of anti-diabetic drugs
23. Fibroblast growth factor 21 participates in adaptation to endoplasmic reticulum stress and attenuates obesity-induced hepatic metabolic stress
24. Curcumin inhibits hepatitis C virus replication via suppressing the Akt-SREBP-1 pathway
25. FGF21 as a Stress Hormone: The Roles of FGF21 in Stress Adaptation and the Treatment of Metabolic Diseases
26. Hepatitis C virus NS5A protein increases hepatic lipid accumulation via induction of activation and expression of PPARgamma
27. Steering Characteristics of an Autonomous Tractor with Variable Distances to the Waypoint
28. Metformin-induced inhibition of the mitochondrial respiratory chain increases FGF21 expression via ATF4 activation
29. Acute Exercise Induces FGF21 Expression in Mice and in Healthy Humans
30. Autophagy deficiency leads to protection from obesity and insulin resistance by inducing Fgf21 as a mitokine
31. SHP (small heterodimer partner) suppresses the transcriptional activity and nuclear localization of Hedgehog signalling protein Gli1
32. Hepatitis B Virus X Protein Impairs Hepatic Insulin Signaling Through Degradation of IRS1 and Induction of SOCS3
33. Curcumin inhibits hepatitis C virus replication via suppressing the Akt-SREBP-1 pathway
34. Hepatitis B virus X protein induces lipogenic transcription factor SREBP1 and fatty acid synthase through the activation of nuclear receptor LXRα
35. Hepatitis B Virus X Protein Induces Hepatic Steatosis Via Transcriptional Activation of SREBP1 and PPARγ
36. HCV core protein induces hepatic lipid accumulation by activating SREBP1 and PPARγ
37. Discovery of a selective cytochrome P450 4A inhibitor for the treatment of metabolic dysfunction-associated fatty liver disease.
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